Peter J. McKinnon mainly investigates Cell biology, DNA repair, Molecular biology, DNA damage and Genetics. In his research, p53 upregulated modulator of apoptosis, Mediator, Gene targeting, Knockout mouse and Puma is intimately related to Suppressor, which falls under the overarching field of Cell biology. His work carried out in the field of DNA repair brings together such families of science as Neuropathology, DNA ligase and DNA replication.
His studies in Molecular biology integrate themes in fields like Ectoderm, Forebrain, Wnt signaling pathway and Somite. In general DNA damage, his work in Ataxia-telangiectasia is often linked to Ataxia Telangiectasia Mutated Proteins linking many areas of study. Peter J. McKinnon focuses mostly in the field of Genetics, narrowing it down to topics relating to Neurodegeneration and, in certain cases, Cell, Deoxyribose, DNA, DNA Strand Break and Radiosensitivity.
The scientist’s investigation covers issues in DNA damage, DNA repair, Cancer research, Cell biology and Molecular biology. His study in the field of Ataxia-telangiectasia also crosses realms of Ataxia Telangiectasia Mutated Proteins. The concepts of his DNA repair study are interwoven with issues in Neurodegeneration, Genome instability and Homologous recombination.
His studies deal with areas such as Carcinogenesis, Cancer, Apoptosis and Immunology as well as Cancer research. The study incorporates disciplines such as Suppressor, Gene targeting and Cellular differentiation in addition to Cell biology. His Molecular biology research is multidisciplinary, incorporating perspectives in Aprataxin, DNA ligase, Forebrain and Histone.
Peter J. McKinnon focuses on Cancer research, DNA damage, Cell biology, DNA repair and DNA. His Cancer research study incorporates themes from Cancer, Kinase, Genetics, Histone H3 and Ataxia. His Kinase research incorporates themes from Ataxia-telangiectasia and Apoptosis.
His DNA damage research is multidisciplinary, incorporating elements of Carcinogenesis, PARP inhibitor and Homologous recombination. His Cell biology study combines topics from a wide range of disciplines, such as Mutation, Molecular biology and DNA Strand Break. In his study, DNA-PKcs and G2-M DNA damage checkpoint is inextricably linked to Neurogenesis, which falls within the broad field of DNA repair.
His scientific interests lie mostly in DNA repair, DNA damage, DNA, Genetics and Cell biology. His research investigates the link between DNA repair and topics such as Cancer research that cross with problems in Cerebellum. His work deals with themes such as Nuclear DNA and Transcription, which intersect with DNA damage.
His research investigates the connection between DNA and topics such as Nervous system that intersect with issues in Bioinformatics, Disease, Cell and Genome. Peter J. McKinnon usually deals with Genetics and limits it to topics linked to Neurodegeneration and Molecular neuroscience, Topoisomerase, Mutation, Cellular homeostasis and Ataxia. His work on Neurogenesis as part of general Cell biology study is frequently linked to Ataxia Telangiectasia Mutated Proteins, bridging the gap between disciplines.
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Puma is an essential mediator of p53-dependent and -independent apoptotic pathways
John R. Jeffers;Evan Parganas;Evan Parganas;Youngsoo Lee;Chunying Yang.
Cancer Cell (2003)
Subtypes of medulloblastoma have distinct developmental origins
Paul Gibson;Yiai Tong;Giles Robinson;Margaret C. Thompson.
Requirement for Atm in Ionizing Radiation-Induced Cell Death in the Developing Central Nervous System
Karl-Heinz Herzog;Miriam J. Chong;Manuela Kapsetaki;James I. Morgan.
Phosphorylation of SMC1 is a critical downstream event in the ATM-NBS1-BRCA1 pathway.
Risa Kitagawa;Christopher J. Bakkenist;Peter J. McKinnon;Michael B. Kastan.
Genes & Development (2004)
Six3 repression of Wnt signaling in the anterior neuroectoderm is essential for vertebrate forebrain development
Oleg V. Lagutin;Changqi C. Zhu;Daisuke Kobayashi;Jacek Topczewski.
Genes & Development (2003)
ATM and ataxia telangiectasia.
Peter J McKinnon.
EMBO Reports (2004)
Identification of Early Replicating Fragile Sites that Contribute to Genome Instability
Jacqueline H. Barlow;Robert B. Faryabi;Elsa Callén;Nancy Wong.
The neurodegenerative disease protein aprataxin resolves abortive DNA ligation intermediates
Ivan Ahel;Ulrich Rass;Sherif F. El-Khamisy;Sherif F. El-Khamisy;Sachin Katyal.
Human leukocyte antigen-B-associated transcript 3 is released from tumor cells and engages the NKp30 receptor on natural killer cells.
Elke Pogge von Strandmann;Venkateswara Rao Simhadri;Bastian von Tresckow;Stephanie Sasse.
A mouse model of ATR-Seckel shows embryonic replicative stress and accelerated aging
Matilde Murga;Samuel Bunting;Maria F Montaña;Rebeca Soria.
Nature Genetics (2009)
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