What is he best known for?
The fields of study he is best known for:
Immunology, B cell, Molecular biology, Bruton's tyrosine kinase and Genetics are his primary areas of study.
Mary Ellen Conley focuses mostly in the field of Immunology, narrowing it down to matters related to Gene rearrangement and, in some cases, Gene mutation, Severe combined immunodeficiency and B-1 cell.
His Molecular biology study integrates concerns from other disciplines, such as Immunoglobulin light chain, Antibody and Cellular differentiation.
His study in Bruton's tyrosine kinase focuses on X-linked agammaglobulinemia in particular.
He usually deals with X-linked agammaglobulinemia and limits it to topics linked to Cell culture and SH3 domain, Tyrosine, Kinase and Kinase activity.
A large part of his Genetics studies is devoted to Mutation.
His most cited work include:
- Deficient expression of a B cell cytoplasmic tyrosine kinase in human X-linked agammaglobulinemia (1112 citations)
- Deficient expression of a B cell cytoplasmic tyrosine kinase in human X-linked agammaglobulinemia (1112 citations)
- Diagnostic Criteria for Primary Immunodeficiencies (845 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of investigation include Immunology, Genetics, Bruton's tyrosine kinase, Molecular biology and X-linked agammaglobulinemia.
Mary Ellen Conley has included themes like Severe combined immunodeficiency and Family history in his Immunology study.
His Bruton's tyrosine kinase research integrates issues from Genotype, Kinase, B cell and Intron.
His work deals with themes such as Cell and Cellular differentiation, which intersect with B cell.
In his research, Transcription factor is intimately related to Transcription, which falls under the overarching field of Molecular biology.
His X-linked agammaglobulinemia study combines topics in areas such as Cell culture, Neutropenia, Pediatrics and Lymphocyte.
He most often published in these fields:
- Immunology (64.25%)
- Genetics (43.02%)
- Bruton's tyrosine kinase (39.11%)
What were the highlights of his more recent work (between 2008-2016)?
- Immunology (64.25%)
- Primary immunodeficiency (11.73%)
- B cell (25.14%)
In recent papers he was focusing on the following fields of study:
Mary Ellen Conley mainly investigates Immunology, Primary immunodeficiency, B cell, CD19 and B-cell receptor.
The concepts of his Immunology study are interwoven with issues in X-linked agammaglobulinemia, Retrospective cohort study and MEDLINE.
His study in Mutation extends to B cell with its themes.
His CD19 research is included under the broader classification of Molecular biology.
His work in Molecular biology tackles topics such as Antibody which are related to areas like Ex vivo.
His studies in B-cell receptor integrate themes in fields like Cell, Cancer research and Bruton's tyrosine kinase.
Between 2008 and 2016, his most popular works were:
- Primary Immunodeficiency Diseases: An Update on the Classification from the International Union of Immunological Societies Expert Committee for Primary Immunodeficiency (598 citations)
- Primary Immunodeficiency Diseases: An Update on the Classification from the International Union of Immunological Societies Expert Committee for Primary Immunodeficiency (598 citations)
- Primary immunodeficiencies: 2009 update (375 citations)
In his most recent research, the most cited papers focused on:
His scientific interests lie mostly in Primary immunodeficiency, Expert committee, Immunology, B cell and Family medicine.
His Primary immunodeficiency research is mostly focused on the topic Combined immunodeficiencies.
The Immunology study combines topics in areas such as Hyper IgM syndrome and Intensive care medicine.
He interconnects Mast cell, Molecular biology, Mutation and CD19 in the investigation of issues within B cell.
His Molecular biology research incorporates elements of Missense mutation and Frameshift mutation.
His Family medicine study incorporates themes from Alternative medicine, Disease, Bioinformatics and Immunodeficiency.
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