His primary areas of study are Immunology, Transplantation, Kidney, Complement system and Internal medicine. His research in Immune system, Antigen, Innate immune system, Inflammation and Acquired immune system are components of Immunology. His Transplantation research is multidisciplinary, incorporating perspectives in Allotype, Priming, Bone marrow and FOXP3.
He has researched Kidney in several fields, including Reperfusion injury, Receptor and Pathology. He interconnects Nephropathy, Pathogenesis and Ischemia in the investigation of issues within Complement system. The Internal medicine study which covers Endocrinology that intersects with Proto-oncogene tyrosine-protein kinase Src.
Immunology, Transplantation, Complement system, Kidney and Cell biology are his primary areas of study. His work in Immune system, Complement, Innate immune system, Acquired immune system and Inflammation are all subfields of Immunology research. His biological study spans a wide range of topics, including Reperfusion injury and Antigen.
His study ties his expertise on Ischemia together with the subject of Complement system. His Kidney study incorporates themes from Pathogenesis and Pathology. He does research in Cell biology, focusing on Function specifically.
Steven H. Sacks mostly deals with Immunology, Complement system, Transplantation, Cell biology and Lectin pathway. His studies deal with areas such as Disease and Kidney transplantation as well as Immunology. His work carried out in the field of Complement system brings together such families of science as Reperfusion injury, Ischemia and Innate immune system, Collectin.
His Transplantation research is multidisciplinary, incorporating elements of Acquired immune system, Cancer research, Kidney and Complement membrane attack complex. His Cell biology research incorporates themes from Lineage and Ligand. His Lectin pathway research includes themes of Molecular biology, Lectin, Mannan-binding lectin and Ficolin.
His main research concerns Immunology, Complement system, Transplantation, Immune system and Lectin pathway. His Immunology study combines topics from a wide range of disciplines, such as Acute kidney injury and Kidney transplantation. The various areas that he examines in his Complement system study include Innate immune system, Ischemia and Cell biology.
His Innate immune system research is multidisciplinary, relying on both Classical complement pathway and Complement receptor. His Transplantation study is concerned with the field of Internal medicine as a whole. His research integrates issues of Mannan-binding lectin, Ficolin, Inflammation, Biochemistry and Complement in his study of Lectin pathway.
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Banff 2013 meeting report: inclusion of c4d-negative antibody-mediated rejection and antibody-associated arterial lesions
M. Haas;B. Sis;L. C. Racusen;K. Solez.
American Journal of Transplantation (2014)
Local synthesis of complement component C3 regulates acute renal transplant rejection.
Julian R. Pratt;Shamim A. Basheer;Steven H. Sacks.
Nature Medicine (2002)
Predominant role for C5b-9 in renal ischemia/reperfusion injury
Wuding Zhou;Conrad A. Farrar;Katsushige Abe;Julian R. Pratt.
Journal of Clinical Investigation (2000)
Urinary tract infections: new insights into a common problem
R Kucheria;P Dasgupta;S H Sacks;M S Khan.
Postgraduate Medical Journal (2005)
Apical Proteins Stimulate Complement Synthesis by Cultured Human Proximal Tubular Epithelial Cells
Sydney Tang;Neil S. Sheerin;Wuding Zhou;Zarin Brown.
Journal of The American Society of Nephrology (1999)
Local extravascular pool of C3 is a determinant of postischemic acute renal failure
Conrad A. Farrar;Wuding Zhou;Tao Lin;Steven H. Sacks.
The FASEB Journal (2006)
The role of complement in the early immune response to transplantation
Steven H. Sacks;Wuding Zhou.
Nature Reviews Immunology (2012)
Fc-dependent depletion of activated T cells occurs through CD40L-specific antibody rather than costimulation blockade
Nicola J Monk;Roseanna E G Hargreaves;James E Marsh;Conrad A Farrar.
Nature Medicine (2003)
Targeting of mannan-binding lectin-associated serine protease-2 confers protection from myocardial and gastrointestinal ischemia/reperfusion injury
Wilhelm J. Schwaeble;Nicholas J. Lynch;James E. Clark;Michael Marber.
Proceedings of the National Academy of Sciences of the United States of America (2011)
Local production and activation of complement up-regulates the allostimulatory function of dendritic cells through C3a-C3aR interaction.
Qi Peng;Ke Li;Katie Anderson;Conrad A. Farrar.
Blood (2008)
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