Member of the Association of American Physicians
The scientist’s investigation covers issues in Immunology, Complement system, Alternative complement pathway, Arthritis and Rheumatoid arthritis. His studies deal with areas such as Receptor, Internal medicine and Disease as well as Immunology. V. Michael Holers has included themes like Ischemia and In vivo in his Complement system study.
His Alternative complement pathway study integrates concerns from other disciplines, such as Molecular biology, Complement factor B, Kidney and Cell biology. His work in the fields of Inflammatory arthritis overlaps with other areas such as GNLY. His Rheumatoid arthritis research integrates issues from TNFAIP3, Gene, Autoantibody and Autoimmunity.
V. Michael Holers mainly investigates Immunology, Complement system, Alternative complement pathway, Rheumatoid arthritis and Molecular biology. Arthritis, Antibody, Autoantibody, Complement receptor and Complement are the core of his Immunology study. His research in Complement system tackles topics such as Kidney which are related to areas like Ischemia.
His Alternative complement pathway research focuses on Complement factor B and how it relates to Complement factor I. His study focuses on the intersection of Rheumatoid arthritis and fields such as Autoimmunity with connections in the field of Autoimmune disease. His biological study spans a wide range of topics, including Complement receptor 2 and Antigen.
V. Michael Holers mainly focuses on Immunology, Rheumatoid arthritis, Arthritis, Complement system and Antibody. His Immunology and Autoimmune disease, Autoimmunity, Inflammation, Proinflammatory cytokine and Cytokine investigations all form part of his Immunology research activities. His Rheumatoid arthritis research includes elements of Autoantibody and Disease.
V. Michael Holers has researched Arthritis in several fields, including Molecular biology and Neutrophil extracellular traps. The concepts of his Molecular biology study are interwoven with issues in CD8 and Mass cytometry. The various areas that V. Michael Holers examines in his Complement system study include In vivo and Cell biology.
His primary scientific interests are in Immunology, Rheumatoid arthritis, Complement system, Arthritis and Autoantibody. His research in Immunology intersects with topics in Idiopathic pulmonary fibrosis and Genotype. His Rheumatoid arthritis research is multidisciplinary, incorporating perspectives in Gastroenterology, Autoimmunity and Intensive care medicine.
In general Complement system, his work in Alternative complement pathway is often linked to In patient linking many areas of study. His research in Arthritis intersects with topics in Molecular biology, Cell, Neutrophil extracellular traps and Mass cytometry. He combines subjects such as Disease and Rheumatoid factor with his study of Autoantibody.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Arthritis Critically Dependent on Innate Immune System Players
Hong Ji;Hong Ji;Koichiro Ohmura;Koichiro Ohmura;Umar Mahmood;David M Lee.
Complement C5a receptors and neutrophils mediate fetal injury in the antiphospholipid syndrome
Guillermina Girardi;Jessica Berman;Patricia Redecha;Lynn Spruce.
Journal of Clinical Investigation (2003)
Complement C3 activation is required for antiphospholipid antibody-induced fetal loss.
V. Michael Holers;Guillermina Girardi;Lian Mo;Joel M. Guthridge.
Journal of Experimental Medicine (2002)
Antibodies against citrullinated proteins enhance tissue injury in experimental autoimmune arthritis
Kristine A. Kuhn;Liudmila Kulik;Beren Tomooka;Kristin J. Braschler.
Journal of Clinical Investigation (2006)
The central role of the alternative complement pathway in human disease.
Joshua M. Thurman;V. Michael Holers.
Journal of Immunology (2006)
Complement activation induces dysregulation of angiogenic factors and causes fetal rejection and growth restriction
Guillermina Girardi;Dmitry Yarilin;Joshua M. Thurman;V. Michael Holers.
Journal of Experimental Medicine (2006)
Identification of a central role for complement in osteoarthritis
Qian Wang;Andrew L Rozelle;Christin M Lepus;Carla R Scanzello.
Nature Medicine (2011)
C3 glomerulopathy: consensus report
Matthew C. Pickering;Vivette D. D'agati;Carla M. Nester;Richard J. Smith.
Kidney International (2013)
Autoantibody epitope spreading in the pre-clinical phase predicts progression to rheumatoid arthritis.
Jeremy Sokolove;Jeremy Sokolove;Reuven Bromberg;Reuven Bromberg;Kevin D. Deane;Lauren J. Lahey.
PLOS ONE (2012)
REL, encoding a member of the NF-kappaB family of transcription factors, is a newly defined risk locus for rheumatoid arthritis.
Peter K. Gregersen;Chistopher I. Amos;Annette T. Lee;Yue Lu.
Nature Genetics (2009)
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