D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 66 Citations 17,197 219 World Ranking 5506 National Ranking 46

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Apoptosis
  • Cancer

Apoptosis, Cell biology, Programmed cell death, Cancer research and Puma are his primary areas of study. His Apoptosis research integrates issues from Cell and Cytokine. His Cell biology study combines topics from a wide range of disciplines, such as Carcinogenesis, Molecular biology and Suppressor.

The Programmed cell death study combines topics in areas such as Tumor suppressor gene, Neurodegeneration, DNA damage and Neuroscience. His research integrates issues of Fas ligand, Immunology, BH3 Mimetic ABT-737 and Fas receptor in his study of Cancer research. His Puma study integrates concerns from other disciplines, such as Cellular differentiation and Bcl-2 family.

His most cited work include:

  • Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018. (1421 citations)
  • p53- and drug-induced apoptotic responses mediated by BH3-only proteins puma and noxa. (1100 citations)
  • p53- and drug-induced apoptotic responses mediated by BH3-only proteins puma and noxa. (1100 citations)

What are the main themes of his work throughout his whole career to date?

His scientific interests lie mostly in Cell biology, Apoptosis, Programmed cell death, Cancer research and Immunology. His work carried out in the field of Cell biology brings together such families of science as Cell cycle, DNA damage and Puma. Andreas Villunger combines subjects such as CHEK1, Cell cycle checkpoint, Death domain and DNA repair with his study of DNA damage.

His Apoptosis research is multidisciplinary, incorporating perspectives in Cell culture, Immune system and Molecular biology. Andreas Villunger works in the field of Programmed cell death, namely Bcl-2 family. The study incorporates disciplines such as Cytokine, Carcinogenesis, T cell, Downregulation and upregulation and B cell in addition to Cancer research.

He most often published in these fields:

  • Cell biology (57.76%)
  • Apoptosis (49.14%)
  • Programmed cell death (42.24%)

What were the highlights of his more recent work (between 2018-2021)?

  • Cell biology (57.76%)
  • Cancer research (40.52%)
  • Programmed cell death (42.24%)

In recent papers he was focusing on the following fields of study:

His primary scientific interests are in Cell biology, Cancer research, Programmed cell death, DNA damage and Centrosome. His Cell biology study incorporates themes from Apoptosis, Cell cycle and Multiprotein complex. Andreas Villunger regularly ties together related areas like Mutant in his Apoptosis studies.

His work deals with themes such as T cell, Entosis, Downregulation and upregulation and B cell, which intersect with Cancer research. He mostly deals with Caspase in his studies of Programmed cell death. His work focuses on many connections between DNA damage and other disciplines, such as CHEK1, that overlap with his field of interest in Essential gene and Haematopoiesis.

Between 2018 and 2021, his most popular works were:

  • RIPK1 and Caspase-8 Ensure Chromosome Stability Independently of Their Role in Cell Death and Inflammation (21 citations)
  • Glucocorticoid Receptor-Deficient Foxp3 + Regulatory T Cells Fail to Control Experimental Inflammatory Bowel Disease (15 citations)
  • Glucocorticoid Receptor-Deficient Foxp3 + Regulatory T Cells Fail to Control Experimental Inflammatory Bowel Disease (15 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Apoptosis
  • Cancer

The scientist’s investigation covers issues in Cell biology, Cytokinesis, Centrosome, Downregulation and upregulation and Cancer research. His Cell biology research incorporates themes from DNA methylation, Immunoglobulin class switching, Reprogramming, Somatic hypermutation and Germinal center. Andreas Villunger has researched Cytokinesis in several fields, including E2F Transcription Factors, Cell cycle, E2F, E2F1 and Cell fate determination.

His Centrosome research incorporates elements of Multiprotein complex, DNA damage, Neoplastic transformation, Regeneration and Ploidy. His Downregulation and upregulation study combines topics in areas such as Inflammatory bowel disease, T cell, Immune system, Regulatory T cell and Glucocorticoid receptor. His studies deal with areas such as Crosstalk, Venetoclax, Regulation of gene expression, FOXP3 and Glucocorticoid as well as Cancer research.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

p53- and drug-induced apoptotic responses mediated by BH3-only proteins puma and noxa.

Andreas Villunger;Andreas Villunger;Ewa M. Michalak;Leigh Coultas;Franziska Müllauer.
Science (2003)

1544 Citations

Essential versus accessory aspects of cell death: recommendations of the NCCD 2015

L. Galluzzi;J. M. Bravo-San Pedro;I. Vitale;S. A. Aaronson.
Cell Death & Differentiation (2015)

942 Citations

LDHA-Associated Lactic Acid Production Blunts Tumor Immunosurveillance by T and NK Cells.

Almut Brand;Katrin Singer;Gudrun E. Koehl;Marlene Kolitzus.
Cell Metabolism (2016)

859 Citations

Bmf: a proapoptotic BH3-only protein regulated by interaction with the myosin V actin motor complex, activated by anoikis.

Hamsa Puthalakath;Andreas Villunger;Lorraine A. O'Reilly;Jennifer G. Beaumont.
Science (2001)

785 Citations

Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018

Lorenzo Galluzzi;Ilio Vitale;Stuart A. Aaronson;John M. Abrams.
Nature (2018)

784 Citations

FOXO3a-dependent regulation of Puma in response to cytokine/growth factor withdrawal

Han You;Marc Pellegrini;Marc Pellegrini;Katsuya Tsuchihara;Katsuya Tsuchihara;Kazuo Yamamoto;Kazuo Yamamoto.
Journal of Experimental Medicine (2006)

489 Citations

Activation of Fas by FasL induces apoptosis by a mechanism that cannot be blocked by Bcl-2 or Bcl-xL

David C. S. Huang;Michael Hahne;Michael Schroeter;Karl Frei.
Proceedings of the National Academy of Sciences of the United States of America (1999)

373 Citations

Bim and Bad mediate imatinib-induced killing of Bcr/Abl+ leukemic cells, and resistance due to their loss is overcome by a BH3 mimetic

Junya Kuroda;Hamsa Puthalakath;Mark S. Cragg;Priscilla N. Kelly;Priscilla N. Kelly.
Proceedings of the National Academy of Sciences of the United States of America (2006)

372 Citations

Bcl2 family proteins in carcinogenesis and the treatment of cancer.

Anna Frenzel;Francesca Grespi;Waldemar Chmelewskij;Andreas Villunger.
Apoptosis (2009)

342 Citations

BH3-only proteins Puma and Bim are rate-limiting for γ-radiation– and glucocorticoid-induced apoptosis of lymphoid cells in vivo

Miriam Erlacher;Miriam Erlacher;Ewa M. Michalak;Ewa M. Michalak;Priscilla N. Kelly;Priscilla N. Kelly;Verena Labi;Verena Labi.
Blood (2005)

320 Citations

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