Elissa K. Deenick mainly focuses on Immunology, Cellular differentiation, Cytotoxic T cell, Immune system and B-1 cell. Her work carried out in the field of Immunology brings together such families of science as Gene rearrangement and Chronic Candidiasis, Candida albicans. Her study focuses on the intersection of Cellular differentiation and fields such as Cell biology with connections in the field of Naive B cell.
Her research in Cytotoxic T cell intersects with topics in Haploinsufficiency, CTLA4 Haploinsufficiency, Immune dysregulation and Germline. As a part of the same scientific family, Elissa K. Deenick mostly works in the field of Immune system, focusing on Germline mutation and, on occasion, PI3K/AKT/mTOR pathway, Protein kinase B, IL-2 receptor and Molecular biology. Her Immunity research is multidisciplinary, incorporating perspectives in Humoral immunity, Antibody, Autoimmunity and Function.
Her primary areas of study are Immunology, Cell biology, Immune system, Cytotoxic T cell and Cellular differentiation. Her Immunology and B cell, Antibody, Humoral immunity, Immunity and T cell investigations all form part of her Immunology research activities. Elissa K. Deenick has included themes like Receptor, Naive B cell, CD40 and Transcription factor in her Cell biology study.
The concepts of her Immune system study are interwoven with issues in Mutation, Germinal center and Cytokine. Her work investigates the relationship between Cytotoxic T cell and topics such as CD8 that intersect with problems in Cancer research and Virology. Her Cellular differentiation research includes themes of Cell culture, Germline mutation, Cell growth and Antigen.
Her main research concerns Immunology, Immune system, Antibody, Cancer research and Autoimmunity. Her study in B cell and Humoral immunity is done as part of Immunology. Her work is dedicated to discovering how B cell, Peripheral tolerance are connected with Germinal center and other disciplines.
Her study on Immunodeficiency, Immune regulation and Immune dysregulation is often connected to Goldilocks principle as part of broader study in Immune system. Her B-cell activation study in the realm of Antibody interacts with subjects such as 2019-20 coronavirus outbreak and Severe acute respiratory syndrome coronavirus 2. Her Cancer research study incorporates themes from Everolimus, CD8 and CTLA4 Haploinsufficiency.
Elissa K. Deenick focuses on CD8, Cancer research, Cell biology, Transcription factor and Transcriptome. CD8 is the subject of her research, which falls under Immune system. Her research integrates issues of Plasma cell, Carboxyfluorescein succinimidyl ester, Cytokine and Immunity in her study of Cancer research.
Her Cell biology research integrates issues from Cell, Cellular differentiation, Mucosal associated invariant T cell, Cell growth and Signaling lymphocytic activation molecule.
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Immune dysregulation in human subjects with heterozygous germline mutations in CTLA4
Hye Sun Kuehn;Weiming Ouyang;Bernice Lo;Elissa K. Deenick;Elissa K. Deenick.
The origins, function, and regulation of T follicular helper cells.
Cindy S. Ma;Elissa K. Deenick;Elissa K. Deenick;Marcel Batten;Marcel Batten;Stuart G. Tangye;Stuart G. Tangye.
Journal of Experimental Medicine (2012)
The good, the bad and the ugly - TFH cells in human health and disease.
Stuart G. Tangye;Cindy S. Ma;Cindy S. Ma;Robert Brink;Robert Brink;Elissa K. Deenick;Elissa K. Deenick.
Nature Reviews Immunology (2013)
Dominant-activating germline mutations in the gene encoding the PI(3)K catalytic subunit p110δ result in T cell senescence and human immunodeficiency
Carrie L Lucas;Hye Sun Kuehn;Fang Zhao;Julie E Niemela.
Nature Immunology (2014)
B cell-intrinsic signaling through IL-21 receptor and STAT3 is required for establishing long-lived antibody responses in humans
Danielle T. Avery;Elissa K. Deenick;Elissa K. Deenick;Cindy S. Ma;Cindy S. Ma;Santi Suryani;Santi Suryani.
Journal of Experimental Medicine (2010)
Early commitment of naïve human CD4(+) T cells to the T follicular helper (T(FH)) cell lineage is induced by IL-12.
Cindy S Ma;Santi Suryani;Santi Suryani;Danielle T Avery;Anna Chan.
Immunology and Cell Biology (2009)
Follicular helper T cell differentiation requires continuous antigen presentation that is independent of unique B cell signaling
Elissa K. Deenick;Anna Chan;Cindy S. Ma;Cindy S. Ma;Dominique Gatto;Dominique Gatto.
Impairment of immunity to Candida and Mycobacterium in humans with bi-allelic RORC mutations
Satoshi Okada;Satoshi Okada;Janet G Markle;Elissa K Deenick;Elissa K Deenick;Federico Mele.
Intrinsic Differences in the Proliferation of Naive and Memory Human B Cells as a Mechanism for Enhanced Secondary Immune Responses
Stuart G. Tangye;Stuart G. Tangye;Danielle T. Avery;Elissa K. Deenick;Elissa K. Deenick;Philip D. Hodgkin;Philip D. Hodgkin.
Journal of Immunology (2003)
Functional STAT3 deficiency compromises the generation of human T follicular helper cells
Cindy S. Ma;Cindy S. Ma;Danielle T. Avery;Anna Chan;Marcel Batten;Marcel Batten.
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