World's Best Scientists 2026 revealed!

D-Index & Metrics

Neuroscience

D-Index
68
Citations
33762
World Ranking
2696
National Ranking
1263

Overview

Kim N. Green is affiliated with the University of California, Irvine in the United States. Their research primarily focuses on neuroscience and medicine, with significant contributions in subfields such as neurology, physiology, molecular biology, immunology, and cellular and molecular neuroscience.

The main topics covered in their work include:

  • Neuroinflammation and Neurodegeneration Mechanisms
  • Alzheimer's disease research and treatments
  • Immune cells in cancer
  • Tryptophan and brain disorders
  • Neuroscience and Neuropharmacology Research
  • Neurological Disease Mechanisms and Treatments
  • Nuclear Receptors and Signaling

Kim N. Green has co-authored publications with several frequent collaborators, including:

  • Grant R. MacGregor
  • Andrea J. Tenner
  • Frank M. LaFerla
  • Shimako Kawauchi
  • Celia Da Cunha

Their work has appeared extensively in various scientific venues, notably:

  • Alzheimer s & Dementia (26 publications)
  • bioRxiv (Cold Spring Harbor Laboratory) (10 publications)
  • Glia (4 publications)
  • Nature Communications (2 publications)
  • Science Advances (2 publications)

Recent publications authored or co-authored by Kim N. Green include:

  • To Kill a Microglia: A Case for CSF1R Inhibitors, 2020, Trends in Immunology
  • Systematic Phenotyping and Characterization of the 5xFAD Mouse Model of Alzheimer's Disease, 2021, Scientific Data
  • Systematic Phenotyping and Characterization of the 3xTg-AD Mouse Model of Alzheimer's Disease, 2022, Frontiers in Neuroscience

Best Publications

  • Guidelines for the use and interpretation of assays for monitoring autophagy

    Daniel J. Klionsky;Fabio C. Abdalla;Hagai Abeliovich;Robert T. Abraham

  • Intracellular amyloid-β in Alzheimer's disease

    Frank M. LaFerla;Kim N. Green;Salvatore Oddo

  • Colony-Stimulating Factor 1 Receptor Signaling Is Necessary for Microglia Viability, Unmasking a Microglia Progenitor Cell in the Adult Brain

    Monica Renee Pittman Elmore;Allison Rachel Najafi;Maya Allegra Koike;Nabil Nazih Dagher

  • Intraneuronal Aβ Causes the Onset of Early Alzheimer’s Disease-Related Cognitive Deficits in Transgenic Mice

    Lauren M. Billings;Salvatore Oddo;Kim N. Green;James L. McGaugh

  • Neural stem cells improve cognition via BDNF in a transgenic model of Alzheimer disease.

    Mathew Blurton-Jones;Masashi Kitazawa;Hilda Martinez-Coria;Nicholas A. Castello

  • Sustained microglial depletion with CSF1R inhibitor impairs parenchymal plaque development in an Alzheimer's disease model.

    Elizabeth Spangenberg;Paul L. Severson;Lindsay A. Hohsfield;Joshua Crapser

  • Glucocorticoids increase amyloid-beta and tau pathology in a mouse model of Alzheimer's disease.

    Kim N. Green;Lauren M. Billings;Benno Roozendaal;James L. McGaugh

  • Lipopolysaccharide-Induced Inflammation Exacerbates Tau Pathology by a Cyclin-Dependent Kinase 5-Mediated Pathway in a Transgenic Model of Alzheimer's Disease

    Masashi Kitazawa;Salvatore Oddo;Tritia R. Yamasaki;Kim N. Green

  • Eliminating microglia in Alzheimer’s mice prevents neuronal loss without modulating amyloid-β pathology

    Elizabeth E. Spangenberg;Rafael J. Lee;Allison R. Najafi;Rachel A. Rice

  • Animal Models of Alzheimer Disease

    Frank M. LaFerla;Kim N. Green

  • Colony-stimulating factor 1 receptor inhibition prevents microglial plaque association and improves cognition in 3xTg-AD mice

    Nabil N. Dagher;Allison R. Najafi;Kara M. Neely Kayala;Monica R. P. Elmore

  • M1 Receptors Play a Central Role in Modulating AD-like Pathology in Transgenic Mice

    Antonella Caccamo;Salvatore Oddo;Lauren M. Billings;Kim N. Green

  • Nicotinamide restores cognition in Alzheimer's disease transgenic mice via a mechanism involving sirtuin inhibition and selective reduction of Thr231-phosphotau.

    Kim N. Green;Joan S. Steffan;Hilda Martinez-Coria;Xuemin Sun

  • Necroptosis activation in Alzheimer's disease

    Antonella Caccamo;Caterina Branca;Ignazio S. Piras;Eric Ferreira

  • Aβ inhibits the proteasome and enhances amyloid and tau accumulation

    Bertrand P. Tseng;Kim N. Green;Julie L. Chan;Mathew Blurton-Jones

  • IKK phosphorylates Huntingtin and targets it for degradation by the proteasome and lysosome

    Leslie Michels Thompson;Charity T. Aiken;Linda S. Kaltenbach;Namita Agrawal

  • Systematic phenotyping and characterization of the 5xFAD mouse model of Alzheimer's disease.

    Stefania Forner;Shimako Kawauchi;Gabriela Balderrama-Gutierrez;Enikö A. Kramár

  • Dietary docosahexaenoic acid and docosapentaenoic acid ameliorate amyloid-beta and tau pathology via a mechanism involving presenilin 1 levels.

    Kim N Green;Hilda Martinez-Coria;Hasan Khashwji;Eileen B Hall

  • Linking Calcium to Aβ and Alzheimer's Disease

    Kim N. Green;Frank M. LaFerla

  • Inflammation in Alzheimer's disease: Lessons learned from microglia-depletion models.

    Elizabeth E. Spangenberg;Kim N. Green

  • SERCA pump activity is physiologically regulated by presenilin and regulates amyloid β production

    Kim N. Green;Angelo Demuro;Yama Akbari;Brian D. Hitt

Frequent Co-Authors

Marcelo A. Wood
Marcelo A. Wood University of California, Irvine
Salvatore Oddo
Salvatore Oddo Arizona State University
Mathew Blurton-Jones
Mathew Blurton-Jones University of California, Irvine
Antonella Caccamo
Antonella Caccamo University of Messina
Enikö A. Kramár
Enikö A. Kramár University of California, Irvine
James L. McGaugh
James L. McGaugh University of California, Irvine
Aileen J. Anderson
Aileen J. Anderson University of California, Irvine
Gareth R. Howell
Gareth R. Howell The Jackson Laboratory
Bruce T. Lamb
Bruce T. Lamb Indiana University
Frances M. Leslie
Frances M. Leslie University of California, Irvine

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