Her primary areas of study are Neuroscience, Microglia, Synapse, Synaptic pruning and Complement system. As part of her studies on Neuroscience, Beth Stevens frequently links adjacent subjects like Cellular differentiation. Her studies deal with areas such as Neuroinflammation, Neuroplasticity and Immune system as well as Microglia.
Her Synapse research is multidisciplinary, relying on both Immunology and Neurotransmission. Her biological study spans a wide range of topics, including Complement component 4 and Major histocompatibility complex. Her Complement system research is multidisciplinary, incorporating elements of Regulator, Transforming growth factor and Downregulation and upregulation.
Beth Stevens mainly focuses on Neuroscience, Microglia, Synapse, Synaptic pruning and Cell biology. She combines subjects such as Complement system, Immune system and Disease with her study of Neuroscience. Her Microglia study necessitates a more in-depth grasp of Immunology.
The Synapse study combines topics in areas such as Synaptic plasticity, Biological neural network, Premovement neuronal activity and In vivo. Beth Stevens focuses mostly in the field of Synaptic pruning, narrowing it down to matters related to Complement component 4 and, in some cases, C4A. Her work on Kinase, MAPK/ERK pathway and Schwann cell as part of general Cell biology study is frequently connected to Chemistry and Expression, therefore bridging the gap between diverse disciplines of science and establishing a new relationship between them.
Beth Stevens mostly deals with Neuroscience, Microglia, Synapse, Synaptic pruning and Chemistry. Her Neuroscience study integrates concerns from other disciplines, such as Disease and Pathogenesis. Her Microglia research integrates issues from Cell, Cell type, Receptor, Neuroinflammation and Myelin.
Her research integrates issues of Biological neural network, Excitatory postsynaptic potential, Premovement neuronal activity and In vivo in her study of Synapse. Synaptic pruning is closely attributed to Complement component 4 in her research. In her study, Computational biology and C4A is inextricably linked to Complement, which falls within the broad field of Complement component 4.
Beth Stevens spends much of her time researching Neuroscience, Microglia, Synapse, Synaptic pruning and Receptor. The concepts of her Neuroscience study are interwoven with issues in Cell, Cell type, Immune system, Disease and In vivo. Her Cell type research includes elements of Premovement neuronal activity, Astrocyte, Neuron and Nervous system.
Her Microglia research incorporates elements of Neurogenesis, Multiple sclerosis, Olfactory bulb and Excitatory postsynaptic potential. The Synapse study which covers Biological neural network that intersects with Neural stem cell, Myelin and Thalamus. Her studies deal with areas such as Hippocampal formation, TREM2, Annexin, Cell biology and Parenchyma as well as Receptor.
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Neurotoxic reactive astrocytes are induced by activated microglia
Shane A. Liddelow;Kevin A. Guttenplan;Laura E. Clarke;Frederick C. Bennett.
Microglia sculpt postnatal neural circuits in an activity and complement-dependent manner.
Dorothy P. Schafer;Emily K. Lehrman;Amanda G. Kautzman;Ryuta Koyama.
The classical complement cascade mediates CNS synapse elimination.
Beth Stevens;Nicola J. Allen;Luis E. Vazquez;Gareth R. Howell.
Complement and microglia mediate early synapse loss in Alzheimer mouse models
Soyon Hong;Victoria F. Beja-Glasser;Bianca M. Nfonoyim;Arnaud Frouin.
Schizophrenia risk from complex variation of complement component 4
Aswin Sekar;Aswin Sekar;Allison R. Bialas;Heather de Rivera;Heather de Rivera;Avery Davis;Avery Davis.
The Role of Microglia in the Healthy Brain
Marie-Ève Tremblay;Beth Stevens;Amanda Sierra;Hiroaki Wake.
The Journal of Neuroscience (2011)
Astrocytes Promote Myelination in Response to Electrical Impulses
Tomoko Ishibashi;Kelly A. Dakin;Beth Stevens;Philip R. Lee.
Microglia emerge as central players in brain disease.
Michael W Salter;Beth Stevens.
Nature Medicine (2017)
The complement system: an unexpected role in synaptic pruning during development and disease.
Alexander H. Stephan;Ben A. Barres;Beth Stevens.
Annual Review of Neuroscience (2012)
Adenosine: a neuron-glial transmitter promoting myelination in the CNS in response to action potentials.
Beth Stevens;Stefania Porta;Laurel L. Haak;Vittorio Gallo.
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