What is he best known for?
The fields of study he is best known for:
- Internal medicine
- Genetics
- Neuron
His primary areas of investigation include Microglia, Neuroscience, Immunology, Visual cortex and Synaptic plasticity.
Microglia is the subject of his research, which falls under Inflammation.
His works in Neuroplasticity, Chronic stress, Dendritic spine, Central nervous system and Oligodendrocyte are all subjects of inquiry into Neuroscience.
The study incorporates disciplines such as Long-term potentiation and Anatomy in addition to Dendritic spine.
In his research, Molecular biology is intimately related to Cell biology, which falls under the overarching field of Immunology.
His Visual cortex course of study focuses on Sensory system and Sensory loss, Apposition, Synaptic pruning, Extracellular and Parenchyma.
His most cited work include:
- Microglial interactions with synapses are modulated by visual experience. (882 citations)
- The Role of Microglia in the Healthy Brain (630 citations)
- Inefficient clearance of myelin debris by microglia impairs remyelinating processes (249 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of study are Microglia, Neuroscience, Cell biology, Immune system and Central nervous system.
His research in Microglia intersects with topics in Dendritic spine and Neuroinflammation.
His work on Chronic stress as part of his general Neuroscience study is frequently connected to Cognitive decline, thereby bridging the divide between different branches of science.
His Cell biology research is multidisciplinary, relying on both Ultrastructure and Cell type.
His Immune system research integrates issues from Receptor and Antigen.
His research investigates the connection with Synaptic plasticity and areas like Visual cortex which intersect with concerns in Metaplasticity.
He most often published in these fields:
- Microglia (70.47%)
- Neuroscience (65.10%)
- Cell biology (20.81%)
What were the highlights of his more recent work (between 2019-2021)?
- Neuroscience (65.10%)
- Microglia (70.47%)
- Immune system (24.16%)
In recent papers he was focusing on the following fields of study:
The scientist’s investigation covers issues in Neuroscience, Microglia, Immune system, Neurodegeneration and Neuroinflammation.
His Neuroscience research is multidisciplinary, incorporating elements of Disease and Depolarization.
His Microglia study improves the overall literature in Internal medicine.
His Immune system research incorporates themes from Fluorescence microscope, Immunostaining and Microscopy.
The various areas that Marie-Ève Tremblay examines in his Neurodegeneration study include Parkinson's disease, Alpha-synuclein, TREM2, Gliosis and Cell biology.
The concepts of his Neuroinflammation study are interwoven with issues in Glutamate homeostasis, Bioinformatics, Glutamate receptor, Innate immune system and Comorbidity.
Between 2019 and 2021, his most popular works were:
- Impact of TREM2R47H variant on tau pathology–induced gliosis and neurodegeneration (33 citations)
- Impact of TREM2R47H variant on tau pathology–induced gliosis and neurodegeneration (33 citations)
- An antibody for analysis of autophagy induction (15 citations)
In his most recent research, the most cited papers focused on:
- Internal medicine
- Genetics
- Neuron
Marie-Ève Tremblay spends much of his time researching Microglia, Neuroscience, Central nervous system, Immune system and Neurodegeneration.
His Microglia research entails a greater understanding of Immunology.
His work in the fields of Neuroscience, such as Gliosis, intersects with other areas such as Context.
His Central nervous system study integrates concerns from other disciplines, such as Synaptic plasticity, Ultrastructure, Nervous system, Schizophrenia and Disease.
He has researched Immune system in several fields, including Neuroinflammation, Chronic stress and Neurotropism.
Marie-Ève Tremblay combines subjects such as Striatum, Pathogenesis, Cerebral cortex, Dendritic spine and Huntington's disease with his study of Neurodegeneration.
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