Christian C. Naus focuses on Gap junction, Connexin, Cell biology, Transfection and Connexon. His research in Gap junction intersects with topics in Cell, Cell junction, Biophysics, Anatomy and Neuroscience. His Connexin research is multidisciplinary, incorporating perspectives in Gene expression, Cell growth and Pathology.
His study explores the link between Cell biology and topics such as Apoptosis that cross with problems in Neuronal damage. His Transfection research includes elements of Molecular biology and Green fluorescent protein. His Pannexin study incorporates themes from Membrane channel and Neuroglia.
His primary areas of investigation include Cell biology, Gap junction, Connexin, Neuroscience and Molecular biology. His Cell biology research incorporates themes from Cellular differentiation and Cell growth. His Gap junction study is concerned with Intracellular in general.
His studies deal with areas such as Membrane channel, Glioma and Pathology as well as Connexin. The Molecular biology study which covers Messenger RNA that intersects with Neural development. He has researched Astrocyte in several fields, including Neuroglia and Neuroprotection.
His primary areas of study are Cell biology, Connexin, Gap junction, Neuroscience and Glioma. His study looks at the relationship between Cell biology and topics such as Cartilage, which overlap with Ossification. His research integrates issues of Disease and Nervous system in his study of Connexin.
His study in the field of Connexon is also linked to topics like Population. His work on Hippocampus and Dentate gyrus as part of general Neuroscience research is often related to Large pore and Bridging, thus linking different fields of science. His Glioma study incorporates themes from Downregulation and upregulation, Motility, Parenchyma and Nestin.
Christian C. Naus mainly investigates Gap junction, Cell biology, Connexin, Cell signaling and Neuroscience. His research on Gap junction frequently connects to adjacent areas such as Hippocampal formation. His research on Cell biology focuses in particular on Signal transduction.
His work deals with themes such as Glioma and Nervous system, which intersect with Connexin. His work carried out in the field of Cell signaling brings together such families of science as Gap junction channel activity, Cytoplasm, microRNA and Membrane protein. Neuroprotection is closely connected to Disease in his research, which is encompassed under the umbrella topic of Neuroscience.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Intercellular calcium signaling in astrocytes via ATP release through connexin hemichannels.
Charles E. Stout;James L. Costantin;Christian C.G. Naus;Andrew C. Charles.
Journal of Biological Chemistry (2002)
Connexins regulate calcium signaling by controlling ATP release.
M. L. Cotrina;J. H.-C. Lin;A. Alves-Rodrigues;Shujun Liu.
Proceedings of the National Academy of Sciences of the United States of America (1998)
Role of connexin genes in growth control.
Hiroshi Yamasaki;Christian C.G. Naus.
Carcinogenesis (1996)
ATP-mediated glia signaling.
Maria Luisa Cotrina;Jane H.-C. Lin;Juan Carlos López-Garcı́a;Christian C. G. Naus.
The Journal of Neuroscience (2000)
Transfection of C6 glioma cells with connexin 43 cDNA: analysis of expression, intercellular coupling, and cell proliferation.
D. Zhu;S. Caveney;G. M. Kidder;C. C. G. Naus.
Proceedings of the National Academy of Sciences of the United States of America (1991)
ATP and glutamate released via astroglial connexin 43 hemichannels mediate neuronal death through activation of pannexin 1 hemichannels
Juan A. Orellana;Nicolas Froger;Pascal Ezan;Jean X. Jiang.
Journal of Neurochemistry (2011)
Intercellular calcium signaling via gap junctions in glioma cells.
A. C. Charles;C. C. G. Naus;Daguang Zhu;G. M. Kidder.
Journal of Cell Biology (1992)
Implications and challenges of connexin connections to cancer
Christian C. Naus;Dale W. Laird.
Nature Reviews Cancer (2010)
Pannexin channels are not gap junction hemichannels
Gina E. Sosinsky;Daniela Boassa;Rolf Dermietzel;Heather S. Duffy.
Channels (2011)
Amyloid β-Induced Death in Neurons Involves Glial and Neuronal Hemichannels
Juan A. Orellana;Kenji F. Shoji;Verónica Abudara;Pascal Ezan.
The Journal of Neuroscience (2011)
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