Bodo Grimbacher focuses on Immunology, Common variable immunodeficiency, Immunodeficiency, Primary immunodeficiency and Hypogammaglobulinemia. His research integrates issues of Mutation and Genetics in his study of Immunology. His Common variable immunodeficiency research is multidisciplinary, relying on both Enteropathy, T cell, Selective IgA deficiency, Autoimmunity and B-cell activating factor.
Within one scientific family, Bodo Grimbacher focuses on topics pertaining to Hematopoietic stem cell transplantation under Immunodeficiency, and may sometimes address concerns connected to Inflammatory bowel disease and Colitis. He interconnects Genotype, Molecular biology, Immune system and Antigen in the investigation of issues within Hypogammaglobulinemia. His biological study spans a wide range of topics, including Dock8 and Hyperimmunoglobulin E syndrome.
His primary scientific interests are in Immunology, Common variable immunodeficiency, Primary immunodeficiency, Immunodeficiency and Genetics. His Immunology research integrates issues from Hematopoietic stem cell transplantation and Disease. His Hematopoietic stem cell transplantation study incorporates themes from DOCK8 Deficiency and Inflammatory bowel disease.
His work in Common variable immunodeficiency covers topics such as B cell which are related to areas like Molecular biology. His research in Primary immunodeficiency intersects with topics in T cell, Immune dysregulation, Chronic mucocutaneous candidiasis and Pediatrics. His studies deal with areas such as Missense mutation, Internal medicine and Virology as well as Immunodeficiency.
Bodo Grimbacher mainly focuses on Immunology, Common variable immunodeficiency, Primary immunodeficiency, Immunodeficiency and Immune dysregulation. The various areas that Bodo Grimbacher examines in his Immunology study include Penetrance and Hematopoietic stem cell transplantation. The Common variable immunodeficiency study combines topics in areas such as Hypogammaglobulinemia, Internal medicine, Disease, Cohort and Pediatrics.
His Primary immunodeficiency study combines topics in areas such as Immunophenotyping, Peripheral blood and Receiver operating characteristic. His Immunodeficiency research is multidisciplinary, incorporating elements of Missense mutation, Compound heterozygosity, Progressive multifocal leukoencephalopathy and STAT3, Glycoprotein 130. His study on Immune dysregulation also encompasses disciplines like
His primary areas of study are Immunology, Common variable immunodeficiency, Primary immunodeficiency, Immune dysregulation and Immunodeficiency. He studies Autoimmunity which is a part of Immunology. The concepts of his Common variable immunodeficiency study are interwoven with issues in Guideline, Antibiotics and Disease.
Bodo Grimbacher focuses mostly in the field of Primary immunodeficiency, narrowing it down to matters related to Cohort and, in some cases, Pediatrics, Risk factor, Disease burden and Comorbidity. His work carried out in the field of Immune dysregulation brings together such families of science as CTLA-4, Loss function, Immunosuppression and Hypogammaglobulinemia. His Immunodeficiency study integrates concerns from other disciplines, such as Missense mutation, Interleukin 6, Leukemia inhibitory factor, Oncostatin M and Cytokine receptor.
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Inflammatory bowel disease and mutations affecting the interleukin-10 receptor.
Erik-Oliver Glocker;Daniel Kotlarz;Kaan Boztug;E. Michael Gertz.
The New England Journal of Medicine (2009)
STAT3 Mutations in the Hyper-IgE Syndrome
Steven M. Holland;Frank R. DeLeo;Houda Z. Elloumi;Amy P. Hsu.
The New England Journal of Medicine (2007)
Hyper-IgE Syndrome with Recurrent Infections — An Autosomal Dominant Multisystem Disorder
Bodo Grimbacher;Steven M. Holland;John I. Gallin;Frank Greenberg.
The New England Journal of Medicine (1999)
Homozygous loss of ICOS is associated with adult-onset common variable immunodeficiency
Bodo Grimbacher;Andreas Hutloff;Michael Schlesier;Erik Glocker.
Nature Immunology (2003)
A Homozygous CARD9 Mutation in a Family with Susceptibility to Fungal Infections
Erik-Oliver Glocker;Andre Hennigs;Mohammad Nabavi;Alejandro A. Schäffer.
The New England Journal of Medicine (2009)
Common variable immunodeficiency disorders: division into distinct clinical phenotypes
Helen Chapel;Mary Lucas;Martin Lee;Janne Bjorkander.
Mutations in TNFRSF13B encoding TACI are associated with common variable immunodeficiency in humans
U Salzer;H M Chapel;A D B Webster;Q Pan-Hammarström.
Nature Genetics (2005)
Deficiency of Th17 cells in hyper IgE syndrome due to mutations in STAT3
Cindy S. Ma;Gary Y.J. Chew;Nicholas Simpson;Archana Priyadarshi.
Journal of Experimental Medicine (2008)
Autosomal dominant immune dysregulation syndrome in humans with CTLA4 mutations.
Desireé Schubert;Desireé Schubert;Claudia Bode;Rupert Kenefeck;Tie Zheng Hou.
Nature Medicine (2014)
An antibody-deficiency syndrome due to mutations in the CD19 gene
Menno C van Zelm;Ismail Reisli;Mirjam van der Burg;Diana Castaño.
The New England Journal of Medicine (2006)
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