D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 48 Citations 6,505 110 World Ranking 14336 National Ranking 1027

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • DNA
  • Enzyme

Thomas Dobner mostly deals with Molecular biology, Cell biology, Adenovirus E1B protein, Retinoblastoma-like protein 1 and Adenoviridae. His Molecular biology study combines topics from a wide range of disciplines, such as Peptide sequence, Mutant protein, HSPA4 and Transactivation. His Cell biology research includes themes of Virology, Gene and Ubiquitin ligase, Cullin, CUL5.

His Adenovirus E1B protein research incorporates elements of RNA, Messenger RNA, Protein A/G, GPS2 and Protein G. He combines subjects such as Binding protein, DDB1, Mutant, Adenovirus Protein and SUMO-1 Protein with his study of Retinoblastoma-like protein 1. The concepts of his Adenoviridae study are interwoven with issues in Death-associated protein 6, Nuclear protein, Promyelocytic leukemia protein, RNA interference and Viral protein.

His most cited work include:

  • Blockage by Adenovirus E4orf6 of Transcriptional Activation by the p53 Tumor Suppressor (289 citations)
  • p300 Acts as a Transcriptional Coactivator for Mammalian Notch-1 (250 citations)
  • Induction of apoptosis by adenovirus E4orf4 protein is specific to transformed cells and requires an interaction with protein phosphatase 2A (139 citations)

What are the main themes of his work throughout his whole career to date?

His scientific interests lie mostly in Molecular biology, Cell biology, Virology, Viral replication and Adenovirus E1B protein. His Molecular biology research incorporates themes from Transcription factor, Transactivation, Mutant, Retinoblastoma-like protein 1 and Adenoviridae. Thomas Dobner has included themes like Cell, Death-associated protein 6, Transcription, SUMO protein and Ubiquitin ligase in his Cell biology study.

His biological study spans a wide range of topics, including Cell culture and Transfection. As a part of the same scientific study, he usually deals with the Viral replication, concentrating on DNA replication and frequently concerns with Capsid. His Adenovirus E1B protein research is multidisciplinary, incorporating perspectives in Nuclear export signal, RNA, Heterogeneous nuclear ribonucleoprotein, Nuclear protein and Viral protein.

He most often published in these fields:

  • Molecular biology (40.57%)
  • Cell biology (39.62%)
  • Virology (25.47%)

What were the highlights of his more recent work (between 2013-2020)?

  • Cell biology (39.62%)
  • Viral replication (25.47%)
  • Virology (25.47%)

In recent papers he was focusing on the following fields of study:

Thomas Dobner mainly focuses on Cell biology, Viral replication, Virology, DNA replication and SUMO protein. His research in Cell biology intersects with topics in Cell, Epigenetics, Transcription and Viral transformation. The various areas that he examines in his Viral replication study include RNA splicing, Molecular biology, Ubiquitin ligase and Gene.

His Virology research is multidisciplinary, relying on both RNA interference, Promyelocytic leukemia protein and Transfection. The study incorporates disciplines such as Virus and Capsid in addition to DNA replication. His research integrates issues of Death-associated protein 6, Transcription factor, Lytic cycle and Suppressor in his study of SUMO protein.

Between 2013 and 2020, his most popular works were:

  • DNA Virus Replication Compartments (89 citations)
  • High-Affinity Rb Binding, p53 Inhibition, Subcellular Localization, and Transformation by Wild-Type or Tumor-Derived Shortened Merkel Cell Polyomavirus Large T Antigens (71 citations)
  • Influence of ND10 components on epigenetic determinants of early KSHV latency establishment. (43 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • DNA
  • Enzyme

Thomas Dobner focuses on Viral replication, Virology, Cell biology, Gene and Genetics. The Viral replication study combines topics in areas such as Molecular biology, Cell nucleus and Transcriptional regulation. In his research on the topic of Molecular biology, Carcinogenesis is strongly related with Cell cycle.

His Virology study incorporates themes from Acquired immune system, Regulation of gene expression and Promyelocytic leukemia protein. Cell biology connects with themes related to Viral shedding in his study. His SUMO protein research incorporates elements of Transcription, Protein degradation and Viral Oncogene.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Blockage by Adenovirus E4orf6 of Transcriptional Activation by the p53 Tumor Suppressor

Thomas Dobner;Nobuo Horikoshi;Susanne Rubenwolf;Thomas Eugene Shenk.
Science (1996)

443 Citations

p300 Acts as a Transcriptional Coactivator for Mammalian Notch-1

Franz Oswald;Birgitt Täuber;Thomas Dobner;Soizic Bourteele.
Molecular and Cellular Biology (2001)

357 Citations

Induction of apoptosis by adenovirus E4orf4 protein is specific to transformed cells and requires an interaction with protein phosphatase 2A

Ronit Shtrichman;Rakefet Sharf;Haim Barr;Thomas Dobner.
Proceedings of the National Academy of Sciences of the United States of America (1999)

208 Citations

Regulation of DNA-End Resection by hnRNPU-like Proteins Promotes DNA Double-Strand Break Signaling and Repair

Sophie E. Polo;Andrew N. Blackford;J. Ross Chapman;Linda Baskcomb.
Molecular Cell (2012)

180 Citations

The adenovirus E4orf6 protein can promote E1A/E1B-induced focus formation by interfering with p53 tumor suppressor function

Michael Nevels;Susanne Rubenwolf;Thilo Spruss;Hans Wolf.
Proceedings of the National Academy of Sciences of the United States of America (1997)

172 Citations

"Hit-and-run" transformation by adenovirus oncogenes.

Michael Nevels;Birgitt Täuber;Thilo Spruss;Hans Wolf.
Journal of Virology (2001)

166 Citations

Proteasome-Dependent Degradation of Daxx by the Viral E1B-55K Protein in Human Adenovirus-Infected Cells

Sabrina Schreiner;Peter Wimmer;Hüseyin Sirma;Roger D. Everett.
Journal of Virology (2010)

163 Citations

SUMO-1 modification required for transformation by adenovirus type 5 early region 1B 55-kDa oncoprotein.

Christian Endter;Julia Kzhyshkowska;Roland Stauber;Thomas Dobner.
Proceedings of the National Academy of Sciences of the United States of America (2001)

161 Citations

Human Pathogens and the Host Cell SUMOylation System

Peter Wimmer;Sabrina Schreiner;Thomas Dobner.
Journal of Virology (2012)

158 Citations

The adenovirus type 5 E1B-55K oncoprotein is a highly active shuttle protein and shuttling is independent of E4orf6, p53 and Mdm2.

Friedrich Krätzer;Olaf Rosorius;Peter Heger;Nicole Hirschmann.
Oncogene (2000)

154 Citations

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