What is he best known for?
The fields of study he is best known for:
Samuel Benchimol focuses on Gene, Cell biology, Molecular biology, Cancer research and Gene expression.
His Gene research incorporates elements of Radioresistance and Cell type.
The study incorporates disciplines such as Apoptosis, Regulation of gene expression and Intrinsic apoptosis in addition to Cell biology.
The Molecular biology study combines topics in areas such as RNA, DNA damage, Telomerase reverse transcriptase and DNA replication.
His research in Cancer research intersects with topics in Cell cycle checkpoint, Cell culture, Transfection, Carcinogenesis and Psychological repression.
His research investigates the connection with Gene expression and areas like Gene rearrangement which intersect with concerns in Murine leukemia virus, Oncovirus, Oncogene, Friend virus and Friend leukemia.
His most cited work include:
- Reconstitution of telomerase activity in normal human cells leads to elongation of telomeres and extended replicative life span (912 citations)
- Regulation of PTEN transcription by p53. (746 citations)
- p53: oncogene or anti-oncogene? (685 citations)
What are the main themes of his work throughout his whole career to date?
His scientific interests lie mostly in Molecular biology, Gene, Cancer research, Cell biology and DNA damage.
His biological study spans a wide range of topics, including Cell culture, Transfection, Mutation, Mutant and DNA.
His research on Gene concerns the broader Genetics.
As a part of the same scientific family, Samuel Benchimol mostly works in the field of Cancer research, focusing on Tumor suppressor gene and, on occasion, Carcinoma.
His work deals with themes such as Apoptosis, Cell cycle, Ubiquitin and Immunoprecipitation, which intersect with Cell biology.
His DNA damage study incorporates themes from Radiosensitivity, Transcription factor, Mitosis, DNA repair and Telomere.
He most often published in these fields:
- Molecular biology (49.46%)
- Gene (36.56%)
- Cancer research (36.56%)
What were the highlights of his more recent work (between 2008-2019)?
- Cell biology (31.18%)
- DNA damage (22.58%)
- Cancer research (36.56%)
In recent papers he was focusing on the following fields of study:
Samuel Benchimol spends much of his time researching Cell biology, DNA damage, Cancer research, Ubiquitin and Cancer.
His Cell biology study combines topics in areas such as Telomere and Genetics.
He interconnects Cell cycle checkpoint, Receptor, In Situ Nick-End Labeling, Caspase 2 and Gene targeting in the investigation of issues within DNA damage.
His Cancer research study combines topics from a wide range of disciplines, such as Transcription factor, Programmed cell death, DNA repair and Morpholino.
His Ubiquitin research includes elements of Molecular biology, Proto-Oncogene Proteins c-mdm2, Subcellular localization and Promoter.
He performs multidisciplinary study on Molecular biology and HIV Long Terminal Repeat in his works.
Between 2008 and 2019, his most popular works were:
- ROS-mediated p53 induction of Lpin1 regulates fatty acid oxidation in response to nutritional stress. (121 citations)
- Abnormal DNA content in oral epithelial dysplasia is associated with increased risk of progression to carcinoma (69 citations)
- Role of Pirh2 in mediating the regulation of p53 and c-Myc. (49 citations)
In his most recent research, the most cited papers focused on:
Samuel Benchimol mainly focuses on DNA damage, Adipocyte, Oral and maxillofacial pathology, Carcinoma and Mouth neoplasm.
His DNA damage research includes themes of Suppressor, HEK 293 cells, Molecular biology, Proto-Oncogene Proteins c-myc and Ubiquitin ligase.
His study in Adipocyte intersects with areas of studies such as Metabolism, C2C12, Lipid metabolism, Phosphorylation and Biochemistry.
His Metabolism study often links to related topics such as Enzyme activator.
His Oral and maxillofacial pathology study overlaps with Cancer registry, Pathology, Dysplasia, Epithelial dysplasia and DNA Image Cytometry.
His research on Carcinoma often connects related areas such as Cancer.
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