His primary areas of investigation include Neuroscience, Optogenetics, Serotonin, Hippocampal formation and Transgene. His research combines Glutamate receptor and Neuroscience. His work carried out in the field of Optogenetics brings together such families of science as Dopamine, Cell type, Globus pallidus and Premovement neuronal activity.
His Serotonin research integrates issues from Endocrinology and Arousal. His Serotonergic study incorporates themes from Brain-derived neurotrophic factor, Hypothalamus and Appetite. His Receptor research includes themes of Osteoporosis and Bone remodeling.
His main research concerns Neuroscience, Optogenetics, Cell biology, Internal medicine and Endocrinology. His Optogenetics research incorporates elements of Substantia nigra, Dopamine, Cell type and Stimulation. His study in Cell biology is interdisciplinary in nature, drawing from both Myelin, OLIG2, Genetically modified mouse and Cellular differentiation.
The various areas that Kenji F. Tanaka examines in his Internal medicine study include Schizophrenia and Antipsychotic. His Endocrinology study integrates concerns from other disciplines, such as Serotonergic and Serotonin. His study on Serotonin is covered under Receptor.
His scientific interests lie mostly in Neuroscience, Optogenetics, Cell biology, Astrocyte and Hippocampus. His study explores the link between Neuroscience and topics such as Serotonin that cross with problems in Dopamine. Kenji F. Tanaka combines subjects such as Loss function, Substantia nigra, Medium spiny neuron, Cerebral blood flow and Dorsal raphe nucleus with his study of Optogenetics.
His work in Dorsal raphe nucleus covers topics such as 5-HT1A receptor which are related to areas like Endocrinology. His Cell biology research incorporates themes from Microgliosis, Oligodendrocyte and Channelrhodopsin. In his work, Sleep in non-human animals, Eye movement and Hypothalamus is strongly intertwined with Pons, which is a subfield of Astrocyte.
Kenji F. Tanaka focuses on Neuroscience, Serotonin, Optogenetics, Microglia and Dorsal raphe nucleus. The Serotonin study combines topics in areas such as Catecholamine, Endocrinology and Cell biology. Kenji F. Tanaka has researched Optogenetics in several fields, including Substantia nigra, Medium spiny neuron and Ventrolateral striatum.
The various areas that Kenji F. Tanaka examines in his Microglia study include Myelin oligodendrocyte glycoprotein, Receptor and Demyelinating disease, Multiple sclerosis. He focuses mostly in the field of Receptor, narrowing it down to topics relating to Animal models of depression and, in certain cases, Hippocampus. His Serotonergic research incorporates elements of Orbitofrontal cortex, Transgene, Nucleus accumbens, Hippocampal formation and Stimulation.
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Lrp5 Controls Bone Formation by Inhibiting Serotonin Synthesis in the Duodenum
Vijay K. Yadav;Je Hwang Ryu;Nina Suda;Kenji F. Tanaka.
A serotonin-dependent mechanism explains the leptin regulation of bone mass, appetite, and energy expenditure.
Vijay K. Yadav;Franck Oury;Nina Suda;Zhong Wu Liu.
Near-infrared deep brain stimulation via upconversion nanoparticle–mediated optogenetics
Shuo Chen;Adam Z. Weitemier;Xiao Zeng;Linmeng He.
Hippocampal Memory Traces Are Differentially Modulated by Experience, Time, and Adult Neurogenesis
Christine A. Denny;Mazen A. Kheirbek;Eva L. Alba;Kenji F. Tanaka.
Functional Connectome of the Striatal Medium Spiny Neuron
Nao Chuhma;Kenji F. Tanaka;Kenji F. Tanaka;René Hen;Stephen Rayport.
The Journal of Neuroscience (2011)
Shared Synaptic Pathophysiology in Syndromic and Nonsyndromic Rodent Models of Autism
Stéphane J. Baudouin;Julien Gaudias;Stefan Gerharz;Laetitia Hatstatt.
Optogenetic activation of dorsal raphe serotonin neurons enhances patience for future rewards
Kayoko W. Miyazaki;Katsuhiko Miyazaki;Kenji F. Tanaka;Akihiro Yamanaka.
Current Biology (2014)
Transformation of Astrocytes to a Neuroprotective Phenotype by Microglia via P2Y1 Receptor Downregulation
Youichi Shinozaki;Keisuke Shibata;Keitaro Yoshida;Eiji Shigetomi.
Cell Reports (2017)
Behavioral and serotonergic consequences of decreasing or increasing hippocampus brain-derived neurotrophic factor protein levels in mice.
T. Deltheil;B.P. Guiard;J. Cerdan;D.J. David;D.J. David.
Optogenetic Manipulation of Activity and Temporally Controlled Cell-Specific Ablation Reveal a Role for MCH Neurons in Sleep/Wake Regulation
Tomomi Tsunematsu;Tomomi Tsunematsu;Takafumi Ueno;Sawako Tabuchi;Sawako Tabuchi;Sawako Tabuchi;Ayumu Inutsuka.
The Journal of Neuroscience (2014)
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