What is he best known for?
The fields of study he is best known for:
- Gene
- Immune system
- Mutation
Ulrich Salzer mostly deals with Immunology, Common variable immunodeficiency, Hypogammaglobulinemia, Mutation and B-cell activating factor.
The concepts of his Immunology study are interwoven with issues in Genetics and Candidate gene.
His research integrates issues of Selective IgA deficiency, Primary immunodeficiency, Gene and Immunodeficiency in his study of Common variable immunodeficiency.
His studies examine the connections between Hypogammaglobulinemia and genetics, as well as such issues in Genotype, with regards to Immunoglobulin D, Allele, Loss of heterozygosity and Humoral immune deficiency.
His study in Mutation is interdisciplinary in nature, drawing from both Haploinsufficiency, CD80, Inflammatory bowel disease and FOXP3.
His work carried out in the field of B-cell activating factor brings together such families of science as Receptor and Immunoglobulin class switching.
His most cited work include:
- Inflammatory bowel disease and mutations affecting the interleukin-10 receptor. (1005 citations)
- A Homozygous CARD9 Mutation in a Family with Susceptibility to Fungal Infections (572 citations)
- Mutations in TNFRSF13B encoding TACI are associated with common variable immunodeficiency in humans (563 citations)
What are the main themes of his work throughout his whole career to date?
Ulrich Salzer mainly investigates Immunology, Common variable immunodeficiency, B cell, Cell biology and Genetics.
Immunology is closely attributed to Mutation in his work.
The study incorporates disciplines such as Virology, Phenotype, Gene, Selective IgA deficiency and B-cell activating factor in addition to Common variable immunodeficiency.
His B-cell activating factor research includes elements of Receptor and Immunoglobulin class switching.
The B cell study combines topics in areas such as B-1 cell, Cancer research, CD19 and Rituximab.
Ulrich Salzer usually deals with Cell biology and limits it to topics linked to Cytoskeleton and Membrane.
He most often published in these fields:
- Immunology (63.95%)
- Common variable immunodeficiency (31.97%)
- B cell (21.09%)
What were the highlights of his more recent work (between 2016-2021)?
- Immunology (63.95%)
- Internal medicine (12.24%)
- Cell biology (16.33%)
In recent papers he was focusing on the following fields of study:
Ulrich Salzer focuses on Immunology, Internal medicine, Cell biology, Antibody and Immune system.
His study involves Common variable immunodeficiency and Hypogammaglobulinemia, a branch of Immunology.
His Internal medicine research incorporates elements of Gastroenterology and Magnetic resonance imaging.
His Cell biology research is multidisciplinary, relying on both Cell, Cytoskeleton and Membrane, Membrane protein.
His study in the field of CD20, CD19 and Plasma cell is also linked to topics like Intrathecal.
His Immune system research integrates issues from Inflammation, Genetic variants and Bioinformatics.
Between 2016 and 2021, his most popular works were:
- Phenotype, penetrance, and treatment of 133 cytotoxic T-lymphocyte antigen 4–insufficient subjects (132 citations)
- MicroRNA-146a reduces MHC-II expression via targeting JAK/STAT-signaling in dendritic cells after stem cell transplantation (43 citations)
- Rituximab as Induction Therapy in Eosinophilic Granulomatosis with Polyangiitis Refractory to Conventional Immunosuppressive Treatment: A 36-Month Follow-Up Analysis. (38 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Immune system
- Mutation
His primary scientific interests are in Immunology, Internal medicine, Cell biology, Cell culture and CTLA-4.
His research in Immunology intersects with topics in Stem cell and Transplantation.
His work on Behcet's disease as part of general Internal medicine study is frequently linked to Varicella zoster virus, therefore connecting diverse disciplines of science.
His studies in Cell biology integrate themes in fields like Integral membrane protein, Membrane protein and BAFF receptor, B-cell activating factor.
His CTLA-4 research is multidisciplinary, incorporating perspectives in Autoimmunity, CD86, CD80 and Antigen-presenting cell.
His Hypogammaglobulinemia study incorporates themes from Germline mutation, Immunosuppression, Common variable immunodeficiency, Penetrance and Primary immunodeficiency.
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