Guillermina Lozano mostly deals with Cancer research, Mdm2, Molecular biology, Carcinogenesis and Mutant. His Cancer research research integrates issues from Cell cycle checkpoint, Mutation, Regulation of gene expression, Gene and Apoptosis. Guillermina Lozano has included themes like Nuclear export signal, Cell growth, Proto-Oncogene Proteins c-mdm2, Cell biology and Ubiquitin ligase in his Mdm2 study.
His Molecular biology study combines topics from a wide range of disciplines, such as In vivo, Transcription, Transactivation, Tumor suppressor gene and Binding site. His Carcinogenesis study combines topics in areas such as RNA and Cell cycle. The study incorporates disciplines such as Missense mutation and Metastasis in addition to Mutant.
Guillermina Lozano focuses on Cancer research, Mdm2, Molecular biology, Cell biology and Carcinogenesis. Guillermina Lozano combines subjects such as Mutation, Gene, Mutant, Apoptosis and Tumor suppressor gene with his study of Cancer research. His Mdm2 study incorporates themes from Embryonic stem cell, DNA damage, Proto-Oncogene Proteins c-mdm2, Cell cycle and Ubiquitin ligase.
His Molecular biology research incorporates themes from Transgene, Allele, Null allele, Regulation of gene expression and Transcription. The Cell biology study combines topics in areas such as Cell, Ubiquitin and Programmed cell death. His work deals with themes such as Downregulation and upregulation, Signal transduction, Loss of heterozygosity, Genetically modified mouse and In vivo, which intersect with Carcinogenesis.
His scientific interests lie mostly in Cancer research, Mutant, Carcinogenesis, Cancer and Gene. His Cancer research research integrates issues from Mdm2, Metastasis, Allele, Stem cell and Genetically modified mouse. The various areas that Guillermina Lozano examines in his Mutant study include Molecular biology, Signal transduction, Bioinformatics and Transcription factor.
As a member of one scientific family, Guillermina Lozano mostly works in the field of Molecular biology, focusing on Cell cycle and, on occasion, Cell growth and Cell type. His study in Carcinogenesis is interdisciplinary in nature, drawing from both Mutation, Wild type, In vivo and Progenitor cell. Guillermina Lozano has researched Gene in several fields, including Cell survival and Cell biology.
Cancer research, Mutant, Gene, Cell biology and Carcinogenesis are his primary areas of study. His Cancer research research is multidisciplinary, incorporating elements of Pathology, EZH2, Metastasis, Stem cell and Adenoid cystic carcinoma. He interconnects Small intestine, Immunology, Cell growth and Somatic cell in the investigation of issues within Mutant.
Within one scientific family, he focuses on topics pertaining to Cancer under Gene, and may sometimes address concerns connected to Mdm2. His Cell biology research is multidisciplinary, relying on both Phenotype, Molecular biology, Chemokine and Cell division. The concepts of his Carcinogenesis study are interwoven with issues in Pancreatic tumor, Pancreatic cancer, Mutation, Allele and In vivo.
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Rescue of early embryonic lethality in mdm2-deficient mice by deletion of p53
Roberta Montes de Oca Luna;Daniel S. Wagner;Guillermina Lozano.
A single nucleotide polymorphism in the MDM2 promoter attenuates the p53 tumor suppressor pathway and accelerates tumor formation in humans.
Gareth L. Bond;Wenwei Hu;Elisabeth E. Bond;Harlan Robins.
Gain of Function of a p53 Hot Spot Mutation in a Mouse Model of Li-Fraumeni Syndrome
Gene A. Lang;Tomoo Iwakuma;Young Ah Suh;Geng Liu.
Transcriptional activation by wild-type but not transforming mutants of the p53 anti-oncogene
Loretta Raycroft;Hongyun Wu;Guillermina Lozano.
Pirh2, a p53-Induced Ubiquitin-Protein Ligase, Promotes p53 Degradation
Roger P. Leng;Yunping Lin;Weili Ma;Hong Wu.
Rescue of embryonic lethality in Mdm4 -null mice by loss of Trp53 suggests a nonoverlapping pathway with MDM2 to regulate p53
John Parant;Arturo Chavez-Reyes;Arturo Chavez-Reyes;Natalie A. Little;Wen Yan.
Nature Genetics (2001)
MDM2, An Introduction
Tomoo Iwakuma;Guillermina Lozano.
Molecular Cancer Research (2003)
NF-kappa B activation of p53. A potential mechanism for suppressing cell growth in response to stress.
Hongyun Wu;Guillermina Lozano.
Journal of Biological Chemistry (1994)
p53-independent functions of the p19ARF tumor suppressor
Jason D. Weber;John R. Jeffers;Jerold E. Rehg;David H. Randle.
Genes & Development (2000)
CRITICAL ROLE FOR SER20 OF HUMAN P53 IN THE NEGATIVE REGULATION OF P53 BY MDM2
Tamar Unger;Tamar Juven-Gershon;Eli Moallem;Michael Berger.
The EMBO Journal (1999)
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