What is he best known for?
The fields of study he is best known for:
- Gene
- Internal medicine
- Immune system
Richard M. Ransohoff mainly focuses on Immunology, Microglia, Multiple sclerosis, Neuroscience and Chemokine.
Richard M. Ransohoff regularly ties together related areas like Central nervous system in his Immunology studies.
His work deals with themes such as CX3CR1, Mononuclear phagocyte system, Alzheimer's disease and Myeloid, which intersect with Microglia.
His biological study spans a wide range of topics, including Peripheral blood mononuclear cell, Magnetic resonance imaging and Pathology.
His Neuroscience study combines topics from a wide range of disciplines, such as Neuroinflammation, Signal transduction, Neurodegeneration and Transcriptome.
His Neuroinflammation research includes elements of Phenotype and Innate immune system.
His most cited work include:
- Axonal transection in the lesions of multiple sclerosis. (3395 citations)
- Neuroinflammation in Alzheimer's disease (2213 citations)
- The Many Roles of Chemokines and Chemokine Receptors in Inflammation (1941 citations)
What are the main themes of his work throughout his whole career to date?
His scientific interests lie mostly in Immunology, Multiple sclerosis, Chemokine, Microglia and Pathology.
His Immunology study frequently links to related topics such as Central nervous system.
His Multiple sclerosis study combines topics in areas such as Myelin, Internal medicine, Disease and Immune system.
Richard M. Ransohoff studied Chemokine and Cell biology that intersect with Blood–brain barrier and Astrocyte.
Richard M. Ransohoff has researched Microglia in several fields, including CX3CR1, Neuroscience and Neurodegeneration.
His work carried out in the field of Pathology brings together such families of science as Magnetic resonance imaging and Macrophage.
He most often published in these fields:
- Immunology (49.54%)
- Multiple sclerosis (24.03%)
- Chemokine (24.58%)
What were the highlights of his more recent work (between 2015-2021)?
- Microglia (19.22%)
- Immunology (49.54%)
- Neuroscience (17.38%)
In recent papers he was focusing on the following fields of study:
His primary areas of investigation include Microglia, Immunology, Neuroscience, Neuroinflammation and Cell biology.
His Microglia study integrates concerns from other disciplines, such as CX3CR1, Transcriptome, Central nervous system, Receptor and Cell type.
His works in Immune system, Multiple sclerosis, Neuromyelitis optica, Experimental autoimmune encephalomyelitis and Pathogen are all subjects of inquiry into Immunology.
His study looks at the relationship between Multiple sclerosis and topics such as Internal medicine, which overlap with Endocrinology.
His Neuroscience research is multidisciplinary, incorporating elements of Inflammation, Amyotrophic lateral sclerosis, Downregulation and upregulation and Gene expression profiling.
His studies in Neuroinflammation integrate themes in fields like TREM2, Genetically modified mouse and Neurodegeneration.
Between 2015 and 2021, his most popular works were:
- How neuroinflammation contributes to neurodegeneration. (724 citations)
- A polarizing question: do M1 and M2 microglia exist? (648 citations)
- An environment-dependent transcriptional network specifies human microglia identity (474 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Internal medicine
- Immune system
His main research concerns Microglia, Neuroscience, Immunology, Neuroinflammation and Central nervous system.
He has included themes like CCL2, CCR2, Bioinformatics, Monocyte and Proinflammatory cytokine in his Microglia study.
His research in Neuroscience intersects with topics in Inflammation, Receptor, Transcriptome and Cell type.
His study connects Induced pluripotent stem cell and Immunology.
The concepts of his Neuroinflammation study are interwoven with issues in Myeloid, Neurodegeneration, TREM2, Alzheimer's disease and Traumatic brain injury.
Within one scientific family, he focuses on topics pertaining to Disease under Central nervous system, and may sometimes address concerns connected to CX3CR1 and Pathogenesis.
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