World's Best Scientists 2026 revealed!

D-Index & Metrics

Genetics

D-Index
46
Citations
10518
World Ranking
4152
National Ranking
471

Grant S. Stewart publication distribution in Genetics in 2026

The chart shows the distribution of publications by all Research.com ranked scientists in the field of Genetics in 2026. The highlighted bar marks where Grant S. Stewart sits on this spectrum.

45–54 publications: 6 scientists 55–64 publications: 10 scientists 65–74 publications: 35 scientists 75–84 publications: 84 scientists 85–94 publications: 102 scientists 95–104 publications: 151 scientists 105–114 publications: 175 scientists 115–124 publications: 203 scientists 125–134 publications: 217 scientists 135–144 publications: 205 scientists 145–154 publications: 193 scientists 155–164 publications: 188 scientists 165–174 publications: 170 scientists 175–184 publications: 178 scientists 185–194 publications: 164 scientists 195–204 publications: 173 scientists 205–214 publications: 159 scientists 215–224 publications: 134 scientists 225–234 publications: 143 scientists 235–244 publications: 105 scientists 245–254 publications: 114 scientists 255–264 publications: 92 scientists 265–274 publications: 88 scientists 275–284 publications: 87 scientists 285–294 publications: 80 scientists 295–304 publications: 62 scientists 305–314 publications: 75 scientists 315–324 publications: 67 scientists 325–334 publications: 60 scientists 335–344 publications: 52 scientists 345–354 publications: 40 scientists 355–364 publications: 48 scientists 365–374 publications: 47 scientists 375–384 publications: 46 scientists 385–394 publications: 31 scientists 395–404 publications: 27 scientists 405–414 publications: 40 scientists 415–424 publications: 30 scientists 425–434 publications: 43 scientists 435–444 publications: 29 scientists 445–454 publications: 14 scientists 455–464 publications: 28 scientists 465–474 publications: 21 scientists 475–484 publications: 21 scientists 485–494 publications: 22 scientists 495–504 publications: 17 scientists 505–514 publications: 12 scientists 515–524 publications: 11 scientists 525–534 publications: 8 scientists 535–544 publications: 8 scientists 545–554 publications: 14 scientists 555–564 publications: 4 scientists 565–574 publications: 11 scientists 575–584 publications: 5 scientists 585–594 publications: 11 scientists 595–604 publications: 12 scientists 605–614 publications: 7 scientists 615–624 publications: 6 scientists 625–634 publications: 10 scientists 635–644 publications: 9 scientists 645–654 publications: 10 scientists 655–664 publications: 6 scientists 665–674 publications: 6 scientists 675–684 publications: 6 scientists 685–694 publications: 4 scientists 695–702 publications: 6 scientists 703+ publications: 100 scientists
45 publications 703+

This scientist: 108 publications — 11th percentile

11% of scientists in this discipline score the same or lower.

The last bar groups every scientist with 703 publications or more.

Grant S. Stewart D-index placement in Genetics in 2026

The chart shows the D-index (discipline H-index) distribution of Genetics scientists ranked by Research.com in 2026. The highlighted bar marks where Grant S. Stewart sits on this spectrum.

