D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Microbiology D-index 46 Citations 6,481 106 World Ranking 3839 National Ranking 340

Overview

What is she best known for?

The fields of study she is best known for:

  • Gene
  • Enzyme
  • Genetics

Claire L. Harris spends much of her time researching Complement system, Factor H, Immunology, Innate immune system and Alternative complement pathway. Her Complement system research integrates issues from Proteases, Biochemistry, Binding site and Cell biology. Her biological study spans a wide range of topics, including Mutation, Molecular biology, Internal medicine and Complement factor B.

Her Immunology study focuses mostly on Inflammation and Complement. Her research in Inflammation tackles topics such as Disease which are related to areas like Drug development, Systemic inflammation, Immune defence and Genotype. Her Innate immune system research focuses on Classical complement pathway and how it connects with T cell, Complement receptor and Acquired immune system.

Her most cited work include:

  • Gain-of-function mutations in complement factor B are associated with atypical hemolytic uremic syndrome. (371 citations)
  • C3 glomerulopathy: consensus report (326 citations)
  • Complement, a target for therapy in inflammatory and degenerative diseases. (214 citations)

What are the main themes of her work throughout her whole career to date?

Her primary areas of investigation include Immunology, Complement system, Factor H, Cell biology and Alternative complement pathway. Her study in Immunology concentrates on Complement, Inflammation, Complement receptor, Classical complement pathway and Complement membrane attack complex. Claire L. Harris combines subjects such as Molecular biology, Innate immune system, Disease and Macular degeneration with her study of Complement system.

The study incorporates disciplines such as Drug development, Bioinformatics and Intensive care medicine in addition to Disease. Her Factor H study combines topics from a wide range of disciplines, such as Complement factor I, Complement factor B and CD46. As a part of the same scientific study, she usually deals with the Cell biology, concentrating on Decay-accelerating factor and frequently concerns with Complement receptor 1.

She most often published in these fields:

  • Immunology (37.06%)
  • Complement system (35.03%)
  • Factor H (23.35%)

What were the highlights of her more recent work (between 2015-2021)?

  • Complement system (35.03%)
  • Macular degeneration (12.18%)
  • Complement (18.27%)

In recent papers she was focusing on the following fields of study:

Her primary areas of study are Complement system, Macular degeneration, Complement, Disease and Factor H. She has researched Complement system in several fields, including Clusterin, Endosome and Cell biology. Claire L. Harris has included themes like Odds ratio, Alternative complement pathway, Lysosome, Fibrinogen and Immunology in her Macular degeneration study.

She interconnects Downregulation and upregulation and Peritoneal dialysis in the investigation of issues within Immunology. Her work deals with themes such as Drug development and Bioinformatics, which intersect with Disease. Her Factor H research is classified as research in Genetics.

Between 2015 and 2021, her most popular works were:

  • Complement Factor H Inhibits CD47-Mediated Resolution of Inflammation (67 citations)
  • Developments in anti-complement therapy; from disease to clinical trial (38 citations)
  • Developments in anti-complement therapy; from disease to clinical trial (38 citations)

In her most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • Genetics

Claire L. Harris focuses on Disease, Tissue homeostasis, Eculizumab, Complement system and Drug development. Her Tissue homeostasis research includes elements of Risk analysis, Clinical trial, Complement and Drug discovery. Claire L. Harris specializes in Complement system, namely Complement factor I.

Her Biomarker research is multidisciplinary, relying on both Inflammation and Immunology. CD46 is frequently linked to Factor H in her study. Her study in Factor H is interdisciplinary in nature, drawing from both Complement factor B and C4A.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

C3 glomerulopathy: consensus report

Matthew C. Pickering;Vivette D. D'agati;Carla M. Nester;Richard J. Smith.
Kidney International (2013)

557 Citations

Gain-of-function mutations in complement factor B are associated with atypical hemolytic uremic syndrome.

Elena Goicoechea de Jorge;Claire L. Harris;Jorge Esparza-Gordillo;Luis Carreras.
Proceedings of the National Academy of Sciences of the United States of America (2007)

505 Citations

Complement, a target for therapy in inflammatory and degenerative diseases.

B. Paul Morgan;Claire L. Harris;Claire L. Harris.
Nature Reviews Drug Discovery (2015)

342 Citations

Antigen-presenting cell exosomes are protected from complement-mediated lysis by expression of CD55 and CD59.

Aled Clayton;Claire L. Harris;Jacquelyn Court;Malcolm D. Mason.
European Journal of Immunology (2003)

297 Citations

Dimerization of complement factor H-related proteins modulates complement activation in vivo

E. Goicoechea de Jorge;J. J. E. Caesar;T. H. Malik;M. Patel.
Proceedings of the National Academy of Sciences of the United States of America (2013)

266 Citations

Complement: central to innate immunity and bridging to adaptive responses.

B. Paul Morgan;Kevin J. Marchbank;M. Paula Longhi;Claire L. Harris.
Immunology Letters (2005)

241 Citations

Human C3 mutation reveals a mechanism of dense deposit disease pathogenesis and provides insights into complement activation and regulation

Rubén Martínez-Barricarte;Meike Heurich;Francisco Valdes-Cañedo;Eduardo Vazquez-Martul.
Journal of Clinical Investigation (2010)

239 Citations

Common polymorphisms in C3, factor B, and factor H collaborate to determine systemic complement activity and disease risk

Meike Heurich;Ruben Martínez-Barricarte;Nigel J. Francis;Dawn L. Roberts.
Proceedings of the National Academy of Sciences of the United States of America (2011)

220 Citations

Complement therapeutics; history and current progress

B Paul Morgan;Claire L Harris.
Molecular Immunology (2003)

215 Citations

Treatment of B-RAF mutant human tumor cells with a MEK inhibitor requires Bim and is enhanced by a BH3 mimetic

Mark S. Cragg;Elisa S. Jansen;Michele Cook;Claire Harris.
Journal of Clinical Investigation (2008)

214 Citations

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