What is she best known for?
The fields of study she is best known for:
- Gene
- Immune system
- Internal medicine
Immunology, Autoimmunity, IL-2 receptor, FOXP3 and T cell are her primary areas of study.
Her Immunology research is multidisciplinary, relying on both Type 1 diabetes and Disease.
The concepts of her Autoimmunity study are interwoven with issues in PTPN22, Function, B cell, Signal transduction and Phosphorylation.
She combines subjects such as Antigen-presenting cell, Molecular biology, T-cell receptor, Interleukin 2 and Cell biology with her study of IL-2 receptor.
Her biological study spans a wide range of topics, including Regulatory T cell, Psoriasis, CD4 antigen and Pathogenesis.
Her T cell study frequently draws connections between related disciplines such as Cytotoxic T cell.
Her most cited work include:
- Induction of FoxP3 and acquisition of T regulatory activity by stimulated human CD4 + CD25 – T cells (1172 citations)
- Mechanisms of impaired regulation by CD4+CD25+FOXP3+ regulatory T cells in human autoimmune diseases (586 citations)
- Type 1 diabetes immunotherapy using polyclonal regulatory T cells (454 citations)
What are the main themes of her work throughout her whole career to date?
Her primary areas of investigation include Immunology, Autoimmunity, T cell, Internal medicine and Antigen.
Her Immunology and Rheumatoid arthritis, Immune system, FOXP3, IL-2 receptor and Autoantibody investigations all form part of her Immunology research activities.
Her FOXP3 research is multidisciplinary, incorporating perspectives in Regulatory T cell, Cancer research, Cytokine and Cell biology.
Her IL-2 receptor study improves the overall literature in Cytotoxic T cell.
Her Autoimmunity research includes elements of Autoimmune disease, Signal transduction, Type 1 diabetes and B cell.
The T cell study combines topics in areas such as Epitope, Molecular biology and Major histocompatibility complex.
She most often published in these fields:
- Immunology (54.15%)
- Autoimmunity (23.41%)
- T cell (24.88%)
What were the highlights of her more recent work (between 2018-2021)?
- Immunology (54.15%)
- Rheumatoid arthritis (14.63%)
- Internal medicine (17.07%)
In recent papers she was focusing on the following fields of study:
Jane H. Buckner mainly investigates Immunology, Rheumatoid arthritis, Internal medicine, Immune system and Disease.
Her Immunology study frequently involves adjacent topics like Phenotype.
Her Rheumatoid arthritis study incorporates themes from Autoantibody and Cohort.
Her work deals with themes such as Enhancer and Chromatin, Gene, Quantitative trait locus, which intersect with Immune system.
Her Autoimmunity research incorporates elements of Systemic lupus erythematosus, Type 1 diabetes and Receptor.
Her study in FOXP3 is interdisciplinary in nature, drawing from both IL-2 receptor, NOD mice and Genetic enhancement.
Between 2018 and 2021, her most popular works were:
- Citrullinated Aggrecan Epitopes as Targets of Autoreactive CD4+ T Cells in Patients With Rheumatoid Arthritis. (22 citations)
- Gene editing to induce FOXP3 expression in human CD4+ T cells leads to a stable regulatory phenotype and function. (19 citations)
- Gene editing to induce FOXP3 expression in human CD4+ T cells leads to a stable regulatory phenotype and function. (19 citations)
In her most recent research, the most cited papers focused on:
- Gene
- Immune system
- Internal medicine
Her primary areas of study are Immune system, Immune tolerance, T cell, Immunology and Autoimmunity.
Her research integrates issues of Phenotype, Enhancer, Colocalization and Effector in her study of Immune system.
Her Phenotype study integrates concerns from other disciplines, such as Autoimmune disease, Transcription factor, Cell biology, Genome editing and FOXP3.
Her studies in T cell integrate themes in fields like Synovial Cell, Cell type, Natural killer cell and Synovial membrane.
Her work carried out in the field of Immunology brings together such families of science as Systemic lupus erythematosus and Receptor.
Her Autoimmunity study deals with the bigger picture of Disease.
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