D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Immunology D-index 76 Citations 19,338 212 World Ranking 784 National Ranking 444
Medicine D-index 82 Citations 21,809 252 World Ranking 8603 National Ranking 4668

Research.com Recognitions

Awards & Achievements

Member of the Association of American Physicians

Overview

What is he best known for?

The fields of study he is best known for:

  • Internal medicine
  • Gene
  • Immune system

The scientist’s investigation covers issues in Immunology, Complement system, Alternative complement pathway, Arthritis and Rheumatoid arthritis. His studies deal with areas such as Receptor, Internal medicine and Disease as well as Immunology. V. Michael Holers has included themes like Ischemia and In vivo in his Complement system study.

His Alternative complement pathway study integrates concerns from other disciplines, such as Molecular biology, Complement factor B, Kidney and Cell biology. His work in the fields of Inflammatory arthritis overlaps with other areas such as GNLY. His Rheumatoid arthritis research integrates issues from TNFAIP3, Gene, Autoantibody and Autoimmunity.

His most cited work include:

  • Arthritis Critically Dependent on Innate Immune System Players (625 citations)
  • Complement C5a receptors and neutrophils mediate fetal injury in the antiphospholipid syndrome (535 citations)
  • Complement C3 activation is required for antiphospholipid antibody-induced fetal loss. (500 citations)

What are the main themes of his work throughout his whole career to date?

V. Michael Holers mainly investigates Immunology, Complement system, Alternative complement pathway, Rheumatoid arthritis and Molecular biology. Arthritis, Antibody, Autoantibody, Complement receptor and Complement are the core of his Immunology study. His research in Complement system tackles topics such as Kidney which are related to areas like Ischemia.

His Alternative complement pathway research focuses on Complement factor B and how it relates to Complement factor I. His study focuses on the intersection of Rheumatoid arthritis and fields such as Autoimmunity with connections in the field of Autoimmune disease. His biological study spans a wide range of topics, including Complement receptor 2 and Antigen.

He most often published in these fields:

  • Immunology (49.51%)
  • Complement system (38.83%)
  • Alternative complement pathway (23.79%)

What were the highlights of his more recent work (between 2016-2021)?

  • Immunology (49.51%)
  • Rheumatoid arthritis (20.39%)
  • Arthritis (16.26%)

In recent papers he was focusing on the following fields of study:

V. Michael Holers mainly focuses on Immunology, Rheumatoid arthritis, Arthritis, Complement system and Antibody. His Immunology and Autoimmune disease, Autoimmunity, Inflammation, Proinflammatory cytokine and Cytokine investigations all form part of his Immunology research activities. His Rheumatoid arthritis research includes elements of Autoantibody and Disease.

V. Michael Holers has researched Arthritis in several fields, including Molecular biology and Neutrophil extracellular traps. The concepts of his Molecular biology study are interwoven with issues in CD8 and Mass cytometry. The various areas that V. Michael Holers examines in his Complement system study include In vivo and Cell biology.

Between 2016 and 2021, his most popular works were:

  • Defining inflammatory cell states in rheumatoid arthritis joint synovial tissues by integrating single-cell transcriptomics and mass cytometry. (229 citations)
  • Complement proteins bind to nanoparticle protein corona and undergo dynamic exchange in vivo (206 citations)
  • Genetic and environmental risk factors for rheumatoid arthritis (134 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Internal medicine
  • Immune system

His primary scientific interests are in Immunology, Rheumatoid arthritis, Complement system, Arthritis and Autoantibody. His research in Immunology intersects with topics in Idiopathic pulmonary fibrosis and Genotype. His Rheumatoid arthritis research is multidisciplinary, incorporating perspectives in Gastroenterology, Autoimmunity and Intensive care medicine.

In general Complement system, his work in Alternative complement pathway is often linked to In patient linking many areas of study. His research in Arthritis intersects with topics in Molecular biology, Cell, Neutrophil extracellular traps and Mass cytometry. He combines subjects such as Disease and Rheumatoid factor with his study of Autoantibody.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Arthritis Critically Dependent on Innate Immune System Players

Hong Ji;Hong Ji;Koichiro Ohmura;Koichiro Ohmura;Umar Mahmood;David M Lee.
Immunity (2002)

751 Citations

Complement C5a receptors and neutrophils mediate fetal injury in the antiphospholipid syndrome

Guillermina Girardi;Jessica Berman;Patricia Redecha;Lynn Spruce.
Journal of Clinical Investigation (2003)

720 Citations

Complement C3 activation is required for antiphospholipid antibody-induced fetal loss.

V. Michael Holers;Guillermina Girardi;Lian Mo;Joel M. Guthridge.
Journal of Experimental Medicine (2002)

651 Citations

Antibodies against citrullinated proteins enhance tissue injury in experimental autoimmune arthritis

Kristine A. Kuhn;Liudmila Kulik;Beren Tomooka;Kristin J. Braschler.
Journal of Clinical Investigation (2006)

608 Citations

The central role of the alternative complement pathway in human disease.

Joshua M. Thurman;V. Michael Holers.
Journal of Immunology (2006)

559 Citations

Complement activation induces dysregulation of angiogenic factors and causes fetal rejection and growth restriction

Guillermina Girardi;Dmitry Yarilin;Joshua M. Thurman;V. Michael Holers.
Journal of Experimental Medicine (2006)

500 Citations

Identification of a central role for complement in osteoarthritis

Qian Wang;Andrew L Rozelle;Christin M Lepus;Carla R Scanzello.
Nature Medicine (2011)

487 Citations

C3 glomerulopathy: consensus report

Matthew C. Pickering;Vivette D. D'agati;Carla M. Nester;Richard J. Smith.
Kidney International (2013)

487 Citations

Autoantibody epitope spreading in the pre-clinical phase predicts progression to rheumatoid arthritis.

Jeremy Sokolove;Jeremy Sokolove;Reuven Bromberg;Reuven Bromberg;Kevin D. Deane;Lauren J. Lahey.
PLOS ONE (2012)

416 Citations

REL, encoding a member of the NF-kappaB family of transcription factors, is a newly defined risk locus for rheumatoid arthritis.

Peter K. Gregersen;Chistopher I. Amos;Annette T. Lee;Yue Lu.
Nature Genetics (2009)

372 Citations

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