40–41 D-Index: 24 scientists 42–43 D-Index: 52 scientists 44–45 D-Index: 84 scientists 46–47 D-Index: 112 scientists 48–49 D-Index: 118 scientists 50–51 D-Index: 141 scientists 52–53 D-Index: 143 scientists 54–55 D-Index: 145 scientists 56–57 D-Index: 179 scientists 58–59 D-Index: 162 scientists 60–61 D-Index: 175 scientists 62–63 D-Index: 191 scientists 64–65 D-Index: 172 scientists 66–67 D-Index: 184 scientists 68–69 D-Index: 164 scientists 70–71 D-Index: 158 scientists 72–73 D-Index: 150 scientists 74–75 D-Index: 136 scientists 76–77 D-Index: 127 scientists 78–79 D-Index: 127 scientists 80–81 D-Index: 111 scientists 82–83 D-Index: 110 scientists 84–85 D-Index: 110 scientists 86–87 D-Index: 84 scientists 88–89 D-Index: 102 scientists 90–91 D-Index: 66 scientists 92–93 D-Index: 72 scientists 94–95 D-Index: 70 scientists 96–97 D-Index: 54 scientists 98–99 D-Index: 60 scientists 100–101 D-Index: 49 scientists 102–103 D-Index: 55 scientists 104–105 D-Index: 45 scientists 106–107 D-Index: 42 scientists 108–109 D-Index: 28 scientists 110–111 D-Index: 39 scientists 112–113 D-Index: 25 scientists 114–115 D-Index: 31 scientists 116–117 D-Index: 29 scientists 118–119 D-Index: 34 scientists 120–121 D-Index: 29 scientists 122–123 D-Index: 29 scientists 124–125 D-Index: 18 scientists 126–127 D-Index: 27 scientists 128–129 D-Index: 22 scientists 130–131 D-Index: 16 scientists 132–133 D-Index: 11 scientists 134–135 D-Index: 17 scientists 136–137 D-Index: 12 scientists 138–139 D-Index: 21 scientists 140–141 D-Index: 4 scientists 142–143 D-Index: 9 scientists 144–145 D-Index: 14 scientists 146–147 D-Index: 6 scientists 148–149 D-Index: 10 scientists 150–151 D-Index: 7 scientists 152–153 D-Index: 9 scientists 154–155 D-Index: 8 scientists 156–157 D-Index: 8 scientists 158–159 D-Index: 9 scientists 160+ D-Index: 96 scientists
40 D-Index 160+

This scientist: 46 D-Index — 5th percentile

5% of scientists in this discipline score the same or lower.

The last bar groups every scientist with 160 D-Index or more.

Overview

Grant S. Stewart is affiliated with the University of Birmingham in the United Kingdom. Their research spans multiple fields primarily within Biochemistry, Genetics and Molecular Biology, complemented by substantial contributions to Medicine. Specifically, their work engages deeply with subfields such as Molecular Biology, Oncology, Cell Biology, Genetics, and Pulmonary and Respiratory Medicine.

The topics central to Stewart's research focus on mechanisms and pathways related to DNA and cellular processes. Notable areas include DNA Repair Mechanisms, Microtubule and mitosis dynamics, Genomics and Chromatin Dynamics, CRISPR and Genetic Engineering, Ubiquitin and proteasome pathways, Renal cell carcinoma treatment, and PARP inhibition in cancer therapy.

Stewart has a significant body of published work with frequent appearance in leading scientific journals. The primary venues for their publications include Nature Communications, bioRxiv (Cold Spring Harbor Laboratory), Nucleic Acids Research, The Journal of Urology, and the Journal of Clinical Investigation.

Major papers authored or co-authored by Stewart include:

  • Warsaw Breakage Syndrome associated DDX11 helicase resolves G-quadruplex structures to support sister chromatid cohesion, 2020, Nature Communications
  • DONSON and FANCM associate with different replisomes distinguished by replication timing and chromatin domain, 2020, Nature Communications
  • Arginine methylation and ubiquitylation crosstalk controls DNA end-resection and homologous recombination repair, 2021, Nature Communications
  • The structural mechanism of dimeric DONSON in replicative helicase activation, 2023, Molecular Cell
  • Bi-allelic MCM10 variants associated with immune dysfunction and cardiomyopathy cause telomere shortening, 2021, Nature Communications

Throughout their career, Stewart has collaborated extensively with several co-authors, most frequently with Satpal S. Jhujh, John J. Reynolds, Gavin S. McNee, Robert M. Hollingworth, and Martin R. Higgs, indicating a broad network of scientific collaboration.

Best Publications

  • The DNA Double-Strand Break Repair Gene hMRE11 Is Mutated in Individuals with an Ataxia-Telangiectasia-like Disorder

    Grant S Stewart;Richard S Maser;Tanja Stankovic;Debra A Bressan

  • MDC1 is a mediator of the mammalian DNA damage checkpoint

    Grant S. Stewart;Bin Wang;Colin R. Bignell;A. Malcolm R. Taylor

  • The RIDDLE Syndrome Protein Mediates a Ubiquitin-Dependent Signaling Cascade at Sites of DNA Damage

    Grant S. Stewart;Stephanie Panier;Stephanie Panier;Kelly Townsend;Abdallah K. Al-Hakim

  • Inactivation of ataxia telangiectasia mutated gene in B-cell chronic lymphocytic leukaemia

    Tatjana Stankovic;Peter Weber;Grant Stewart;Tina Bedenham

  • 53BP1-dependent robust localized KAP-1 phosphorylation is essential for heterochromatic DNA double-strand break repair

    Angela T Noon;Atsushi Shibata;Nicole Rief;Markus Lobrich

  • p53 dysfunction in B-cell chronic lymphocytic leukemia: inactivation of ATM as an alternative to TP53 mutation.

