B. Paul Morgan mainly investigates Complement system, Immunology, Cell biology, Complement membrane attack complex and Factor H. His Complement system research incorporates themes from Inflammation, Molecular biology and Proteases. His Immunology research focuses on Disease and how it connects with Immunopathology and Genotype.
His research in Cell biology intersects with topics in Cell activation, Biochemistry, Cell membrane and CD46. His research integrates issues of Inflammasome, CD59, Anaphylatoxin, Signal transduction and Lipid bilayer in his study of Complement membrane attack complex. The concepts of his Factor H study are interwoven with issues in Complement inhibitor, Complement factor I, Atypical hemolytic uremic syndrome and Complement factor B.
Immunology, Complement system, Complement membrane attack complex, Cell biology and Complement are his primary areas of study. His research brings together the fields of Disease and Immunology. He interconnects Inflammation and Pathology in the investigation of issues within Complement system.
His study focuses on the intersection of Complement membrane attack complex and fields such as CD59 with connections in the field of Molecular biology and Cytolysis. His Cell biology research focuses on CD46 and how it relates to Decay-accelerating factor. As part of one scientific family, he deals mainly with the area of Factor H, narrowing it down to issues related to the Biochemistry, and often Monoclonal antibody.
B. Paul Morgan spends much of his time researching Complement system, Immunology, Complement, Complement membrane attack complex and Disease. His work carried out in the field of Complement system brings together such families of science as Inflammation and Multiple sclerosis. His work in Immunology is not limited to one particular discipline; it also encompasses Locus.
His Complement study combines topics in areas such as iC3b, Innate immune system, Neuroscience and Pathogenesis. His Complement membrane attack complex study combines topics from a wide range of disciplines, such as Inflammasome, Cell membrane, Pathology, Cell activation and Cell biology. In his study, Complement receptor 1 and Bioinformatics is inextricably linked to Clusterin, which falls within the broad field of Disease.
B. Paul Morgan mostly deals with Complement system, Immunology, Complement membrane attack complex, Disease and Inflammation. His Complement system study frequently intersects with other fields, such as C1-inhibitor. The various areas that B. Paul Morgan examines in his Immunology study include Macular degeneration and Pathology.
He combines subjects such as Inflammasome, Cell activation, Cell biology, C5a receptor and Lipid bilayer with his study of Complement membrane attack complex. In general Cell biology study, his work on Signalling pathways often relates to the realm of Molecule, thereby connecting several areas of interest. B. Paul Morgan has researched Disease in several fields, including Biomarker, Neurology, Complement and Intensive care medicine.
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Complement-mediated lysis by anti-CD20 mAb correlates with segregation into lipid rafts
Mark S Cragg;Suzanne M Morgan;H T Claude Chan;B Paul Morgan.
C3 glomerulopathy: consensus report
Matthew C. Pickering;Vivette D. D'agati;Carla M. Nester;Richard J. Smith.
Kidney International (2013)
Gain-of-function mutations in complement factor B are associated with atypical hemolytic uremic syndrome.
Elena Goicoechea de Jorge;Claire L. Harris;Jorge Esparza-Gordillo;Luis Carreras.
Proceedings of the National Academy of Sciences of the United States of America (2007)
Complement deficiency and disease.
B.Paul Morgan;Mark J Walport.
Immunology Today (1991)
Soluble Forms of Toll-Like Receptor (TLR)2 Capable of Modulating TLR2 Signaling Are Present in Human Plasma and Breast Milk
Emmanuel LeBouder;Julia E. Rey-Nores;Neil K. Rushmere;Martin Grigorov.
Journal of Immunology (2003)
Atypical hemolytic uremic syndrome and C3 glomerulopathy: conclusions from a “Kidney Disease: Improving Global Outcomes” (KDIGO) Controversies Conference
Timothy H.J. Goodship;H. Terence Cook;Fadi Fakhouri;Fernando C. Fervenza.
Kidney International (2017)
The complement membrane attack complex triggers intracellular Ca2+ fluxes leading to NLRP3 inflammasome activation.
Kathy Triantafilou;Timothy R. Hughes;Martha Triantafilou;B. Paul Morgan.
Journal of Cell Science (2013)
Membrane proteins that protect against complement lysis
B. Paul Morgan;Seppo Meri.
Springer Seminars in Immunopathology (1994)
Expression of complement in the brain: role in health and disease
B.Paul Morgan;Philippe Gasque.
Immunology Today (1996)
Complement, a target for therapy in inflammatory and degenerative diseases.
B. Paul Morgan;Claire L. Harris;Claire L. Harris.
Nature Reviews Drug Discovery (2015)
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