What is he best known for?
The fields of study he is best known for:
- Gene
- Internal medicine
- Immune system
Richard J. Quigg spends much of his time researching Internal medicine, Endocrinology, Complement system, Immunology and Kidney.
The concepts of his Endocrinology study are interwoven with issues in Receptor and Lupus nephritis.
His Complement system study incorporates themes from In vivo, In vitro and Immune complex.
His work in Immune complex addresses issues such as Polyclonal antibodies, which are connected to fields such as Immune system, Glomerulonephritis and Pathology.
His Immunology research integrates issues from Systemic lupus erythematosus and Neurodegeneration.
His research in Kidney intersects with topics in Apoptosis, T lymphocyte, Hypoalbuminemia and Immunoglobulin G.
His most cited work include:
- Prominent neurodegeneration and increased plaque formation in complement-inhibited Alzheimer's mice (362 citations)
- MicroRNA-377 is up-regulated and can lead to increased fibronectin production in diabetic nephropathy (320 citations)
- Acute Renal Failure in Endotoxemia Is Caused by TNF Acting Directly on TNF Receptor-1 in Kidney (309 citations)
What are the main themes of his work throughout his whole career to date?
Richard J. Quigg mostly deals with Complement system, Immunology, Internal medicine, Endocrinology and Cell biology.
The various areas that Richard J. Quigg examines in his Complement system study include Inflammation and Immune complex.
His studies deal with areas such as Systemic lupus erythematosus and Glomerulonephritis as well as Immunology.
His study connects Apoptosis and Internal medicine.
His Endocrinology research includes elements of Receptor and Genetically modified mouse.
The Cell biology study combines topics in areas such as Complement component 5, Complement membrane attack complex, Complement and CD46.
He most often published in these fields:
- Complement system (42.35%)
- Immunology (39.29%)
- Internal medicine (35.71%)
What were the highlights of his more recent work (between 2010-2021)?
- Immunology (39.29%)
- Complement system (42.35%)
- Glomerulonephritis (15.82%)
In recent papers he was focusing on the following fields of study:
His scientific interests lie mostly in Immunology, Complement system, Glomerulonephritis, Inflammation and Systemic lupus erythematosus.
Within one scientific family, he focuses on topics pertaining to Lupus nephritis under Immunology, and may sometimes address concerns connected to Classical complement pathway, Eculizumab and Complement receptor 1.
His Complement system research is multidisciplinary, incorporating elements of Endocrinology, Internal medicine, Nephritis and Cell biology.
The Glomerulonephritis study combines topics in areas such as Diacylglycerol kinase, Immune complex and Renal function.
His studies in Inflammation integrate themes in fields like Integrin alpha M, Molecular biology, C5a receptor, Curcumin and Kidney.
His Immune system study combines topics in areas such as Receptor and Diabetic nephropathy.
Between 2010 and 2021, his most popular works were:
- In Situ B Cell-Mediated Immune Responses and Tubulointerstitial Inflammation in Human Lupus Nephritis (201 citations)
- DGKE Variants Cause a Glomerular Microangiopathy That Mimics Membranoproliferative GN (94 citations)
- Complement Factor H Deficiency Accelerates Development of Lupus Nephritis (85 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Internal medicine
- Immune system
Richard J. Quigg focuses on Immunology, Complement system, Systemic lupus erythematosus, Glomerulonephritis and Cell biology.
His studies deal with areas such as Nephrotic syndrome, Tight junction, Endothelium and Membranous nephropathy as well as Immunology.
His Complement system research incorporates elements of Inflammation and Internal medicine.
His research in Internal medicine intersects with topics in Curcumin and Immune complex.
In Glomerulonephritis, Richard J. Quigg works on issues like Alternative complement pathway, which are connected to Complement deficiency and CD46.
His work in Anaphylatoxin tackles topics such as Receptor which are related to areas like Endocrinology.
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