What is she best known for?
The fields of study she is best known for:
Immunology, Immunity, Disease, Mycobacterium tuberculosis and Tuberculosis are her primary areas of study.
Her Immunology research integrates issues from Mutation, Germline mutation, Allele and Cellular differentiation.
Stéphanie Boisson-Dupuis has researched Immunity in several fields, including Immunodeficiency, Interferon, Interferon type I, Mucocutaneous Candidiasis and Chronic mucocutaneous candidiasis.
Her Disease research incorporates elements of Evolutionary pressure, Gene, DNA sequencing, Coding region and Exome.
Her Mycobacterium tuberculosis study integrates concerns from other disciplines, such as Proband, Internal medicine, Asymptomatic, Survival analysis and Penetrance.
Her work on Miliary tuberculosis as part of general Tuberculosis research is frequently linked to Small sample, bridging the gap between disciplines.
Her most cited work include:
- Gain-of-function human STAT1 mutations impair IL-17 immunity and underlie chronic mucocutaneous candidiasis (557 citations)
- Gain-of-function human STAT1 mutations impair IL-17 immunity and underlie chronic mucocutaneous candidiasis (557 citations)
- IRF8 mutations and human dendritic-cell immunodeficiency. (447 citations)
What are the main themes of her work throughout her whole career to date?
Stéphanie Boisson-Dupuis mainly investigates Immunology, Genetics, Tuberculosis, Allele and Immunity.
Her studies in Immunology integrate themes in fields like Mutation and Chronic mucocutaneous candidiasis, Disease.
Her work carried out in the field of Chronic mucocutaneous candidiasis brings together such families of science as Missense mutation, Candida albicans and Immunodeficiency.
Her studies deal with areas such as Internal medicine and Vaccination as well as Tuberculosis.
The Allele study combines topics in areas such as Receptor, Dominance and Genotype.
She combines subjects such as Medical microbiology, Etiology, Cytokine and Allelic heterogeneity with her study of Immunity.
She most often published in these fields:
- Immunology (137.77%)
- Genetics (66.52%)
- Tuberculosis (64.38%)
What were the highlights of her more recent work (between 2017-2021)?
- Immunity (53.22%)
- Immunology (137.77%)
- Tuberculosis (64.38%)
In recent papers she was focusing on the following fields of study:
Stéphanie Boisson-Dupuis focuses on Immunity, Immunology, Tuberculosis, Allele and Genetics.
Her Immunity research is multidisciplinary, incorporating perspectives in Myeloid, Virus, Mendelian inheritance and Pneumonia.
Stéphanie Boisson-Dupuis merges Immunology with Tyrosine kinase 2 in her study.
Her research in Tuberculosis intersects with topics in Infectious disease and Disease.
She interconnects Mutation, Receptor and Mutant in the investigation of issues within Allele.
Her study in the fields of Negative selection, Inheritance and Coding region under the domain of Genetics overlaps with other disciplines such as Mechanism.
Between 2017 and 2021, her most popular works were:
- Inborn errors of type I IFN immunity in patients with life-threatening COVID-19 (426 citations)
- A Global Effort to Define the Human Genetics of Protective Immunity to SARS-CoV-2 Infection. (72 citations)
- Tuberculosis and impaired IL-23–dependent IFN-γ immunity in humans homozygous for a common TYK2 missense variant (70 citations)
In her most recent research, the most cited papers focused on:
Stéphanie Boisson-Dupuis mainly focuses on Immunity, Immunology, Allele, Cytokine and Virus.
Her Immunity study combines topics from a wide range of disciplines, such as Myeloid, Mycobacterium bovis, Mycobacterium tuberculosis and Interferon gamma.
Her Immunology research is multidisciplinary, relying on both Disease, Tuberculosis and Human genetics.
Her Allele study incorporates themes from Mutation, Missense mutation, Receptor and Mendelian inheritance.
Her Cytokine research integrates issues from Natural killer T cell, Penetrance, Interleukin 12 and Immune system.
In her study, which falls under the umbrella issue of Virus, Gene silencing, Viral replication, Influenza A virus and STAT1 is strongly linked to Pneumonia.
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