His primary scientific interests are in Ataxia-telangiectasia, DNA damage, Cell biology, DNA repair and Genetics. His work deals with themes such as Positional cloning, Cancer research, Mutation, Molecular biology and Ataxia Telangiectasia Mutated Proteins, which intersect with Ataxia-telangiectasia. His Ataxia Telangiectasia Mutated Proteins research includes themes of SH3 domain and Ataxia telangiectasia and Rad3 related.
His DNA damage study integrates concerns from other disciplines, such as Chromatin, Cell cycle, Signal transduction and Homologous recombination. His Cell biology research incorporates elements of Cell cycle checkpoint, Biochemistry and Programmed cell death. His DNA repair research is multidisciplinary, relying on both Carcinogenesis, Genetic disorder and MRN complex.
Martin F. Lavin mostly deals with Molecular biology, Ataxia-telangiectasia, Cell biology, DNA damage and Cancer research. The study incorporates disciplines such as Cell culture, Ionizing radiation, Biochemistry, DNA and Complementary DNA in addition to Molecular biology. His Ataxia-telangiectasia study contributes to a more complete understanding of Genetics.
His Cell biology research is multidisciplinary, incorporating perspectives in Cell cycle and Cell cycle checkpoint. His study looks at the relationship between DNA damage and topics such as DNA repair, which overlap with MRN complex. His Cancer research research includes elements of Radioresistance and Cancer, Prostate cancer.
Martin F. Lavin mainly focuses on DNA damage, Cell biology, Ataxia-telangiectasia, Prostate cancer and Genetics. His DNA damage research incorporates elements of Oxidative stress, Cancer research, Homologous recombination, Molecular biology and DNA repair. In his research, Immunology and Haematopoiesis is intimately related to Cancer, which falls under the overarching field of Cancer research.
His studies deal with areas such as Reprogramming, Induced pluripotent stem cell and MRN complex as well as Cell biology. Martin F. Lavin interconnects Cerebellum, Neuroscience, Immunodeficiency, Neurodegeneration and Pathology in the investigation of issues within Ataxia-telangiectasia. His study on Prostate cancer also encompasses disciplines like
Martin F. Lavin spends much of his time researching DNA damage, Genetics, DNA repair, Ataxia-telangiectasia and Molecular biology. His biological study spans a wide range of topics, including Oxidative stress, Cancer research, Homologous recombination, Immunology and Ataxia Telangiectasia Mutated Proteins. Genetics and Cell biology are commonly linked in his work.
His DNA repair study combines topics in areas such as MRN complex, Signal transduction, Phosphorylation, Autophosphorylation and Phosphoproteomics. He combines subjects such as Phosphoserine, Cytoplasm, Somatic cell, Reprogramming and Neurodegeneration with his study of Ataxia-telangiectasia. As a part of the same scientific family, Martin F. Lavin mostly works in the field of Molecular biology, focusing on DNA repair protein XRCC4 and, on occasion, RAD51, Homologous Recombination Pathway and Radiosensitivity.
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A SINGLE ATAXIA TELANGIECTASIA GENE WITH A PRODUCT SIMILAR TO PI-3 KINASE
Kinneret Savitsky;Anat Bar-Shira;Shlomit Gilad;Galit Rotman.
Science (1995)
Ataxia-telangiectasia: from a rare disorder to a paradigm for cell signalling and cancer
Martin F. Lavin.
Nature Reviews Molecular Cell Biology (2008)
ATM Activation by Oxidative Stress
Zhi Guo;Sergei Kozlov;Martin F. Lavin;Maria D. Person.
Science (2010)
THE GENETIC DEFECT IN ATAXIA-TELANGIECTASIA
Martin F. Lavin;Yosef Shiloh.
Annual Review of Immunology (1997)
The complexity of p53 stabilization and activation.
M F Lavin;M F Lavin;N Gueven.
Cell Death & Differentiation (2006)
ATM associates with and phosphorylates p53: mapping the region of interaction
Kum Kum Khanna.;Katherine E. Keating;Sergei Kozlov;Shaun Scott.
Nature Genetics (1998)
ATM-dependent phosphorylation of nibrin in response to radiation exposure
Magtouf Gatei;David Young;Karen M. Cerosaletti;Ami Desai-Mehta.
Nature Genetics (2000)
Interaction between ATM protein and c-Abl in response to DNA damage
T Shafman;K K Khanna;P Kedar;K Spring.
Nature (1997)
Robot-assisted laparoscopic prostatectomy versus open radical retropubic prostatectomy: early outcomes from a randomised controlled phase 3 study
John W Yaxley;Geoffrey D Coughlin;Suzanne K Chambers;Stefano Occhipinti.
The Lancet (2016)
DNA-dependent protein kinase catalytic subunit: a target for an ICE-like protease in apoptosis.
Q Song;S P Lees-Miller;S Kumar;Z Zhang.
The EMBO Journal (1996)
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