    Andrew R. Pettitt;Paul D. Sherrington;Grant Stewart;John C. Cawley

  • Constitutive phosphorylation of MDC1 physically links the MRE11–RAD50–NBS1 complex to damaged chromatin

    Christoph Spycher;Edward S. Miller;Kelly Townsend;Lucijana Pavic

  • ATR inhibition induces synthetic lethality and overcomes chemoresistance in TP53- or ATM-defective chronic lymphocytic leukemia cells

    Marwan Kwok;Marwan Kwok;Nicholas Davies;Angelo Agathanggelou;Edward Smith

  • A viral E3 ligase targets RNF8 and RNF168 to control histone ubiquitination and DNA damage responses

    Caroline E Lilley;Mira S Chaurushiya;Mira S Chaurushiya;Chris Boutell;Sebastien Landry

  • Chromosome instability syndromes

    A. Malcolm R. Taylor;Cynthia Rothblum-Oviatt;Nathan A. Ellis;Ian D. Hickson

  • Regulation of DNA-End Resection by hnRNPU-like Proteins Promotes DNA Double-Strand Break Signaling and Repair

    Sophie E. Polo;Andrew N. Blackford;J. Ross Chapman;Linda Baskcomb

  • RIDDLE immunodeficiency syndrome is linked to defects in 53BP1-mediated DNA damage signaling

    Grant S. Stewart;Tatjana Stankovic;Philip J. Byrd;Thomas Wechsler

  • Ataxia telangiectasia mutated-deficient B-cell chronic lymphocytic leukemia occurs in pregerminal center cells and results in defective damage response and unrepaired chromosome damage.

    Tatjana Stankovic;Grant S. Stewart;Christopher Fegan;Paul Biggs

  • BOD1L Is Required to Suppress Deleterious Resection of Stressed Replication Forks.

    Martin R. Higgs;John J. Reynolds;Alicja Winczura;Andrew N. Blackford

  • Human Claspin works with BRCA1 to both positively and negatively regulate cell proliferation

    Shiaw Yih Lin;Kaiyi Li;Grant S. Stewart;Stephen J. Elledge;Stephen J. Elledge

  • PARP1 and PARP2 stabilise replication forks at base excision repair intermediates through Fbh1-dependent Rad51 regulation.

    George E. Ronson;Ann Liza Piberger;Martin R. Higgs;Anna L. Olsen

  • The hMsh2-hMsh6 complex acts in concert with monoubiquitinated PCNA and Pol η in response to oxidative DNA damage in human cells

    Anastasia Zlatanou;Emmanuelle Despras;Tirzah Braz-Petta;Imenne Boubakour-Azzouz

  • Microarray analysis reveals that TP53- and ATM-mutant B-CLLs share a defect in activating proapoptotic responses after DNA damage but are distinguished by major differences in activating prosurvival responses.

    Tatjana Stankovic;Mike Hubank;Debbie Cronin;Grant S. Stewart

  • PRMT5-Dependent Methylation of the TIP60 Coactivator RUVBL1 Is a Key Regulator of Homologous Recombination

    Thomas L. Clarke;Maria Pilar Sanchez-Bailon;Kelly Chiang;John J. Reynolds

  • The APC/C and CBP/p300 cooperate to regulate transcription and cell-cycle progression

    Andrew S. Turnell;Grant S. Stewart;Grant S. Stewart;Roger J. A. Grand;Susan M. Rookes

Frequent Co-Authors

Paul Moss
Paul Moss University of Birmingham
J J Reynolds
J J Reynolds King's College London
Andrew P. Jackson
Andrew P. Jackson University of Edinburgh
Stephen J. Elledge
Stephen J. Elledge Harvard University
Thomas Dobner
Thomas Dobner Heinrich-Pette-Institute
Daniel Durocher
Daniel Durocher Lunenfeld-Tanenbaum Research Institute
Razqallah Hakem
Razqallah Hakem University of Toronto
Christopher G. Mathew
Christopher G. Mathew King's College London
Stephen P. Jackson
Stephen P. Jackson University of Cambridge
Hugh Watkins
Hugh Watkins University of Oxford

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