D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 52 Citations 15,523 86 World Ranking 11856 National Ranking 131

Overview

What is he best known for?

The fields of study Niels Mailand is best known for:

  • Gene
  • Cell cycle
  • DNA repair

Cell biology is closely attributed to Mitosis in his work. His research on Mitosis often connects related areas such as Cell biology. His Genetics study typically links adjacent topics like Histone. Niels Mailand integrates several fields in his works, including Histone and Gene. He brings together Gene and Computational biology to produce work in his papers. Borrowing concepts from DNA, he weaves in ideas under Computational biology. He undertakes interdisciplinary study in the fields of DNA and Eukaryotic DNA replication through his research. Niels Mailand conducts interdisciplinary study in the fields of Eukaryotic DNA replication and DNA replication through his research. Niels Mailand integrates DNA replication with Control of chromosome duplication in his study.

His most cited work include:

  • RNF8 Ubiquitylates Histones at DNA Double-Strand Breaks and Promotes Assembly of Repair Proteins (1034 citations)
  • The ATM–Chk2–Cdc25A checkpoint pathway guards against radioresistant DNA synthesis (972 citations)
  • RNF168 Binds and Amplifies Ubiquitin Conjugates on Damaged Chromosomes to Allow Accumulation of Repair Proteins (778 citations)

What are the main themes of his work throughout his whole career to date

Cell biology is intertwined with Phosphorylation and Mitosis in his research. His research is interdisciplinary, bridging the disciplines of Cell biology and Phosphorylation. His study on Genetics is mostly dedicated to connecting different topics, such as Chromatin. Niels Mailand integrates Chromatin with DNA repair in his study. Niels Mailand merges DNA repair with Homologous recombination in his study. Niels Mailand combines Homologous recombination and DNA damage in his research. His research ties Genome instability and DNA damage together. Niels Mailand integrates several fields in his works, including Gene and RNA. Niels Mailand integrates many fields, such as RNA and Gene, in his works.

Niels Mailand most often published in these fields:

  • Cell biology (95.00%)
  • Genetics (81.25%)
  • Gene (81.25%)

What were the highlights of his more recent work (between 2019-2022)?

  • Cell biology (100.00%)
  • Genetics (88.89%)
  • Gene (77.78%)

In recent works Niels Mailand was focusing on the following fields of study:

His Mitosis research extends to Cell biology, which is thematically connected. In his research, he performs multidisciplinary study on Mitosis and Microtubule. His work often combines Microtubule and Golgi apparatus studies. In his works, Niels Mailand performs multidisciplinary study on Golgi apparatus and Endoplasmic reticulum. While working on this project, Niels Mailand studies both Endoplasmic reticulum and Phosphorylation. He applies his multidisciplinary studies on Phosphorylation and Phosphoprotein in his research. Phosphoprotein is frequently linked to Cell biology in his study. He connects Genetics with Gene duplication in his study. He performs multidisciplinary studies into Gene duplication and Genome in his work.

Between 2019 and 2022, his most popular works were:

  • Proteome dynamics at broken replication forks reveal a distinct ATM-directed repair response suppressing DNA double-strand break ubiquitination (39 citations)
  • FAM111 protease activity undermines cellular fitness and is amplified by gain‐of‐function mutations in human disease (28 citations)
  • The ubiquitin ligase RFWD3 is required for translesion DNA synthesis (26 citations)

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

The ATM-Chk2-Cdc25A checkpoint pathway guards against radioresistant DNA synthesis.

Jacob Falck;Niels Mailand;Randi G. Syljuåsen;Jiri Bartek.
Nature (2001)

1428 Citations

RNF8 Ubiquitylates Histones at DNA Double-Strand Breaks and Promotes Assembly of Repair Proteins

Niels Mailand;Simon Bekker-Jensen;Helene Faustrup;Fredrik Melander.
Cell (2007)

1253 Citations

Rapid destruction of human Cdc25A in response to DNA damage.

Niels Mailand;Jacob Falck;Claudia Lukas;Randi G. Syljuåsen.
Science (2000)

1026 Citations

RNF168 Binds and Amplifies Ubiquitin Conjugates on Damaged Chromosomes to Allow Accumulation of Repair Proteins

Carsten Doil;Niels Mailand;Simon Bekker-Jensen;Patrice Menard.
Cell (2009)

974 Citations

ATR Prohibits Replication Catastrophe by Preventing Global Exhaustion of RPA

Luis Ignacio Toledo;Matthias Altmeyer;Maj-Britt Rask;Claudia Lukas.
Cell (2013)

554 Citations

Regulation of G2/M events by Cdc25A through phosphorylation‐dependent modulation of its stability

Niels Mailand;Alexandre V. Podtelejnikov;Anja Groth;Matthias Mann.
The EMBO Journal (2002)

426 Citations

CDKs Promote DNA Replication Origin Licensing in Human Cells by Protecting Cdc6 from APC/C-Dependent Proteolysis

Niels Mailand;John F.X. Diffley.
Cell (2005)

398 Citations

Centrosome-associated Chk1 prevents premature activation of cyclin-B-Cdk1 kinase.

Alwin Krämer;Niels Mailand;Claudia Lukas;Randi G. Syljuåsen.
Nature Cell Biology (2004)

380 Citations

Assembly and function of DNA double-strand break repair foci in mammalian cells

Simon Bekker-Jensen;Niels Mailand.
DNA Repair (2010)

354 Citations

The Ubiquitin Ligase XIAP Recruits LUBAC for NOD2 Signaling in Inflammation and Innate Immunity

Rune Busk Damgaard;Ueli Nachbur;Ueli Nachbur;Ueli Nachbur;Monica Yabal;Wendy Wei-Lynn Wong;Wendy Wei-Lynn Wong.
Molecular Cell (2012)

347 Citations

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