Member of the European Molecular Biology Organization (EMBO)
His main research concerns Nucleotide excision repair, DNA repair, Molecular biology, DNA damage and Xeroderma pigmentosum. His work carried out in the field of Nucleotide excision repair brings together such families of science as Transcription and Helicase. His biological study deals with issues like Transcription factor, which deal with fields such as Transcription Factor TFIIH and Carcinogenesis.
His Molecular biology study incorporates themes from Transcription factor II H and Chromatin remodeling. His DNA damage study integrates concerns from other disciplines, such as Mutation, Chromatin, Poly ADP ribose polymerase, Proliferating cell nuclear antigen and Cell biology. His Xeroderma pigmentosum research incorporates elements of ERCC1, Cancer research, Progeria and Endonuclease.
Wim Vermeulen spends much of his time researching Nucleotide excision repair, DNA repair, Molecular biology, DNA damage and Cell biology. His Transcription factor II H study in the realm of Nucleotide excision repair connects with subjects such as RNA polymerase II. His DNA repair study contributes to a more complete understanding of DNA.
His Molecular biology research is multidisciplinary, incorporating perspectives in Polymerase and Proliferating cell nuclear antigen. His research integrates issues of Mutation, Ubiquitin and DNA mismatch repair in his study of DNA damage. Wim Vermeulen focuses mostly in the field of Cell biology, narrowing it down to topics relating to Chromatin and, in certain cases, Histone.
Wim Vermeulen mainly investigates Nucleotide excision repair, DNA repair, Cell biology, DNA damage and Genetics. His Nucleotide excision repair research includes elements of Xeroderma pigmentosum and Endonuclease. The various areas that Wim Vermeulen examines in his DNA repair study include Ubiquitin ligase complex, Ubiquitin, Transcription and SWI/SNF.
His Cell biology research integrates issues from Chromatin, Molecular biology and RNA splicing, Spliceosome. His DNA damage study incorporates themes from Genome and DNA replication. His research in the fields of Trichothiodystrophy, Gene, Compound heterozygosity and Phenotype overlaps with other disciplines such as Premature aging.
Wim Vermeulen focuses on Nucleotide excision repair, Cell biology, DNA damage, DNA repair and Molecular biology. His research on Nucleotide excision repair concerns the broader Genetics. His Chromatin research extends to Cell biology, which is thematically connected.
His research in DNA damage focuses on subjects like Homologous recombination, which are connected to Cancer research and Xeroderma pigmentosum. His study on Base excision repair is often connected to XRCC1 as part of broader study in DNA repair. His research in Molecular biology tackles topics such as Histone code which are related to areas like Histone methyltransferase, Histone H2A and Histone H1.
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DNA repair helicase: a component of BTF2 (TFIIH) basic transcription factor
Laurent Schaeffer;Richard Roy;Sandrine Humbert;Vincent Moncollin.
Science (1993)
Sequential Assembly of the Nucleotide Excision Repair Factors In Vivo
Marcel Volker;Martijn J Moné;Parimal Karmakar;Anneke van Hoffen.
Molecular Cell (2001)
Understanding nucleotide excision repair and its roles in cancer and ageing
Jurgen A. Marteijn;Hannes Lans;Wim Vermeulen;Jan H. J. Hoeijmakers.
Nature Reviews Molecular Cell Biology (2014)
ERCC6, a member of a subfamily of putative helicases, is involved in Cockayne's syndrome and preferential repair of active genes.
Christine Troelstra;Alain van Gool;Jan de Wit;Wim Vermeulen.
Cell (1992)
A new progeroid syndrome reveals that genotoxic stress suppresses the somatotroph axis
Laura J. Niedernhofer;George A. Garinis;Anja Raams;Astrid S. Lalai.
Nature (2006)
The MO15 cell cycle kinase is associated with the TFIIH transcription-DNA repair factor
Richard Roy;Jörg P. Adamczewski;Thierry Seroz;Wim Vermeulen.
Cell (1994)
A presumed DNA helicase encoded by ERCC-3 is involved in the human repair disorders xeroderma pigmentosum and Cockayne's syndrome.
Geert Weeda;Reinier C.A. van Ham;Wim Vermeulen;Dirk Bootsma.
Cell (1990)
RNF168 Ubiquitinates K13-15 on H2A/H2AX to Drive DNA Damage Signaling
Francesca Mattiroli;Joseph H.A. Vissers;Willem J. van Dijk;Pauline Ikpa.
Cell (2012)
Clasps are CLIP-115 and -170 associating proteins involved in the regional regulation of microtubule dynamics in motile fibroblasts.
Anna Akhmanova;Casper C. Hoogenraad;Ksenija Drabek;Tatiana Stepanova.
Cell (2001)
Cockayne syndrome A and B proteins differentially regulate recruitment of chromatin remodeling and repair factors to stalled RNA polymerase II in vivo.
Maria Fousteri;Wim Vermeulen;Albert A. van Zeeland;Leon H.F. Mullenders.
Molecular Cell (2006)
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