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Microbiology

D-Index
59
Citations
11107
World Ranking
3304
National Ranking
1303

Overview

Richard J. Quigg is affiliated with the University at Buffalo, State University of New York in the United States. Their research primarily focuses on the fields of Medicine and Immunology and Microbiology, with significant contributions to subfields including Immunology, Nephrology, Physiology, Epidemiology, and Health Information Management.

Their main research topics revolve around the complement system in diseases, renal diseases and glomerulopathies, phagocytosis and immune regulation, chronic disease management strategies, medical coding and health information, erythrocyte function and pathophysiology, and T-cell and B-cell immunology.

Among their recent publications are:

  • Double negative T cells, a potential biomarker for systemic lupus erythematosus (2020) published in Precision Clinical Medicine
  • Local complement factor H protects kidney endothelial cell structure and function (2021) published in Kidney International
  • Complement: Functions, location and implications (2023) published in Immunology
  • Improving Clinical Trials for Anticomplement Therapies in Complement-Mediated Glomerulopathies: Report of a Scientific Workshop Sponsored by the National Kidney Foundation (2021) published in American Journal of Kidney Diseases
  • Unconjugated p-cresol activates macrophage macropinocytosis leading to increased LDL uptake (2021) published in JCI Insight

Frequent coauthors collaborating with Richard J. Quigg include:

  • Jessy J. Alexander
  • Alexander Jacob
  • Anthony Chang
  • Daniel McSkimming
  • Kabir Jalal

The researcher has contributed multiple papers to several publication venues, the most frequent being:

  • Precision Clinical Medicine
  • Immunobiology
  • Kidney International
  • Immunology
  • American Journal of Kidney Diseases

Their work has covered a range of topics across the immune system and kidney-related diseases, reflecting interdisciplinary efforts in both clinical and molecular investigations. The focus on complement system functions and renal pathology is evident across their published research.

Best Publications

  • Prominent neurodegeneration and increased plaque formation in complement-inhibited Alzheimer's mice

    Tony Wyss-Coray;Fengrong Yan;Amy Hsiu-Ti Lin;John D. Lambris

  • Acute Renal Failure in Endotoxemia Is Caused by TNF Acting Directly on TNF Receptor-1 in Kidney

    Patrick N. Cunningham;Hristem M. Dyanov;Pierce Park;Jun Wang

  • MicroRNA-377 is up-regulated and can lead to increased fibronectin production in diabetic nephropathy

    Qiang Wang;Youli Wang;Andrew W. Minto;Jinhua Wang

  • miR-17-92 cluster accelerates adipocyte differentiation by negatively regulating tumor-suppressor Rb2/p130

    Qiang Wang;Yan Chun Li;Jinhua Wang;Juan Kong

  • Renoprotective role of the vitamin D receptor in diabetic nephropathy

    Z. Zhang;L. Sun;Y. Wang;G. Ning

  • In Situ B Cell-Mediated Immune Responses and Tubulointerstitial Inflammation in Human Lupus Nephritis

    Anthony Chang;Scott G. Henderson;Daniel Brandt;Ni Liu

  • Role of Toll-Like Receptor 4 in Endotoxin-Induced Acute Renal Failure

    Patrick N. Cunningham;Ying Wang;Rongqing Guo;Gang He

  • New Approaches to the Treatment of Dense Deposit Disease

    Richard J.H. Smith;Jessy Alexander;Paul N. Barlow;Marina Botto

  • TNF is a key mediator of Septic Encephalopathy acting through its receptor, TNF Receptor-1

    Jessy J. Alexander;Alexander Jacob;Patrick Cunningham;Lauren Hensley

  • Mouse complement receptors type 1 (CR1;CD35) and type 2 (CR2;CD21): expression on normal B cell subpopulations and decreased levels during the development of autoimmunity in MRL/lpr mice.

    K Takahashi;Y Kozono;T J Waldschmidt;D Berthiaume

  • Blockade of antibody-induced glomerulonephritis with Crry-Ig, a soluble murine complement inhibitor.

    Quigg Rj;Kozono Y;Berthiaume D;Lim A

  • C5a promotes development of experimental lupus nephritis which can be blocked with a specific receptor antagonist.

    Lihua Bao;Iyabo Osawe;Tipu Puri;John D. Lambris

  • Anti-diabetic effect of ginsenoside Re in ob/ob mice.

    Jing-Tian Xie;Sangeeta R. Mehendale;Xinmin Li;Richard Quigg

  • Injury in renal ischemia-reperfusion is independent from immunoglobulins and T lymphocytes.

    Pierce Park;Mark Haas;Patrick N. Cunningham;Lihua Bao

  • DGKE Variants Cause a Glomerular Microangiopathy That Mimics Membranoproliferative GN

    Fatih Ozaltin;Binghua Li;Alysha Rauhauser;Sung Wan An

  • C3a Is Required for the Production of CXC Chemokines by Tubular Epithelial Cells after Renal Ishemia/Reperfusion

    Joshua M. Thurman;Amanda M. Lenderink;Pamela A. Royer;Kathrin E. Coleman

  • Experimental membranous nephropathy redux

    Andrey V. Cybulsky;Richard J. Quigg;David J. Salant

  • Pathogenic Natural Antibodies Recognizing Annexin IV Are Required to Develop Intestinal Ischemia-Reperfusion Injury

    Liudmila Kulik;Sherry D. Fleming;Chantal Moratz;Jason W. Reuter

  • Transgenic Mice Overexpressing the Complement Inhibitor Crry as a Soluble Protein Are Protected from Antibody-induced Glomerular Injury

    R. J. Quigg;Chun He;A. Lim;D. Berthiaume

  • Complement Factor H Deficiency Accelerates Development of Lupus Nephritis

    Lihua Bao;Mark Haas;Richard J. Quigg

  • Transgenic Expression of a Soluble Complement Inhibitor Protects Against Renal Disease and Promotes Survival in MRL/lpr Mice

    Lihua Bao;Mark Haas;Susan A. Boackle;Damian M. Kraus

  • C5a alters blood-brain barrier integrity in experimental lupus

    Alexander Jacob;Bradley Hack;Eddie Chiang;Joe G. N. Garcia

  • Immune complex glomerulonephritis in C4- and C3-deficient mice

    Richard J. Quigg;Richard J. Quigg;Alice Lim;Alice Lim;Mark Haas;Mark Haas;Jessy J. Alexander;Jessy J. Alexander

Frequent Co-Authors

Mark Haas
Mark Haas Cedars-Sinai Medical Center
V. Michael Holers
V. Michael Holers University of Colorado Denver
John D. Lambris
John D. Lambris University of Pennsylvania
Michael C. Carroll
Michael C. Carroll Boston Children's Hospital
B. Paul Morgan
B. Paul Morgan Cardiff University
Joel M. Guthridge
Joel M. Guthridge Oklahoma Medical Research Foundation
Joseph V. Bonventre
Joseph V. Bonventre Brigham and Women's Hospital
Stanley A. Schwartz
Stanley A. Schwartz University at Buffalo, State University of New York
Friedhelm Hildebrandt
Friedhelm Hildebrandt Boston Children's Hospital
Graeme I. Bell
Graeme I. Bell University of Chicago

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