What is he best known for?
The fields of study he is best known for:
His scientific interests lie mostly in DNA repair, Genetics, Nucleotide excision repair, Xeroderma pigmentosum and DNA damage.
His work in Ataxia-telangiectasia, Transcription Factor TFIIH, Gene and Fanconi anemia is related to Genetics.
His Ataxia-telangiectasia study combines topics in areas such as Positional cloning and Ataxia Telangiectasia Mutated Proteins.
The Nucleotide excision repair study combines topics in areas such as Replication protein A and Molecular biology.
His Xeroderma pigmentosum research incorporates elements of Cancer research and ERCC4.
His DNA damage study integrates concerns from other disciplines, such as Mutation, CDC25A and Homologous recombination.
His most cited work include:
- A SINGLE ATAXIA TELANGIECTASIA GENE WITH A PRODUCT SIMILAR TO PI-3 KINASE (2378 citations)
- Molecular mechanism of nucleotide excision repair (960 citations)
- A new progeroid syndrome reveals that genotoxic stress suppresses the somatotroph axis (538 citations)
What are the main themes of his work throughout his whole career to date?
The scientist’s investigation covers issues in Genetics, DNA repair, Molecular biology, Nucleotide excision repair and Xeroderma pigmentosum.
His study in Trichothiodystrophy, Gene, Ataxia-telangiectasia, Mutation and Transcription factor II H is done as part of Genetics.
His work focuses on many connections between DNA repair and other disciplines, such as DNA damage, that overlap with his field of interest in Homologous recombination.
The study incorporates disciplines such as Chinese hamster, DNA, DNA replication, DNA synthesis and Complementation in addition to Molecular biology.
His Nucleotide excision repair research includes elements of Replication protein A, DNA-binding protein and Cell biology.
His research in Xeroderma pigmentosum tackles topics such as Cancer research which are related to areas like Progeria.
He most often published in these fields:
- Genetics (43.31%)
- DNA repair (40.94%)
- Molecular biology (37.80%)
What were the highlights of his more recent work (between 2007-2017)?
- DNA repair (40.94%)
- Xeroderma pigmentosum (33.86%)
- Genetics (43.31%)
In recent papers he was focusing on the following fields of study:
His primary areas of study are DNA repair, Xeroderma pigmentosum, Genetics, Nucleotide excision repair and Molecular biology.
Nicolaas G. J. Jaspers interconnects Cell cycle checkpoint, DNA damage and Transcription factor II E in the investigation of issues within DNA repair.
His work is dedicated to discovering how Xeroderma pigmentosum, Dermatology are connected with Pathology and Disease and other disciplines.
His study in Nucleotide excision repair is interdisciplinary in nature, drawing from both Phenotype, Cancer research, Proliferating cell nuclear antigen and DNA polymerase.
The various areas that Nicolaas G. J. Jaspers examines in his Molecular biology study include ERCC1, Mutation, Postreplication repair, Fetus and DNA replication.
The concepts of his Microcephaly study are interwoven with issues in Ataxia-telangiectasia and ERCC6.
Between 2007 and 2017, his most popular works were:
- Three DNA Polymerases, Recruited by Different Mechanisms, Carry Out NER Repair Synthesis in Human Cells (261 citations)
- SLX4, a coordinator of structure-specific endonucleases, is mutated in a new Fanconi anemia subtype (248 citations)
- Mutations in ERCC4, encoding the DNA-repair endonuclease XPF, cause Fanconi anemia. (232 citations)
In his most recent research, the most cited papers focused on:
His primary scientific interests are in DNA repair, Xeroderma pigmentosum, Genetics, Nucleotide excision repair and Dermatology.
His study looks at the relationship between DNA repair and fields such as DNA damage, as well as how they intersect with chemical problems.
His work deals with themes such as Cerebral atrophy, Disease and Immunosuppression, which intersect with Xeroderma pigmentosum.
His Nucleotide excision repair research is multidisciplinary, incorporating elements of Mutation and Point mutation.
Nicolaas G. J. Jaspers has researched Mutation in several fields, including Molecular biology, Mutant, ERCC4 and ERCC1.
His Dermatology study also includes
- Cockayne syndrome together with Defective DNA repair, Incidence, ERCC8 and Trichothiodystrophy,
- Pathology, which have a strong connection to Nijmegen breakage syndrome, Chromosome Fragility and Ataxia-telangiectasia.
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