2020 - German National Academy of Sciences Leopoldina - Deutsche Akademie der Naturforscher Leopoldina – Nationale Akademie der Wissenschaften Gynaecology and Paediatrics
His scientific interests lie mostly in Immunology, Internal medicine, Hemophagocytic lymphohistiocytosis, Disease and Immunodeficiency. His Immunology study deals with Severe combined immunodeficiency intersecting with Ectodermal dysplasia. His Hemophagocytic lymphohistiocytosis study integrates concerns from other disciplines, such as Macrophage activation syndrome, Perforin, Familial Hemophagocytic Lymphohistiocytosis, UNC13D and Degranulation.
The concepts of his Disease study are interwoven with issues in Retrospective cohort study, Cohort and Intensive care medicine. His Immunodeficiency research includes themes of P110δ, Neutropenia and Gene mutation. His CD8 research is multidisciplinary, incorporating elements of Virus and Adoptive cell transfer.
His primary scientific interests are in Immunology, Immunodeficiency, Hemophagocytic lymphohistiocytosis, Internal medicine and Virology. His T cell, Virus, Immune system, CD8 and CTL* study are his primary interests in Immunology. His work in Virus tackles topics such as Lymphocytic choriomeningitis which are related to areas like In vivo.
His studies deal with areas such as Molecular biology and Adoptive cell transfer as well as CD8. His work in Immunodeficiency covers topics such as Primary immunodeficiency which are related to areas like Common variable immunodeficiency. As a member of one scientific family, Stephan Ehl mostly works in the field of Hemophagocytic lymphohistiocytosis, focusing on Familial Hemophagocytic Lymphohistiocytosis and, on occasion, UNC13D.
His primary areas of study are Immunology, Hemophagocytic lymphohistiocytosis, Immunodeficiency, Germline and Immune dysregulation. All of his Immunology and Pathogenesis, Inflammation, Immunity, Cytokine and Immune system investigations are sub-components of the entire Immunology study. His work on CD8 as part of general Immune system research is often related to Induced pluripotent stem cell and Autoimmune lymphoproliferative syndrome, thus linking different fields of science.
Stephan Ehl has included themes like Hematopoietic stem cell transplantation, Cytopenia, Familial Hemophagocytic Lymphohistiocytosis and Sepsis in his Hemophagocytic lymphohistiocytosis study. Stephan Ehl works mostly in the field of Immunodeficiency, limiting it down to concerns involving Cohort and, occasionally, Adult patients, Opportunistic infection, Combined immunodeficiencies and Autoimmune Cytopenia. His Immune dysregulation study incorporates themes from PIK3CG, National registry, Genetic diagnosis, Common variable immunodeficiency and Primary immunodeficiency.
Stephan Ehl mostly deals with Immunodeficiency, Immune dysregulation, Hemophagocytic lymphohistiocytosis, Pediatrics and Germline. His Immunodeficiency research includes elements of Epidermolysis bullosa, Concomitant and Bioinformatics. His work in Immune dysregulation addresses issues such as Hematopoietic stem cell transplantation, which are connected to fields such as Genetic diagnosis, Age of onset, Prevalence, Immune system and Cancer research.
His Hemophagocytic lymphohistiocytosis study necessitates a more in-depth grasp of Internal medicine. His work deals with themes such as Opportunistic infection, National registry, Adult patients and Cohort, which intersect with Pediatrics. Stephan Ehl combines subjects such as Myeloid and Mutant with his study of Germline.
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Antigen localisation regulates immune responses in a dose- and time-dependent fashion: a geographical view of immune reactivity.
Rolf M. Zinkernagel;Stephan Ehl;Peter Aichele;Stephan Oehen.
Immunological Reviews (1997)
Autosomal dominant immune dysregulation syndrome in humans with CTLA4 mutations.
Desireé Schubert;Desireé Schubert;Claudia Bode;Rupert Kenefeck;Tie Zheng Hou.
Nature Medicine (2014)
Large deletions and point mutations involving the dedicator of cytokinesis 8 (DOCK8) in the autosomal-recessive form of hyper-IgE syndrome.
Karin R. Engelhardt;Sean McGhee;Sabine Winkler;Atfa Sassi.
The Journal of Allergy and Clinical Immunology (2009)
Selective predisposition to bacterial infections in IRAK-4–deficient children: IRAK-4–dependent TLRs are otherwise redundant in protective immunity
Cheng Lung Ku;Horst Von Bernuth;Horst Von Bernuth;Capucine Picard;Shen Ying Zhang.
Journal of Experimental Medicine (2007)
Familial hemophagocytic lymphohistiocytosis type 5 (FHL-5) is caused by mutations in Munc18-2 and impaired binding to syntaxin 11.
Udo zur Stadt;Jan Rohr;Wenke Seifert;Wenke Seifert;Florian Koch.
American Journal of Human Genetics (2009)
Lambda interferon renders epithelial cells of the respiratory and gastrointestinal tracts resistant to viral infections.
Markus Mordstein;Eva Neugebauer;Vanessa Ditt;Birthe Jessen.
Journal of Virology (2010)
Clinical Features and Outcome of Patients With IRAK-4 and MyD88 Deficiency
Capucine Picard;Capucine Picard;Horst Von Bernuth;Horst Von Bernuth;Horst Von Bernuth;Pegah Ghandil;Pegah Ghandil;Pegah Ghandil;Maya Chrabieh;Maya Chrabieh.
ORAI1 deficiency and lack of store-operated Ca2+ entry cause immunodeficiency, myopathy, and ectodermal dysplasia
Christie-Ann McCarl;Christie-Ann McCarl;Capucine Picard;Capucine Picard;Sara Khalil;Takumi Kawasaki.
The Journal of Allergy and Clinical Immunology (2009)
A critical role for neutralizing-antibody-producing B cells, CD4(+) T cells, and interferons in persistent and acute infections of mice with lymphocytic choriomeningitis virus: implications for adoptive immunotherapy of virus carriers
Oliver Planz;Stephan Ehl;Esther Furrer;Edit Horvath.
Proceedings of the National Academy of Sciences of the United States of America (1997)
Dendritic Cells Efficiently Induce Protective Antiviral Immunity
Burkhard Ludewig;Stephan Ehl;Urs Karrer;Bernhard Odermatt.
Journal of Virology (1998)
Profile was last updated on December 6th, 2021.
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