1991 - Fellow of the American Association for the Advancement of Science (AAAS)
1988 - Metlife Foundation Award for Medical Research in Alzheimer's Disease
Neuroscience, Alzheimer's disease, Hippocampus, Hippocampal formation and Internal medicine are his primary areas of study. His Neuroscience research includes themes of Glutamate receptor, Neurotrophic factors, Neurodegeneration and Cell biology. His Alzheimer's disease research integrates issues from Neurotoxicity, In vitro and Amyloid.
His Hippocampus study incorporates themes from Cerebral cortex and Nerve growth factor. Brain-derived neurotrophic factor and Neuron is closely connected to Endocrinology in his research, which is encompassed under the umbrella topic of Internal medicine. His work carried out in the field of Pathology brings together such families of science as Entorhinal cortex and Microglia.
His scientific interests lie mostly in Neuroscience, Internal medicine, Endocrinology, Hippocampal formation and Hippocampus. His study in Neuroscience is interdisciplinary in nature, drawing from both Alzheimer's disease, Lesion and Glutamate receptor. His Endocrinology study combines topics in areas such as Brain-derived neurotrophic factor and Neurotrophic factors.
His studies deal with areas such as Kainic acid and Cell biology as well as Hippocampal formation. His Cell biology research incorporates elements of Apoptosis, Biochemistry and Programmed cell death. As part of his studies on Hippocampus, he frequently links adjacent subjects like Pathology.
Carl W. Cotman spends much of his time researching Neuroscience, Internal medicine, Alzheimer's disease, Endocrinology and Cognition. His research in Hippocampus and Hippocampal formation are components of Neuroscience. His studies in Internal medicine integrate themes in fields like Lipoic acid and Immune system.
The concepts of his Alzheimer's disease study are interwoven with issues in Genetically modified mouse, Neurodegeneration and Amyloid. His studies deal with areas such as Temporal cortex and Immunology as well as Endocrinology. His Cognition research is multidisciplinary, incorporating elements of Psychological intervention, Physical medicine and rehabilitation, Developmental psychology, Disease and Cognitive decline.
His primary areas of study are Neuroscience, Alzheimer's disease, Internal medicine, Endocrinology and Cognitive decline. His Neuroscience research incorporates themes from Brain-derived neurotrophic factor and Neurotrophic factors. His Alzheimer's disease study is concerned with the larger field of Pathology.
His biological study spans a wide range of topics, including Mutation, Heteroplasmy and Somatic cell. His Endocrinology research is multidisciplinary, incorporating perspectives in Temporal cortex and Biochemistry. His research integrates issues of Embryonic stem cell and Anatomy in his study of Hippocampal formation.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Common Structure of Soluble Amyloid Oligomers Implies Common Mechanism of Pathogenesis
Rakez Kayed;Elizabeth Head;Jennifer L. Thompson;Theresa M. McIntire.
Exercise: a behavioral intervention to enhance brain health and plasticity
Carl W. Cotman;Nicole C. Berchtold.
Trends in Neurosciences (2002)
A Controlled Trial of Selegiline, Alpha-Tocopherol, or Both as Treatment for Alzheimer's Disease
Mary Sano;Christopher Ernesto;Ronald G. Thomas;Melville R. Klauber.
The New England Journal of Medicine (1997)
The Excitatory Amino Acid Receptors: Their Classes, Pharmacology, and Distinct Properties in the Function of the Central Nervous System
D. T. Monaghan;R. J. Bridges;C. W. Cotman.
Annual Review of Pharmacology and Toxicology (1989)
Exercise builds brain health: key roles of growth factor cascades and inflammation
Carl W. Cotman;Nicole C. Berchtold;Lori-Ann Christie.
Trends in Neurosciences (2007)
Neurodegeneration induced by beta-amyloid peptides in vitro: the role of peptide assembly state
Christian J. Pike;Debra Burdick;Andrea J. Walencewicz;Charles G. Glabe.
The Journal of Neuroscience (1993)
Common variants at MS4A4/MS4A6E , CD2AP , CD33 and EPHA1 are associated with late-onset Alzheimer's disease
Adam C. Naj;Gyungah Jun;Gary W. Beecham;Li-San Wang.
Nature Genetics (2011)
Apoptosis is induced by beta-amyloid in cultured central nervous system neurons
Deryk T. Loo;Agata Copani;Christian J. Pike;Edward R. Whittemore.
Proceedings of the National Academy of Sciences of the United States of America (1993)
Distribution of N-methyl-D-aspartate-sensitive L-[3H]glutamate-binding sites in rat brain
Daniel T Monaghan;C. W. Cotman.
The Journal of Neuroscience (1985)
Assembly and aggregation properties of synthetic Alzheimer's A4/beta amyloid peptide analogs.
Debra Burdick;Brian Soreghan;Michael Kwon;Joseph Kosmoski.
Journal of Biological Chemistry (1992)
Profile was last updated on December 6th, 2021.
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