His primary areas of study are Immunology, Germinal center, B cell, Cellular differentiation and Cell biology. His work is connected to Antigen, Antibody, Autoimmunity, Humoral immunity and Adoptive cell transfer, as a part of Immunology. His Germinal center study incorporates themes from Plasma cell, Molecular biology, CD40 and Follicular dendritic cells.
His B cell study which covers B-1 cell that intersects with Naive B cell. His Cellular differentiation study deals with B-cell activating factor intersecting with Rheumatoid arthritis, T cell and Cell signaling. His Cell biology research incorporates themes from Inhibitor of apoptosis, Programmed cell death, TRAF2 and Autocrine signalling.
His primary areas of investigation include Immunology, Cell biology, Germinal center, B cell and Antigen. His Cell biology research incorporates elements of Receptor, B-cell activating factor, Cellular differentiation and CD40. His work is dedicated to discovering how CD40, Antigen-presenting cell are connected with Interleukin 21 and other disciplines.
Robert Brink interconnects Plasma cell, Somatic hypermutation, Affinity maturation, Molecular biology and Naive B cell in the investigation of issues within Germinal center. His studies in B cell integrate themes in fields like C-C chemokine receptor type 7, CXCL13 and B-1 cell. As a part of the same scientific family, Robert Brink mostly works in the field of Antigen, focusing on Antibody and, on occasion, Virology, Cytoplasm and breakpoint cluster region.
Robert Brink spends much of his time researching Germinal center, B cell, Cell biology, Antibody and Immunology. His work deals with themes such as Plasma cell, Somatic hypermutation and Antigen, which intersect with Germinal center. His research integrates issues of Molecular biology, Protein subunit, Caenorhabditis elegans and Cell cycle in his study of B cell.
The study incorporates disciplines such as Osteoclast, Osteoprotegerin and RANKL in addition to Cell biology. His study focuses on the intersection of Antibody and fields such as Mutation with connections in the field of Allele, Transgene, Acquired immune system and Virology. His studies deal with areas such as Gene silencing and MEDLINE as well as Immunology.
Robert Brink mainly focuses on Immune system, Germinal center, Antibody, Mutation and Plasma cell. His Immune system study integrates concerns from other disciplines, such as TNFAIP3, Cancer research, Transgene and Germline mutation. His Germinal center study incorporates themes from Somatic hypermutation, Antigen, Memory B cell and Cell biology.
The Antibody study combines topics in areas such as Gene and Virology. His Mutation research includes elements of Acquired immune system, Immunity, Allele and Phosphorylation. His research in Plasma cell intersects with topics in Carboxyfluorescein succinimidyl ester, Cytokine, CD8, CXCR3 and FOXP3.
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IAP Antagonists Target cIAP1 to Induce TNFα-Dependent Apoptosis
James E. Vince;W. Wei-Lynn Wong;Nufail Khan;Rebecca Feltham.
Elimination from peripheral lymphoid tissues of self-reactive B lymphocytes recognizing membrane-bound antigens.
Suzanne B. Hartley;Jeffrey Crosbie;Robert Brink;Aaron B. Kantor.
Excess BAFF Rescues Self-Reactive B Cells from Peripheral Deletion and Allows Them to Enter Forbidden Follicular and Marginal Zone Niches
Marilyn Thien;Tri Giang Phan;Sandra Gardam;Michelle Amesbury.
Induction of self-tolerance in mature peripheral B lymphocytes
Christopher C. Goodnow;Jeffrey Crosbie;Helle Jorgensen;Robert A. Brink.
Follicular helper T cells are required for systemic autoimmunity.
Michelle A. Linterman;Robert J. Rigby;Raphael. K. Wong;Di Yu.
Journal of Experimental Medicine (2009)
Antigen recognition strength regulates the choice between extrafollicular plasma cell and germinal center B cell differentiation
Didrik Paus;Tri Giang Phan;Tri Giang Phan;Tyani D. Chan;Tyani D. Chan;Sandra Gardam;Sandra Gardam.
Journal of Experimental Medicine (2006)
Circulating precursor CCR7(lo)PD-1(hi) CXCR5⁺ CD4⁺ T cells indicate Tfh cell activity and promote antibody responses upon antigen reexposure.
Jing He;Louis M Tsai;Yew A Leong;Xin Jack Hu;Xin Jack Hu.
BAFF selectively enhances the survival of plasmablasts generated from human memory B cells
Danielle T. Avery;Susan L. Kalled;Julia I. Ellyard;Christine Ambrose.
Journal of Clinical Investigation (2003)
The good, the bad and the ugly - TFH cells in human health and disease.
Stuart G. Tangye;Cindy S. Ma;Cindy S. Ma;Robert Brink;Robert Brink;Elissa K. Deenick;Elissa K. Deenick.
Nature Reviews Immunology (2013)
Control systems and decision making for antibody production
Christopher C Goodnow;Carola G Vinuesa;Katrina L Randall;Katrina L Randall;Fabienne Mackay.
Nature Immunology (2010)
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