2023 - Research.com Genetics in United Kingdom Leader Award
Fellow of The Academy of Medical Sciences, United Kingdom
Christopher G. Mathew spends much of his time researching Genetics, Genome-wide association study, Genetic association, Single-nucleotide polymorphism and Inflammatory bowel disease. His Genetics study focuses mostly on Fanconi anemia, Fanconi anemia, complementation group C, Allele, Candidate gene and IRGM. His Genome-wide association study research includes elements of Bioinformatics, Copy-number variation, Disease, Locus and Genetic architecture.
His Genetic association study incorporates themes from Psoriasis, Genetic predisposition, Methylenetetrahydrofolate reductase, Blood pressure and Expression quantitative trait loci. His Single-nucleotide polymorphism research incorporates themes from Hemodynamics, ATG16L1 and Allele frequency. Christopher G. Mathew has researched Inflammatory bowel disease in several fields, including Common disease-common variant, Nonsynonymous substitution, NOD2, Ulcerative colitis and Genetic linkage.
His primary areas of study are Genetics, Immunology, Inflammatory bowel disease, Genome-wide association study and Gene. His study in Locus, Genetic association, Single-nucleotide polymorphism, Fanconi anemia and Allele is carried out as part of his studies in Genetics. His studies deal with areas such as Psoriasis and Candidate gene as well as Genetic association.
His study focuses on the intersection of Immunology and fields such as Crohn's disease with connections in the field of Gastroenterology. His Inflammatory bowel disease research is multidisciplinary, relying on both Genetic linkage and Ulcerative colitis. His Genome-wide association study research integrates issues from Odds ratio, Genetic predisposition, Case-control study, Allele frequency and Genetic architecture.
Christopher G. Mathew mostly deals with Genetics, Genome-wide association study, Inflammatory bowel disease, Internal medicine and Genetic association. Gene, Single-nucleotide polymorphism, Allele, Locus and Phenotype are among the areas of Genetics where Christopher G. Mathew concentrates his study. His research investigates the connection with Single-nucleotide polymorphism and areas like Genetic predisposition which intersect with concerns in University hospital and Tissue damage.
His Genome-wide association study research includes themes of Odds ratio, Psychosis, Allele frequency, Disease and Genetic architecture. His Inflammatory bowel disease study combines topics in areas such as Lamina propria, Cancer research and Ulcerative colitis. His biological study spans a wide range of topics, including Exome sequencing, Exome, Missense mutation, Genotyping and Missing heritability problem.
Christopher G. Mathew mainly focuses on Genetics, Genome-wide association study, Inflammatory bowel disease, Genetic association and Allele. His DNA replication, Chromosome instability and Gene duplication study, which is part of a larger body of work in Genetics, is frequently linked to DNA-binding protein and Replisome, bridging the gap between disciplines. The subject of his Genome-wide association study research is within the realm of Single-nucleotide polymorphism.
The various areas that he examines in his Inflammatory bowel disease study include Cancer research, Genotype and Ulcerative colitis. His research in Genetic association focuses on subjects like Missense mutation, which are connected to Exome. His work carried out in the field of Allele brings together such families of science as Immune system and ITGB8.
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Genome-wide association study of 14,000 cases of seven common diseases and 3,000 shared controls
Paul R. Burton;David G. Clayton;Lon R. Cardon;Nick Craddock.
Host-microbe interactions have shaped the genetic architecture of inflammatory bowel disease
Luke Jostins;Stephan Ripke;Rinse K Weersma;Richard H Duerr.
Genome-wide association defines more than 30 distinct susceptibility loci for Crohn's disease
Jeffrey C. Barrett;Sarah Hansoul;Dan L. Nicolae;Judy H. Cho.
Nature Genetics (2008)
Genome-wide meta-analysis increases to 71 the number of confirmed Crohn's disease susceptibility loci
Andre Franke;Dermot P B McGovern;Jeffrey C. Barrett;Kai Wang.
Nature Genetics (2010)
Genetic risk and a primary role for cell-mediated immune mechanisms in multiple sclerosis
Stephen Sawcer;Garrett Hellenthal;Matti Pirinen;Chris C. A. Spencer.
A genome-wide association scan of nonsynonymous SNPs identifies a susceptibility variant for Crohn disease in ATG16L1
Jochen Hampe;Andre Franke;Philip Rosenstiel;Philip Rosenstiel;Andreas Till.
Nature Genetics (2007)
Meta-analysis identifies 29 additional ulcerative colitis risk loci, increasing the number of confirmed associations to 47.
Carl A. Anderson;Gabrielle Boucher;Charlie W. Lees;Andre Franke.
Nature Genetics (2011)
Genome-wide association study identifies eight loci associated with blood pressure
Christopher Newton-Cheh;Christopher Newton-Cheh;Toby Johnson;Toby Johnson;Vesela Gateva;Martin D. Tobin.
Nature Genetics (2009)
Sequence variants in the autophagy gene IRGM and multiple other replicating loci contribute to Crohn's disease susceptibility.
Miles Parkes;Jeffrey C Barrett;Natalie J Prescott;Mark Tremelling.
Nature Genetics (2007)
Association between insertion mutation in NOD2 gene and Crohn's disease in German and British populations
Jochen Hampe;Andrew Cuthbert;Peter J. P. Croucher;Muddassar M. Mirza.
The Lancet (2001)
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