What is he best known for?
The fields of study he is best known for:
- Gene
- Internal medicine
- Alzheimer's disease
His main research concerns Alzheimer's disease, Neuroscience, Genetically modified mouse, Disease and Internal medicine.
His studies deal with areas such as Autophagy, Immunology, Phosphorylation, Cognitive decline and Hippocampus as well as Alzheimer's disease.
Salvatore Oddo applies his multidisciplinary studies on Neuroscience and Tangle in his research.
His Genetically modified mouse study typically links adjacent topics like Pathology.
His Disease research focuses on subjects like Transgene, which are linked to Progressive neurodegenerative disorder, Aβ oligomers, Tau pathology and Fibril.
His Internal medicine research incorporates themes from Endocrinology and Effects of sleep deprivation on cognitive performance.
His most cited work include:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3242 citations)
- Triple-Transgenic Model of Alzheimer's Disease with Plaques and Tangles: Intracellular Aβ and Synaptic Dysfunction (2978 citations)
- Intracellular amyloid-β in Alzheimer's disease (1365 citations)
What are the main themes of his work throughout his whole career to date?
His primary scientific interests are in Neuroscience, Alzheimer's disease, Disease, Genetically modified mouse and Pathology.
His Neuroscience research integrates issues from Autophagy, PI3K/AKT/mTOR pathway, Pathogenesis and Cognitive decline.
His Alzheimer's disease study integrates concerns from other disciplines, such as Endocrinology, Hippocampus and Amyloid.
The various areas that he examines in his Disease study include Cancer research, Transgenic Model, Microglia, Intracellular and Necroptosis.
His Genetically modified mouse research is multidisciplinary, relying on both Phenotype, Frontotemporal lobar degeneration, Tau protein and Endogeny.
His Amyloid precursor protein research includes themes of Neuropathology and Phosphorylation, Cell biology.
He most often published in these fields:
- Neuroscience (56.76%)
- Alzheimer's disease (33.78%)
- Disease (22.97%)
What were the highlights of his more recent work (between 2016-2020)?
- Neuroscience (56.76%)
- Disease (22.97%)
- Pathology (24.32%)
In recent papers he was focusing on the following fields of study:
His primary areas of investigation include Neuroscience, Disease, Pathology, Insulin and Necroptosis.
His study brings together the fields of Neurodegeneration and Neuroscience.
His study in Disease is interdisciplinary in nature, drawing from both Autophagy, Clinical trial, Transgene, Cognition and Intensive care medicine.
Salvatore Oddo has included themes like Inflammation, Microglia, Choline and Choline chloride in his Pathology study.
His Inflammation research integrates issues from Entorhinal cortex, Hippocampus and Genetically modified mouse.
His Amyloid course of study focuses on DYRK1A and Pathogenesis, Neurofibrillary tangle, Biochemistry of Alzheimer's disease and Alzheimer's disease.
Between 2016 and 2020, his most popular works were:
- Necroptosis activation in Alzheimer's disease (120 citations)
- p62 improves AD-like pathology by increasing autophagy. (68 citations)
- Temporal and regional progression of Alzheimer's disease-like pathology in 3xTg-AD mice. (47 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Internal medicine
- Cancer
Salvatore Oddo mostly deals with Pathology, Neuroscience, Transgene, Alzheimer's disease and Amyloid.
His work carried out in the field of Pathology brings together such families of science as Autophagy, Schizophrenia and In vivo.
His biological study spans a wide range of topics, including mTORC1, PI3K/AKT/mTOR pathway, Neuron death, RAC1 and Disease.
Salvatore Oddo is interested in Genetically modified mouse, which is a branch of Transgene.
His Alzheimer's disease research is multidisciplinary, incorporating perspectives in DYRK1A, Amyloid beta and Pathogenesis.
His studies in Amyloid integrate themes in fields like Inflammation, Entorhinal cortex and Phosphorylation.
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