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Neuroscience

D-Index
73
Citations
50311
World Ranking
2156
National Ranking
1024

Overview

Beth Stevens is affiliated with Boston Children's Hospital in the United States. The main fields of their research include Neuroscience and Medicine, with a substantial focus on Neurology, Immunology, Molecular Biology, Cellular and Molecular Neuroscience, and Physiology as notable subfields of study.

Their work extensively covers topics such as Neuroinflammation and Neurodegeneration Mechanisms, Immune cells in cancer, Alzheimer's disease research and treatments, Neuroscience and Neuropharmacology Research, Neurogenesis and neuroplasticity mechanisms, Single-cell and spatial transcriptomics, and Genetic Neurodegenerative Diseases.

The following recent papers reflect some of the key contributions made by this scientist:

  • Microglia states and nomenclature: A field at its crossroads, 2022, Neuron
  • Dissection of artifactual and confounding glial signatures by single-cell sequencing of mouse and human brain, 2022, Nature Neuroscience
  • Local externalization of phosphatidylserine mediates developmental synaptic pruning by microglia, 2020, The EMBO Journal
  • Overexpression of schizophrenia susceptibility factor human complement C4A promotes excessive synaptic loss and behavioral changes in mice, 2020, Nature Neuroscience
  • GABA-receptive microglia selectively sculpt developing inhibitory circuits, 2021, Cell

Frequent coauthors in their publications include Martine Therrien, Samuel E. Marsh, Alec J. Walker, Evan Z. Macosko, and Nader Morshed. This collaboration network indicates an active engagement with peers specializing in related aspects of neuroscience and immunology.

The venues where this scientist has published most frequently include bioRxiv (Cold Spring Harbor Laboratory), Alzheimer's & Dementia, Neuron, Nature Neuroscience, and The EMBO Journal. These publication venues underline a strong presence in both pre-publication archives and high-impact peer-reviewed journals.

Best Publications

  • Neurotoxic reactive astrocytes are induced by activated microglia

    Shane A. Liddelow;Kevin A. Guttenplan;Laura E. Clarke;Frederick C. Bennett

  • Microglia sculpt postnatal neural circuits in an activity and complement-dependent manner.

    Dorothy P. Schafer;Emily K. Lehrman;Amanda G. Kautzman;Ryuta Koyama

  • The classical complement cascade mediates CNS synapse elimination.

    Beth Stevens;Nicola J. Allen;Luis E. Vazquez;Gareth R. Howell

  • Complement and microglia mediate early synapse loss in Alzheimer mouse models

    Soyon Hong;Victoria F. Beja-Glasser;Bianca M. Nfonoyim;Arnaud Frouin

  • Schizophrenia risk from complex variation of complement component 4

    Aswin Sekar;Aswin Sekar;Allison R. Bialas;Heather de Rivera;Heather de Rivera;Avery Davis;Avery Davis

  • Single-Cell RNA Sequencing of Microglia throughout the Mouse Lifespan and in the Injured Brain Reveals Complex Cell-State Changes.

    Timothy R. Hammond;Timothy R. Hammond;Timothy R. Hammond;Connor Dufort;Lasse Dissing-Olesen;Lasse Dissing-Olesen;Lasse Dissing-Olesen;Stefanie Giera;Stefanie Giera

  • Microglia states and nomenclature: A field at its crossroads

    Unknown

  • Microglia emerge as central players in brain disease.

    Michael W Salter;Beth Stevens

  • The complement system: an unexpected role in synaptic pruning during development and disease.

    Alexander H. Stephan;Ben A. Barres;Beth Stevens

  • The Role of Microglia in the Healthy Brain

    Marie-Ève Tremblay;Beth Stevens;Amanda Sierra;Hiroaki Wake

  • Astrocytes Promote Myelination in Response to Electrical Impulses

    Tomoko Ishibashi;Kelly A. Dakin;Beth Stevens;Philip R. Lee

  • Adenosine: a neuron-glial transmitter promoting myelination in the CNS in response to action potentials.

    Beth Stevens;Stefania Porta;Laurel L. Haak;Vittorio Gallo

  • Microglia: Dynamic Mediators of Synapse Development and Plasticity.

    Yuwen Wu;Yuwen Wu;Lasse Dissing-Olesen;Lasse Dissing-Olesen;Brian A. MacVicar;Beth Stevens;Beth Stevens

  • TGF-β Signaling Regulates Neuronal C1q Expression and Developmental Synaptic Refinement

    Allison R Bialas;Beth Stevens;Beth Stevens

  • The "quad-partite" synapse: microglia-synapse interactions in the developing and mature CNS.

    Dorothy P. Schafer;Emily K. Lehrman;Beth Stevens;Beth Stevens

  • A complement–microglial axis drives synapse loss during virus-induced memory impairment

    Michael J. Vasek;Charise Garber;Denise Dorsey;Douglas M. Durrant;Douglas M. Durrant

  • New insights on the role of microglia in synaptic pruning in health and disease.

    Soyon Hong;Lasse Dissing-Olesen;Beth Stevens

  • Complement C3 deficiency protects against neurodegeneration in aged plaque-rich APP/PS1 mice

    Qiaoqiao Shi;Qiaoqiao Shi;Saba Chowdhury;Rong Ma;Kevin X. Le

  • Dissection of artifactual and confounding glial signatures by single-cell sequencing of mouse and human brain

    Unknown

  • Molecular clustering identifies complement and endothelin induction as early events in a mouse model of glaucoma

    Gareth R. Howell;Danilo G. Macalinao;Gregory L. Sousa;Michael Walden

  • ATP: an extracellular signaling molecule between neurons and glia

    R.Douglas Fields;Beth Stevens

  • PL-03-01: INGE GRUNDKE-IQBAL AWARD FOR ALZHEIMER'S RESEARCH: NEUROTOXIC REACTIVE ASTROCYTES ARE INDUCED BY ACTIVATED MICROGLIA

    Shane A. Liddelow

Frequent Co-Authors

Ben A. Barres
Ben A. Barres Stanford University
Dorothy P. Schafer
Dorothy P. Schafer University of Massachusetts Chan Medical School
Steven A. McCarroll
Steven A. McCarroll Harvard University
R. Douglas Fields
R. Douglas Fields National Institutes of Health
Cagla Eroglu
Cagla Eroglu Duke University
Brian A. MacVicar
Brian A. MacVicar University of British Columbia
Venkatesh N. Murthy
Venkatesh N. Murthy Harvard University
Michael E. Greenberg
Michael E. Greenberg Harvard University
Gareth R. Howell
Gareth R. Howell The Jackson Laboratory
Mark J. Daly
Mark J. Daly Massachusetts General Hospital

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