What is he best known for?
The fields of study he is best known for:
His primary areas of investigation include DNA damage, Cell biology, Ataxia-telangiectasia, Ataxia Telangiectasia Mutated Proteins and Genetics.
His biological study spans a wide range of topics, including MRE11 Homologue Protein and Homologous recombination.
His Cell biology research is multidisciplinary, incorporating elements of Chromatin, DNA, DNA repair and Molecular biology.
Yosef Shiloh has researched Ataxia-telangiectasia in several fields, including Mutation, Cancer research and Nibrin.
He combines subjects such as Phenotype and MDC1 with his study of Ataxia Telangiectasia Mutated Proteins.
His Kinase research includes elements of Nuclear protein, Signal transduction, Cellular homeostasis and Phosphorylation.
His most cited work include:
- A SINGLE ATAXIA TELANGIECTASIA GENE WITH A PRODUCT SIMILAR TO PI-3 KINASE (2378 citations)
- ATM and ATR substrate analysis reveals extensive protein networks responsive to DNA damage (2356 citations)
- ATM and related protein kinases: safeguarding genome integrity (2189 citations)
What are the main themes of his work throughout his whole career to date?
Yosef Shiloh focuses on Genetics, DNA damage, Ataxia-telangiectasia, Cell biology and Molecular biology.
His DNA damage study combines topics from a wide range of disciplines, such as Ataxia Telangiectasia Mutated Proteins, Protein kinase A, Phosphorylation and DNA repair.
His Ataxia Telangiectasia Mutated Proteins study typically links adjacent topics like Ataxia telangiectasia and Rad3 related.
The study incorporates disciplines such as Oxidative stress, Cancer research, Phenotype, Mutation and Chromosome instability in addition to Ataxia-telangiectasia.
The Cell biology study combines topics in areas such as Chromatin, Nuclear protein and DNA-binding protein.
His work deals with themes such as Cell culture, Mutant and DNA, which intersect with Molecular biology.
He most often published in these fields:
- Genetics (40.30%)
- DNA damage (38.81%)
- Ataxia-telangiectasia (37.81%)
What were the highlights of his more recent work (between 2009-2021)?
- DNA damage (38.81%)
- Cell biology (33.83%)
- DNA repair (14.43%)
In recent papers he was focusing on the following fields of study:
His scientific interests lie mostly in DNA damage, Cell biology, DNA repair, Genome instability and Genetics.
The various areas that Yosef Shiloh examines in his DNA damage study include Chromatin, Molecular biology, Ataxia Telangiectasia Mutated Proteins and Ubiquitin.
His studies deal with areas such as Nuclear protein, Signal transduction and Cellular homeostasis as well as Ataxia Telangiectasia Mutated Proteins.
Kinase is the focus of his Cell biology research.
His Genome instability study combines topics in areas such as Ataxia-telangiectasia, Neuroscience, Disease and Cerebellar atrophy.
His Ataxia-telangiectasia study integrates concerns from other disciplines, such as Endocrinology, Retinal and Pathology.
Between 2009 and 2021, his most popular works were:
- The ATM protein kinase: regulating the cellular response to genotoxic stress, and more (1000 citations)
- Requirement of ATM-Dependent Monoubiquitylation of Histone H2B for Timely Repair of DNA Double-Strand Breaks (277 citations)
- ATM-Dependent and -Independent Dynamics of the Nuclear Phosphoproteome After DNA Damage (213 citations)
In his most recent research, the most cited papers focused on:
DNA damage, Cell biology, DNA repair, Ataxia Telangiectasia Mutated Proteins and Molecular biology are his primary areas of study.
His study on DNA damage is covered under Genetics.
His studies in Cell biology integrate themes in fields like Promoter, Chromatin immunoprecipitation, Transcriptional regulation, RNA-binding protein and Intron.
His DNA repair research incorporates elements of G2-M DNA damage checkpoint, Mutation, Genome, Genome instability and Signal transduction.
His Ataxia Telangiectasia Mutated Proteins study is associated with Kinase.
His work carried out in the field of Molecular biology brings together such families of science as Cell, Chromatin, Gene expression, Gene and Programmed cell death.
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