2009 - Presidential Award, Clinical Immunology Society
His primary scientific interests are in Immunology, Genetics, Molecular biology, Immunodeficiency and Gene. His study in FOXP3, Immune dysregulation, Immune system, Primary immunodeficiency and IL-2 receptor is carried out as part of his studies in Immunology. Hans D. Ochs works mostly in the field of Immune system, limiting it down to topics relating to Antibody and, in certain cases, Immunity and Lymphocyte.
His work is connected to X chromosome, Wiskott–Aldrich syndrome protein, Mutation, Wiskott–Aldrich syndrome and Genetic linkage, as a part of Genetics. Hans D. Ochs works mostly in the field of Wiskott–Aldrich syndrome protein, limiting it down to topics relating to Genotype and, in certain cases, Actin cytoskeleton organization, as a part of the same area of interest. His Molecular biology research incorporates themes from Point mutation, Hyper IgM syndrome, B cell and Antigen-presenting cell.
Hans D. Ochs focuses on Immunology, Antibody, Immune system, Primary immunodeficiency and Genetics. Hans D. Ochs has researched Immunology in several fields, including Transplantation and Virology. His Primary immunodeficiency study combines topics from a wide range of disciplines, such as Severe combined immunodeficiency and Newborn screening.
His work in Genetics covers topics such as Molecular biology which are related to areas like CD40. His work carried out in the field of FOXP3 brings together such families of science as Enteropathy, IL-2 receptor, IPEX syndrome, Immune dysregulation and Autoimmunity. Hans D. Ochs regularly links together related areas like Wiskott–Aldrich syndrome protein in his Gene studies.
His scientific interests lie mostly in Immunology, Primary immunodeficiency, Immunodeficiency, Immune system and Antibody. The study incorporates disciplines such as Immunologic Deficiency Syndromes, X-linked agammaglobulinemia and Pediatrics, Newborn screening in addition to Primary immunodeficiency. His research integrates issues of Hematopoietic stem cell transplantation, Internal medicine, Wiskott–Aldrich syndrome, Hypogammaglobulinemia and Chronic mucocutaneous candidiasis in his study of Immunodeficiency.
He interconnects Autoantibody and Gene in the investigation of issues within Immune system. As a part of the same scientific family, Hans D. Ochs mostly works in the field of Immune dysregulation, focusing on FOXP3 and, on occasion, IPEX syndrome. His Disease research incorporates elements of Alternative medicine and Genetics.
Hans D. Ochs mainly investigates Immunology, Primary immunodeficiency, Immunodeficiency, Expert committee and Disease. His study ties his expertise on Hyper IgM syndrome together with the subject of Immunology. His Primary immunodeficiency research is multidisciplinary, incorporating perspectives in Newborn screening, Dock8, Common variable immunodeficiency and Family medicine.
His research in Immunodeficiency intersects with topics in Hematopoietic stem cell transplantation, Internal medicine, Malignancy, Cohort and Immune dysregulation. His Disease research is multidisciplinary, relying on both Epidemiology and Intensive care medicine. His Immune system research integrates issues from Enteropathy and Autoantibody.
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The immune dysregulation, polyendocrinopathy, enteropathy, X-linked syndrome (IPEX) is caused by mutations of FOXP3.
Craig L. Bennett;Jacinda Christie;Fred Ramsdell;Mary E. Brunkow.
Nature Genetics (2001)
Chronic granulomatous disease. Report on a national registry of 368 patients.
Jerry A. Winkelstein;Mary C. Marino;Richard B. Johnston;John Boyle.
Isolation of a novel gene mutated in Wiskott-Aldrich syndrome
Jonathan M.J. Derry;Hans D. Ochs;Uta Francke;Uta Francke.
The CD40 ligand, gp39, is defective in activated T cells from patients with X-linked hyper-IgM syndrome
Alejandro Aruffo;Alejandro Aruffo;Mary Farrington;Diane Hollenbaugh;Xu Li.
Single-cell analysis of normal and FOXP3-mutant human T cells: FOXP3 expression without regulatory T cell development.
Marc A. Gavin;Troy R. Torgerson;Evan Houston;Paul deRoos.
Proceedings of the National Academy of Sciences of the United States of America (2006)
Immune dysregulation, polyendocrinopathy, enteropathy, and X-linked inheritance (IPEX), a syndrome of systemic autoimmunity caused by mutations of FOXP3, a critical regulator of T-cell homeostasis.
Eleonora Gambineri;Troy R. Torgerson;Hans D. Ochs.
Current Opinion in Rheumatology (2003)
CTLA4Ig-mediated blockade of T cell costimulation in patients with psoriasis vulgaris
Judith R. Abrams;Mark G. Lebwohl;Cynthia A. Guzzo;Brian V. Jegasothy.
Journal of Clinical Investigation (1999)
X-linked agammaglobulinemia: report on a United States registry of 201 patients.
Jerry A. Winkelstein;Mary C. Marino;Howard M. Lederman;Stacie M. Jones.
Primary Immunodeficiency Diseases: an Update on the Classification from the International Union of Immunological Societies Expert Committee for Primary Immunodeficiency 2015
Capucine Picard;Waleed Al-Herz;Aziz Bousfiha;Jean Laurent Casanova.
Journal of Clinical Immunology (2015)
X-Linked Lymphoproliferative Disease 2b4 Molecules Displaying Inhibitory Rather than Activating Function Are Responsible for the Inability of Natural Killer Cells to Kill Epstein-Barr Virus–Infected Cells
Silvia Parolini;Cristina Bottino;Michela Falco;Raffaella Augugliaro.
Journal of Experimental Medicine (2000)
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