What is he best known for?
The fields of study he is best known for:
Bernd Epe mainly focuses on DNA damage, DNA, Biochemistry, Molecular biology and DNA glycosylase.
Bernd Epe specializes in DNA damage, namely Pyrimidine dimer.
His study in the fields of Endonuclease under the domain of DNA overlaps with other disciplines such as Potassium bromate.
His study in the field of Carcinogen and Mitochondrial DNA is also linked to topics like Ascorbic acid and Risk assessment.
His research investigates the link between Molecular biology and topics such as DNA repair that cross with problems in Mutation.
His studies in DNA glycosylase integrate themes in fields like Uracil, Deamination and Deoxyuridine.
His most cited work include:
- ACCUMULATION OF PREMUTAGENIC DNA LESIONS IN MICE DEFECTIVE IN REMOVAL OF OXIDATIVE BASE DAMAGE (710 citations)
- Wavelength dependence of oxidative DNA damage induced by UV and visible light. (488 citations)
- Occupational exposure to heavy metals: DNA damage induction and DNA repair inhibition prove co-exposures to cadmium, cobalt and lead as more dangerous than hitherto expected (207 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of study are DNA damage, DNA, Biochemistry, Molecular biology and DNA glycosylase.
His biological study spans a wide range of topics, including Oxidative stress, Photochemistry, Radical and Singlet oxygen.
In DNA, Bernd Epe works on issues like Exonuclease III, which are connected to DNA- lyase.
His Molecular biology study combines topics from a wide range of disciplines, such as Base excision repair, Gene expression, Nucleotide excision repair, Gene and DNA repair.
His work in DNA repair covers topics such as Cell biology which are related to areas like Chromatin and Epigenetics.
His study in DNA glycosylase is interdisciplinary in nature, drawing from both Nuclear DNA, Mitochondrial DNA, Mitochondrion and AP site.
He most often published in these fields:
- DNA damage (55.70%)
- DNA (50.00%)
- Biochemistry (36.08%)
What were the highlights of his more recent work (between 2012-2020)?
- DNA damage (55.70%)
- DNA glycosylase (24.68%)
- DNA (50.00%)
In recent papers he was focusing on the following fields of study:
His scientific interests lie mostly in DNA damage, DNA glycosylase, DNA, Base excision repair and Molecular biology.
His DNA damage study deals with the bigger picture of Biochemistry.
The study incorporates disciplines such as Cell culture, Porphyrin, Singlet oxygen, Circular dichroism and Hematoporphyrin in addition to DNA glycosylase.
His DNA research is multidisciplinary, relying on both Cell growth, Biophysics, Photochemistry, Gene and Stereochemistry.
His work deals with themes such as Nucleotide excision repair and Transcription factor, which intersect with Base excision repair.
His research integrates issues of Oxidative phosphorylation, Topoisomerase inhibitor and Proliferating cell nuclear antigen in his study of DNA repair.
Between 2012 and 2020, his most popular works were:
- Nitrate and nitrite in the diet: How to assess their benefit and risk for human health (107 citations)
- Widespread transcriptional gene inactivation initiated by a repair intermediate of 8-oxoguanine (37 citations)
- Oxidatively generated base modifications in DNA: Not only carcinogenic risk factor but also regulatory mark? ☆ (35 citations)
In his most recent research, the most cited papers focused on:
Bernd Epe mostly deals with DNA glycosylase, Base excision repair, DNA repair, DNA damage and Nucleotide excision repair.
His DNA glycosylase research is under the purview of Biochemistry.
Promoter, Cell growth, Cancer research and Ubiquitin is closely connected to Molecular biology in his research, which is encompassed under the umbrella topic of Base excision repair.
His DNA damage study introduces a deeper knowledge of DNA.
His study on Pyrimidine dimer is often connected to Protonation as part of broader study in DNA.
Bernd Epe has included themes like Oxidative stress, Oxidative phosphorylation and DNA-binding protein in his Nucleotide excision repair study.
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