What is he best known for?
The fields of study he is best known for:
- Enzyme
- Internal medicine
- Neuron
His primary areas of investigation include Microglia, Inflammation, Cell biology, Internal medicine and Alzheimer's disease.
His Microglia research is multidisciplinary, incorporating elements of Proinflammatory cytokine, Receptor, Phagocytosis and Neuroscience.
The study incorporates disciplines such as Neuroprotection and Pathology in addition to Inflammation.
His work in Cell biology addresses issues such as Senile plaques, which are connected to fields such as Coated vesicle.
His work in Internal medicine tackles topics such as Endocrinology which are related to areas like Peroxisome proliferator-activated receptor.
Gary E. Landreth combines subjects such as Apolipoprotein E, Neurodegeneration and Amyloid with his study of Alzheimer's disease.
His most cited work include:
- Inflammation and Alzheimer's disease. (3451 citations)
- Neuroinflammation in Alzheimer's disease (2213 citations)
- ApoE-directed therapeutics rapidly clear β-amyloid and reverse deficits in AD mouse models. (795 citations)
What are the main themes of his work throughout his whole career to date?
Gary E. Landreth mainly investigates Neuroscience, Microglia, Cell biology, Disease and Internal medicine.
His research integrates issues of TREM2, Neurodegeneration and MAPK/ERK pathway in his study of Neuroscience.
His biological study spans a wide range of topics, including Alzheimer's disease, Receptor and Amyloid.
Gary E. Landreth has researched Alzheimer's disease in several fields, including Apolipoprotein E and Amyloid beta.
His work on Signal transduction, Phosphorylation and Tyrosine kinase as part of his general Cell biology study is frequently connected to Cellular differentiation, thereby bridging the divide between different branches of science.
His research in Internal medicine intersects with topics in Endocrinology and Pioglitazone.
He most often published in these fields:
- Neuroscience (32.77%)
- Microglia (27.66%)
- Cell biology (21.70%)
What were the highlights of his more recent work (between 2017-2021)?
- Neuroscience (32.77%)
- Microglia (27.66%)
- Neuroinflammation (17.02%)
In recent papers he was focusing on the following fields of study:
Gary E. Landreth focuses on Neuroscience, Microglia, Neuroinflammation, TREM2 and Cognitive decline.
His work in the fields of Neuroscience, such as Hippocampus, overlaps with other areas such as Autism.
His Microglia research integrates issues from Receptor, Immune system and Amyloid.
The various areas that Gary E. Landreth examines in his Neuroinflammation study include Agonist, Alzheimer's disease, Pharmacology and Pathogenesis.
The Agonist study combines topics in areas such as Cerebral arteries, Apolipoprotein E, Proinflammatory cytokine and Ischemia.
His Inflammation study combines topics from a wide range of disciplines, such as Phagocytosis, Extracellular, Secretion, Neuroprotection and Pathology.
Between 2017 and 2021, his most popular works were:
- The Trem2 R47H variant confers loss-of-function-like phenotypes in Alzheimer's disease (76 citations)
- The Trem2 R47H variant confers loss-of-function-like phenotypes in Alzheimer's disease (76 citations)
- Pharmacological Inhibition of ERK Signaling Rescues Pathophysiology and Behavioral Phenotype Associated with 16p11.2 Chromosomal Deletion in Mice. (34 citations)
In his most recent research, the most cited papers focused on:
His primary scientific interests are in Neuroscience, TREM2, Microscopy, Autism and MAPK/ERK pathway.
His study in Neuroscience is interdisciplinary in nature, drawing from both Receptor and Microglia.
His TREM2 research includes themes of Neuroinflammation and Neurodegeneration.
His Microscopy study combines topics in areas such as Single Molecule Imaging, Point spread, Optical phenomena and Adaptive optics.
His Autism investigation overlaps with Kinase, Neurogenesis, Intellectual disability, Copy-number variation and Chromosomal Deletion.
His MAPK/ERK pathway research is multidisciplinary, incorporating perspectives in Mitogen-activated protein kinase, Corticogenesis, Central nervous system and Intracellular.
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