D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Molecular Biology D-index 79 Citations 50,984 195 World Ranking 625 National Ranking 12

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • DNA
  • Cancer

His scientific interests lie mostly in Cell biology, DNA damage, DNA repair, Cancer research and Cell cycle. His biological study spans a wide range of topics, including Genetics, Replication protein A, Genotoxic Stress, Chromatin and MRN complex. His DNA damage research integrates issues from G2-M DNA damage checkpoint, Carcinogenesis and DNA replication.

Jiri Bartek has researched G2-M DNA damage checkpoint in several fields, including CHEK1 and Checkpoint Kinase 2. He interconnects Mutation and Molecular biology in the investigation of issues within DNA repair. His research in Cancer research intersects with topics in Tumor suppressor gene and Gene, Locus.

His most cited work include:

  • The DNA-damage response in human biology and disease (3401 citations)
  • DNA damage response as a candidate anti-cancer barrier in early human tumorigenesis (2235 citations)
  • Cell-cycle checkpoints and cancer (2109 citations)

What are the main themes of his work throughout his whole career to date?

Jiri Bartek mainly investigates DNA damage, Cell biology, Cancer research, DNA repair and DNA replication. He combines subjects such as Carcinogenesis, Molecular biology, G2-M DNA damage checkpoint and CHEK1 with his study of DNA damage. His Cell biology research incorporates themes from Chromatin, DNA and Cell cycle.

His study in Chromatin is interdisciplinary in nature, drawing from both Histone, Ubiquitin and MDC1. Jiri Bartek has researched Cancer research in several fields, including Cancer cell, Cancer, Breast cancer, Apoptosis and Immunology. His biological study spans a wide range of topics, including Transcription and Homologous recombination.

He most often published in these fields:

  • DNA damage (52.71%)
  • Cell biology (49.75%)
  • Cancer research (46.80%)

What were the highlights of his more recent work (between 2018-2021)?

  • Cell biology (49.75%)
  • DNA damage (52.71%)
  • DNA replication (24.63%)

In recent papers he was focusing on the following fields of study:

His main research concerns Cell biology, DNA damage, DNA replication, Cancer research and Cancer. His Cell biology study integrates concerns from other disciplines, such as Plasmon, DNA, DNA repair and Genome instability. Jiri Bartek combines subjects such as Chromatin and Transcription with his study of DNA repair.

His Genome instability research integrates issues from Cell cycle and Cell growth. Jiri Bartek integrates many fields, such as DNA damage and Premature aging, in his works. His Cancer research study combines topics from a wide range of disciplines, such as Cancer cell, Apoptosis, Kinase, Bone marrow and Demethylating agent.

Between 2018 and 2021, his most popular works were:

  • A Deep Learning Framework for Predicting Response to Therapy in Cancer. (31 citations)
  • A Deep Learning Framework for Predicting Response to Therapy in Cancer. (31 citations)
  • The Nucleolus: A Multiphase Condensate Balancing Ribosome Synthesis and Translational Capacity in Health, Aging and Ribosomopathies. (30 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Cancer
  • DNA

His primary areas of study are Cell biology, Cancer research, Cancer, Artificial intelligence and Precision medicine. His research in Cell biology intersects with topics in Carcinogenesis, Transcription, Autophagy and DNA repair. The study incorporates disciplines such as A549 cell, Metabolite, Cytotoxicity, Aldehyde dehydrogenase and In vivo in addition to Cancer research.

His Cancer research incorporates elements of Artificial neural network, Deep learning, Deep neural networks and Response to therapy. His Artificial intelligence research includes elements of Machine learning, Pharmacogenomics and Data mining. Jiri Bartek has included themes like Association rule learning and Process in his Precision medicine study.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

The DNA-damage response in human biology and disease

Stephen P. Jackson;Jiri Bartek.
Nature (2009)

5485 Citations

Cell-cycle checkpoints and cancer

Michael B. Kastan;Jiri Bartek.
Nature (2004)

3357 Citations

DNA damage response as a candidate anti-cancer barrier in early human tumorigenesis

Jirina Bartkova;Zuzana Horejsí;Karen Koed;Alwin Krämer.
Nature (2005)

3051 Citations

The causes and consequences of genetic heterogeneity in cancer evolution.

Rebecca A. Burrell;Nicholas McGranahan;Nicholas McGranahan;Jiri Bartek;Charles Swanton.
Nature (2013)

2129 Citations

Oncogene-induced senescence is part of the tumorigenesis barrier imposed by DNA damage checkpoints

Jirina Bartkova;Nousin Rezaei;Michalis Liontos;Panagiotis Karakaidos.
Nature (2006)

2088 Citations

An oncogene-induced DNA damage model for cancer development.

Thanos D. Halazonetis;Vassilis G. Gorgoulis;Jiri Bartek.
Science (2008)

1897 Citations

Chk1 and Chk2 kinases in checkpoint control and cancer.

Jiri Bartek;Jiri Lukas.
Cancer Cell (2003)

1817 Citations

Human CtIP promotes DNA end resection

Alessandro A. Sartori;Claudia Lukas;Julia Coates;Martin Mistrik.
Nature (2007)

1529 Citations

The ATM-Chk2-Cdc25A checkpoint pathway guards against radioresistant DNA synthesis.

Jacob Falck;Niels Mailand;Randi G. Syljuåsen;Jiri Bartek.
Nature (2001)

1428 Citations

Increased expression of mutant forms of p53 oncogene in primary lung cancer.

R Iggo;K Gatter;J Bartek;D Lane.
The Lancet (1990)

1361 Citations

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