Bernd Epe mainly focuses on DNA damage, DNA, Biochemistry, Molecular biology and DNA glycosylase. Bernd Epe specializes in DNA damage, namely Pyrimidine dimer. His study in the fields of Endonuclease under the domain of DNA overlaps with other disciplines such as Potassium bromate.
His study in the field of Carcinogen and Mitochondrial DNA is also linked to topics like Ascorbic acid and Risk assessment. His research investigates the link between Molecular biology and topics such as DNA repair that cross with problems in Mutation. His studies in DNA glycosylase integrate themes in fields like Uracil, Deamination and Deoxyuridine.
His primary areas of study are DNA damage, DNA, Biochemistry, Molecular biology and DNA glycosylase. His biological study spans a wide range of topics, including Oxidative stress, Photochemistry, Radical and Singlet oxygen. In DNA, Bernd Epe works on issues like Exonuclease III, which are connected to DNA- lyase.
His Molecular biology study combines topics from a wide range of disciplines, such as Base excision repair, Gene expression, Nucleotide excision repair, Gene and DNA repair. His work in DNA repair covers topics such as Cell biology which are related to areas like Chromatin and Epigenetics. His study in DNA glycosylase is interdisciplinary in nature, drawing from both Nuclear DNA, Mitochondrial DNA, Mitochondrion and AP site.
His scientific interests lie mostly in DNA damage, DNA glycosylase, DNA, Base excision repair and Molecular biology. His DNA damage study deals with the bigger picture of Biochemistry. The study incorporates disciplines such as Cell culture, Porphyrin, Singlet oxygen, Circular dichroism and Hematoporphyrin in addition to DNA glycosylase.
His DNA research is multidisciplinary, relying on both Cell growth, Biophysics, Photochemistry, Gene and Stereochemistry. His work deals with themes such as Nucleotide excision repair and Transcription factor, which intersect with Base excision repair. His research integrates issues of Oxidative phosphorylation, Topoisomerase inhibitor and Proliferating cell nuclear antigen in his study of DNA repair.
Bernd Epe mostly deals with DNA glycosylase, Base excision repair, DNA repair, DNA damage and Nucleotide excision repair. His DNA glycosylase research is under the purview of Biochemistry. Promoter, Cell growth, Cancer research and Ubiquitin is closely connected to Molecular biology in his research, which is encompassed under the umbrella topic of Base excision repair.
His DNA damage study introduces a deeper knowledge of DNA. His study on Pyrimidine dimer is often connected to Protonation as part of broader study in DNA. Bernd Epe has included themes like Oxidative stress, Oxidative phosphorylation and DNA-binding protein in his Nucleotide excision repair study.
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ACCUMULATION OF PREMUTAGENIC DNA LESIONS IN MICE DEFECTIVE IN REMOVAL OF OXIDATIVE BASE DAMAGE
Arne Klungland;Ian Rosewell;Stephan Hollenbach;Elisabeth Larsen.
Proceedings of the National Academy of Sciences of the United States of America (1999)
Wavelength dependence of oxidative DNA damage induced by UV and visible light.
Christopher Kielbassa;Len Roza;Bernd Epe.
Occupational exposure to heavy metals: DNA damage induction and DNA repair inhibition prove co-exposures to cadmium, cobalt and lead as more dangerous than hitherto expected
Jan G. Hengstler;Ulrich Bolm-Audorff;Andreas Faldum;Kai Janssen.
Problems in the measurement of 8-oxoguanine in human DNA. Report of a workshop, DNA Oxidation, held in Aberdeen, UK, 19-21 January, 1997
Andrew Collins;Jean Cadet;Bernd Epe;Catherine Gedik.
Age-related and tissue-specific accumulation of oxidative DNA base damage in 7,8-dihydro-8-oxoguanine-DNA glycosylase (Ogg1) deficient mice.
Marcel Osterod;Stephan Hollenbach;Jan G. Hengstler;Deborah E. Barnes.
Genotoxicity of singlet oxygen.
Chemico-Biological Interactions (1991)
DNA damage induced by photosensitizers in cellular and cell-free systems.
Bernd Epe;Michael Pflaum;Serge Boiteux.
Mutation Research/genetic Toxicology (1993)
DNA damage by peroxynitrite characterized with DNA repair enzymes
Bernd Epe;Daniel Ballmaier;Ivan Roussyn;Karlis Briviba.
Nucleic Acids Research (1996)
A global DNA repair mechanism involving the Cockayne syndrome B (CSB) gene product can prevent the in vivo accumulation of endogenous oxidative DNA base damage
Marcel Osterod;Elisabeth Larsen;Florence Le Page;Jan G. Hengstler.
Determination of steady-state levels of oxidative DNA base modifications in mammalian cells by means of repair endonucleases.
Michael Pflaum;Olaf Will;Bernd Epe.
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