David H. Cribbs focuses on Alzheimer's disease, Immunology, Neuroscience, Microglia and Antibody. His studies deal with areas such as Common disease-common variant, Minor allele frequency, Hippocampus and Shock as well as Alzheimer's disease. His research investigates the link between Neuroscience and topics such as Synaptic plasticity that cross with problems in Neurotransmitter receptor and Synapse.
David H. Cribbs combines subjects such as Senile plaques, Amyloid, Complement system, Classical complement pathway and Pathology with his study of Microglia. His Pathology research is multidisciplinary, relying on both Caspase, Cell culture and Cell biology. The Antibody study combines topics in areas such as Molecular biology and Cerebral amyloid angiopathy.
His main research concerns Immunology, Pathology, Antibody, Epitope and Immune system. His study in Pathology concentrates on Cerebral amyloid angiopathy, Amyloid, Alzheimer's disease and Senile plaques. His Alzheimer's disease research integrates issues from Transgene, Hippocampus, Neuroscience and Microglia.
His work is dedicated to discovering how Senile plaques, Cell biology are connected with Receptor and Cell culture and other disciplines. His Antibody study combines topics from a wide range of disciplines, such as Genetically modified mouse and Peptide. His Epitope study integrates concerns from other disciplines, such as Molecular biology, Amyloid beta, B cell and Virology.
His primary areas of investigation include Antibody, Pathology, Genetically modified mouse, Immunology and Epitope. His Antibody study incorporates themes from Cancer research and Disease. His Pathology research incorporates themes from Stroke, Blood pressure and Blood vessel.
His work deals with themes such as Vaccination, Virology, DNA vaccination and B cell, which intersect with Epitope. His study in Anesthesia is interdisciplinary in nature, drawing from both Alzheimer's disease and Amyloid beta. His Alzheimer's disease research is multidisciplinary, incorporating elements of Genetics and Genome-wide association study.
David H. Cribbs mainly focuses on Genetically modified mouse, Blood–brain barrier, Pathology, Genome-wide association study and Genetics. His Genetically modified mouse research includes themes of Epitope, Fusion protein, Transferrin receptor, Monoclonal antibody and Tauopathy. His Transferrin receptor research incorporates elements of Tumor necrosis factor alpha, Etanercept and Immunology.
In the field of Pathology, his study on Cognitive decline overlaps with subjects such as Nephrectomy. His Genome-wide association study research is multidisciplinary, incorporating perspectives in Apolipoprotein E, Allele, Locus and Genetic architecture. The various areas that David H. Cribbs examines in his Genetics study include SNP, TREM2, Case-control study and Linkage disequilibrium.
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