Immunology, Cell biology, Interleukin 10, Inflammatory bowel disease and Immune system are his primary areas of study. Scott B. Snapper combines subjects such as Mutation, Cellular differentiation and Candidate gene with his study of Immunology. His Cell biology study combines topics in areas such as MDia1, Actin cytoskeleton, Arp2/3 complex and Lamellipodium.
His research in Interleukin 10 intersects with topics in Inflammasome, Adoptive cell transfer and Intestinal mucosa. His Inflammatory bowel disease study incorporates themes from Crohn's disease, Mucosal immunology and Ulcerative colitis. His work on Cellular immunity as part of general Immune system study is frequently connected to Mycobacterium bovis, therefore bridging the gap between diverse disciplines of science and establishing a new relationship between them.
His primary areas of study are Immunology, Inflammatory bowel disease, Cell biology, Immune system and Colitis. His Immunology study frequently draws connections between adjacent fields such as Wiskott–Aldrich syndrome protein. The concepts of his Wiskott–Aldrich syndrome protein study are interwoven with issues in Molecular biology and Wiskott–Aldrich syndrome.
The Inflammatory bowel disease study combines topics in areas such as Age of onset, Primary immunodeficiency and Ulcerative colitis. His Cell biology study frequently involves adjacent topics like Actin cytoskeleton. His research integrates issues of Mutation, Receptor and Signal transduction in his study of Interleukin 10.
His primary areas of investigation include Inflammatory bowel disease, Immunology, Immune system, Internal medicine and Ulcerative colitis. His study in Inflammatory bowel disease is interdisciplinary in nature, drawing from both Exome sequencing, Immunodeficiency, Age of onset, Primary immunodeficiency and Pediatrics. His Immunology research includes elements of Very early onset and Peripheral blood mononuclear cell.
As part of the same scientific family, Scott B. Snapper usually focuses on Immune system, concentrating on Gene and intersecting with Humanized mouse. His Internal medicine study integrates concerns from other disciplines, such as Gastroenterology and MEDLINE. His Ulcerative colitis research includes themes of Crohn's disease, Colonoscopy and Cohort.
Scott B. Snapper mainly investigates Inflammatory bowel disease, Internal medicine, Age of onset, Immunology and Primary immunodeficiency. His Inflammatory bowel disease study combines topics from a wide range of disciplines, such as Immune system, Pediatrics and Etiology. His Immune system research is multidisciplinary, relying on both Peripheral blood mononuclear cell and CD38.
Scott B. Snapper has researched Internal medicine in several fields, including Position paper and MEDLINE. His Age of onset research incorporates elements of Enteropathy, CYBB, Case-control study and Allele. Scott B. Snapper works in the field of Immunology, focusing on Dendritic cell in particular.
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Dysfunction of the intestinal microbiome in inflammatory bowel disease and treatment
Xochitl C Morgan;Timothy L Tickle;Timothy L Tickle;Harry Sokol;Harry Sokol;Dirk Gevers.
Genome Biology (2012)
SARS-CoV-2 Receptor ACE2 Is an Interferon-Stimulated Gene in Human Airway Epithelial Cells and Is Detected in Specific Cell Subsets across Tissues.
Carly G.K. Ziegler;Samuel J. Allon;Sarah K. Nyquist;Ian M. Mbano.
Cell (2020)
New use of BCG for recombinant vaccines.
C. K. Stover;V. F. De La Cruz;T. R. Fuerst;J. E. Burlein.
Nature (1991)
Inflammatory bowel disease and mutations affecting the interleukin-10 receptor.
Erik-Oliver Glocker;Daniel Kotlarz;Kaan Boztug;E. Michael Gertz.
The New England Journal of Medicine (2009)
Isolation and characterization of efficient plasmid transformation mutants of Mycobacterium smegmatis.
S. B. Snapper;R. E. Melton;S. Mustafa;T. Kieser.
Molecular Microbiology (1990)
Impaired B Cell Development and Proliferation in Absence of Phosphoinositide 3-Kinase p85α
David A. Fruman;Scott B. Snapper;Claudine M. Yballe;Laurie Davidson.
Science (1999)
Anti-inflammatory effect of IL-10 mediated by metabolic reprogramming of macrophages.
W. K. Eddie Ip;Namiko Hoshi;Dror S. Shouval;Scott Snapper.
Science (2017)
Individual intestinal symbionts induce a distinct population of RORγ+ regulatory T cells
Esen Sefik;Naama Geva-Zatorsky;Sungwhan Oh;Liza Konnikova.
Science (2015)
The diagnostic approach to monogenic very early onset inflammatory bowel disease.
Holm H. Uhlig;Tobias Schwerd;Sibylle Koletzko;Neil Shah;Neil Shah.
Gastroenterology (2014)
Wiskott-Aldrich Syndrome Protein-Deficient Mice Reveal a Role for WASP in T but Not B Cell Activation
Scott B. Snapper;Fred S. Rosen;Emiko Mizoguchi;Paul Cohen.
Immunity (1998)
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