What is he best known for?
The fields of study he is best known for:
His primary scientific interests are in Cell biology, B cell, Immunology, Bruton's tyrosine kinase and Tyrosine kinase.
His Cell biology research integrates issues from Innate immune system and Biochemistry.
His B cell research is multidisciplinary, incorporating elements of B-1 cell, Antigen-presenting cell and Immune system.
His work in Autoimmunity, Germinal center, Antibody, T cell and Antigen are all subfields of Immunology research.
His Bruton's tyrosine kinase research includes themes of SH3 domain, Molecular biology and Kinase.
His studies deal with areas such as Cancer research, Pleckstrin homology domain, Protein kinase A, Point mutation and Tyrosine phosphorylation as well as Tyrosine kinase.
His most cited work include:
- Deficient expression of a B cell cytoplasmic tyrosine kinase in human X-linked agammaglobulinemia (1112 citations)
- CD19+CD24hiCD38hi B Cells Exhibit Regulatory Capacity in Healthy Individuals but Are Functionally Impaired in Systemic Lupus Erythematosus Patients (1100 citations)
- Mutation of unique region of Bruton's tyrosine kinase in immunodeficient XID mice. (772 citations)
What are the main themes of his work throughout his whole career to date?
Immunology, B cell, Molecular biology, Cell biology and Genetic enhancement are his primary areas of study.
His Immunology research focuses on Immune system, Antibody, Autoimmunity, Antigen and Germinal center.
His B cell study incorporates themes from T cell, Cancer research and Autoantibody.
The Molecular biology study combines topics in areas such as Haematopoiesis, Gene, Genome editing, Bruton's tyrosine kinase and Homology directed repair.
His Cell biology research is multidisciplinary, incorporating perspectives in Transcription factor and T-cell receptor.
The various areas that David J. Rawlings examines in his Genetic enhancement study include Virology, Viral vector, Stem cell and Severe combined immunodeficiency, In vivo.
He most often published in these fields:
- Immunology (61.36%)
- B cell (45.72%)
- Molecular biology (38.94%)
What were the highlights of his more recent work (between 2018-2021)?
- Antigen (16.81%)
- Immunology (61.36%)
- Antibody (20.06%)
In recent papers he was focusing on the following fields of study:
David J. Rawlings mostly deals with Antigen, Immunology, Antibody, Genome editing and Molecular biology.
His biological study spans a wide range of topics, including Receptor and Disease.
His Antibody research incorporates elements of Virus and Function.
His work deals with themes such as Haematopoiesis, Stem cell, Recombinant DNA and B-cell receptor, B cell, which intersect with Molecular biology.
David J. Rawlings connects B cell with Translational elongation in his research.
His Immune system research is multidisciplinary, incorporating elements of Phenotype and Cell biology.
Between 2018 and 2021, his most popular works were:
- Functional SARS-CoV-2-Specific Immune Memory Persists after Mild COVID-19. (128 citations)
- Therapeutically relevant engraftment of a CRISPR-Cas9-edited HSC-enriched population with HbF reactivation in nonhuman primates. (42 citations)
- Therapeutically relevant engraftment of a CRISPR-Cas9-edited HSC-enriched population with HbF reactivation in nonhuman primates. (42 citations)
In his most recent research, the most cited papers focused on:
His primary areas of study are Immunology, Transplantation, Molecular biology, Receptor and Stem cell.
His study in Antigen, Virus and Antibody is carried out as part of his studies in Immunology.
The study incorporates disciplines such as Survival rate, Wiskott–Aldrich syndrome and Immunodeficiency in addition to Transplantation.
His research in Molecular biology intersects with topics in Reversion and Homology directed repair.
The various areas that he examines in his Receptor study include T cell, Germinal center, Systemic lupus erythematosus, Interleukin 12 and Autoimmunity.
His biological study spans a wide range of topics, including Humanized mouse, Fetal hemoglobin, Hereditary persistence of fetal hemoglobin and Transfection.
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