His primary scientific interests are in Factor H, Genetics, Complement system, Atypical hemolytic uremic syndrome and Immunology. His Factor H research incorporates elements of Mutation, Alternative complement pathway, Complement factor I and Complement. The Complement system study combines topics in areas such as Endocrinology, Internal medicine and Cell biology.
He combines subjects such as Glomerulonephritis, Hemolytic anemia, Kidney transplantation, Dialysis and Etiology with his study of Atypical hemolytic uremic syndrome. The concepts of his Immunology study are interwoven with issues in Thrombotic microangiopathy, Disease and Kidney disease. His Complement factor B study integrates concerns from other disciplines, such as Penetrance and Pathogenesis.
His primary areas of investigation include Immunology, Complement system, Genetics, Atypical hemolytic uremic syndrome and Factor H. His study in the field of Antibody, Autoantibody and Pathogenesis also crosses realms of Haemolytic-uraemic syndrome. His Complement system study also includes fields such as
His Atypical hemolytic uremic syndrome research is multidisciplinary, incorporating perspectives in Hemolytic anemia, Internal medicine, Thrombotic microangiopathy, Disease and Eculizumab. His research investigates the connection between Factor H and topics such as Complement factor I that intersect with issues in CD46. His Alternative complement pathway research is multidisciplinary, relying on both Properdin and Biochemistry.
Santiago Rodríguez de Córdoba focuses on Atypical hemolytic uremic syndrome, Internal medicine, Immunology, Complement system and Disease. His research integrates issues of Factor H and Thrombotic microangiopathy in his study of Atypical hemolytic uremic syndrome. His Factor H research is multidisciplinary, incorporating elements of Mutation and Allele.
Santiago Rodríguez de Córdoba has included themes like Immunohistochemistry and Transplantation in his Complement system study. His Disease study combines topics from a wide range of disciplines, such as Gene and Glomerulopathy. His study in Computational biology is interdisciplinary in nature, drawing from both Gene duplication, MAP1LC3B, BECN1 and Autolysosome.
Santiago Rodríguez de Córdoba spends much of his time researching Atypical hemolytic uremic syndrome, Immunology, Disease, Alternative complement pathway and Gene. He has researched Atypical hemolytic uremic syndrome in several fields, including Mutation, Thrombotic microangiopathy, Cohort and Eculizumab. Santiago Rodríguez de Córdoba interconnects Kidney disease and CD40 in the investigation of issues within Immunology.
Alternative complement pathway is a subfield of Complement system that Santiago Rodríguez de Córdoba investigates. The concepts of his Complement system study are interwoven with issues in CD86 and Cell biology. His study on Gene is covered under Genetics.
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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)
Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)
Guidelines for the use and interpretation of assays for monitoring autophagy
Daniel J. Klionsky;Fabio C. Abdalla;Hagai Abeliovich;Robert T. Abraham.
Autophagy (2012)
Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356
Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)
Genomic sequence of the pathogenic and allergenic filamentous fungus Aspergillus fumigatus
William C. Nierman;William C. Nierman;Arnab Pain;Michael J. Anderson;Jennifer R. Wortman;Jennifer R. Wortman.
Nature (2005)
The human complement factor H: functional roles, genetic variations and disease associations.
Santiago Rodrı́guez de Córdoba;Jorge Esparza-Gordillo;Elena Goicoechea de Jorge;Margarita Lopez-Trascasa.
Molecular Immunology (2004)
C3 glomerulopathy: consensus report
Matthew C. Pickering;Vivette D. D'agati;Carla M. Nester;Richard J. Smith.
Kidney International (2013)
Gain-of-function mutations in complement factor B are associated with atypical hemolytic uremic syndrome.
Elena Goicoechea de Jorge;Claire L. Harris;Jorge Esparza-Gordillo;Luis Carreras.
Proceedings of the National Academy of Sciences of the United States of America (2007)
Mechanism suppressing glycogen synthesis in neurons and its demise in progressive myoclonus epilepsy.
David Vilchez;Susana Ros;Daniel Cifuentes;Lluís Pujadas.
Nature Neuroscience (2007)
Atypical hemolytic uremic syndrome and C3 glomerulopathy: conclusions from a “Kidney Disease: Improving Global Outcomes” (KDIGO) Controversies Conference
Timothy H.J. Goodship;H. Terence Cook;Fadi Fakhouri;Fernando C. Fervenza.
Kidney International (2017)
Differences in reactive oxygen species production explain the phenotypes associated with common mouse mitochondrial DNA variants.
Raquel Moreno-Loshuertos;Rebeca Acín-Pérez;Patricio Fernández-Silva;Nieves Movilla.
Nature Genetics (2006)
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Publications: 153
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