What is he best known for?
The fields of study he is best known for:
Guillermina Lozano mostly deals with Cancer research, Mdm2, Molecular biology, Carcinogenesis and Mutant.
His Cancer research research integrates issues from Cell cycle checkpoint, Mutation, Regulation of gene expression, Gene and Apoptosis.
Guillermina Lozano has included themes like Nuclear export signal, Cell growth, Proto-Oncogene Proteins c-mdm2, Cell biology and Ubiquitin ligase in his Mdm2 study.
His Molecular biology study combines topics from a wide range of disciplines, such as In vivo, Transcription, Transactivation, Tumor suppressor gene and Binding site.
His Carcinogenesis study combines topics in areas such as RNA and Cell cycle.
The study incorporates disciplines such as Missense mutation and Metastasis in addition to Mutant.
His most cited work include:
- Rescue of early embryonic lethality in mdm2-deficient mice by deletion of p53 (1218 citations)
- A single nucleotide polymorphism in the MDM2 promoter attenuates the p53 tumor suppressor pathway and accelerates tumor formation in humans. (1059 citations)
- Gain of Function of a p53 Hot Spot Mutation in a Mouse Model of Li-Fraumeni Syndrome (818 citations)
What are the main themes of his work throughout his whole career to date?
Guillermina Lozano focuses on Cancer research, Mdm2, Molecular biology, Cell biology and Carcinogenesis.
Guillermina Lozano combines subjects such as Mutation, Gene, Mutant, Apoptosis and Tumor suppressor gene with his study of Cancer research.
His Mdm2 study incorporates themes from Embryonic stem cell, DNA damage, Proto-Oncogene Proteins c-mdm2, Cell cycle and Ubiquitin ligase.
His Molecular biology research incorporates themes from Transgene, Allele, Null allele, Regulation of gene expression and Transcription.
The Cell biology study combines topics in areas such as Cell, Ubiquitin and Programmed cell death.
His work deals with themes such as Downregulation and upregulation, Signal transduction, Loss of heterozygosity, Genetically modified mouse and In vivo, which intersect with Carcinogenesis.
He most often published in these fields:
- Cancer research (45.53%)
- Mdm2 (28.02%)
- Molecular biology (24.90%)
What were the highlights of his more recent work (between 2014-2021)?
- Cancer research (45.53%)
- Mutant (22.57%)
- Carcinogenesis (22.96%)
In recent papers he was focusing on the following fields of study:
His scientific interests lie mostly in Cancer research, Mutant, Carcinogenesis, Cancer and Gene.
His Cancer research research integrates issues from Mdm2, Metastasis, Allele, Stem cell and Genetically modified mouse.
The various areas that Guillermina Lozano examines in his Mutant study include Molecular biology, Signal transduction, Bioinformatics and Transcription factor.
As a member of one scientific family, Guillermina Lozano mostly works in the field of Molecular biology, focusing on Cell cycle and, on occasion, Cell growth and Cell type.
His study in Carcinogenesis is interdisciplinary in nature, drawing from both Mutation, Wild type, In vivo and Progenitor cell.
Guillermina Lozano has researched Gene in several fields, including Cell survival and Cell biology.
Between 2014 and 2021, his most popular works were:
- Improving survival by exploiting tumour dependence on stabilized mutant p53 for treatment (190 citations)
- Mutant p53 partners in crime. (117 citations)
- Novel MYBL1 Gene Rearrangements with Recurrent MYBL1–NFIB Fusions in Salivary Adenoid Cystic Carcinomas Lacking t(6;9) Translocations (96 citations)
In his most recent research, the most cited papers focused on:
Cancer research, Mutant, Gene, Cell biology and Carcinogenesis are his primary areas of study.
His Cancer research research is multidisciplinary, incorporating elements of Pathology, EZH2, Metastasis, Stem cell and Adenoid cystic carcinoma.
He interconnects Small intestine, Immunology, Cell growth and Somatic cell in the investigation of issues within Mutant.
Within one scientific family, he focuses on topics pertaining to Cancer under Gene, and may sometimes address concerns connected to Mdm2.
His Cell biology research is multidisciplinary, relying on both Phenotype, Molecular biology, Chemokine and Cell division.
The concepts of his Carcinogenesis study are interwoven with issues in Pancreatic tumor, Pancreatic cancer, Mutation, Allele and In vivo.
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