Apoptosis, Cell biology, Programmed cell death, Cancer research and Puma are his primary areas of study. His Apoptosis research integrates issues from Cell and Cytokine. His Cell biology study combines topics from a wide range of disciplines, such as Carcinogenesis, Molecular biology and Suppressor.
The Programmed cell death study combines topics in areas such as Tumor suppressor gene, Neurodegeneration, DNA damage and Neuroscience. His research integrates issues of Fas ligand, Immunology, BH3 Mimetic ABT-737 and Fas receptor in his study of Cancer research. His Puma study integrates concerns from other disciplines, such as Cellular differentiation and Bcl-2 family.
His scientific interests lie mostly in Cell biology, Apoptosis, Programmed cell death, Cancer research and Immunology. His work carried out in the field of Cell biology brings together such families of science as Cell cycle, DNA damage and Puma. Andreas Villunger combines subjects such as CHEK1, Cell cycle checkpoint, Death domain and DNA repair with his study of DNA damage.
His Apoptosis research is multidisciplinary, incorporating perspectives in Cell culture, Immune system and Molecular biology. Andreas Villunger works in the field of Programmed cell death, namely Bcl-2 family. The study incorporates disciplines such as Cytokine, Carcinogenesis, T cell, Downregulation and upregulation and B cell in addition to Cancer research.
His primary scientific interests are in Cell biology, Cancer research, Programmed cell death, DNA damage and Centrosome. His Cell biology study incorporates themes from Apoptosis, Cell cycle and Multiprotein complex. Andreas Villunger regularly ties together related areas like Mutant in his Apoptosis studies.
His work deals with themes such as T cell, Entosis, Downregulation and upregulation and B cell, which intersect with Cancer research. He mostly deals with Caspase in his studies of Programmed cell death. His work focuses on many connections between DNA damage and other disciplines, such as CHEK1, that overlap with his field of interest in Essential gene and Haematopoiesis.
The scientist’s investigation covers issues in Cell biology, Cytokinesis, Centrosome, Downregulation and upregulation and Cancer research. His Cell biology research incorporates themes from DNA methylation, Immunoglobulin class switching, Reprogramming, Somatic hypermutation and Germinal center. Andreas Villunger has researched Cytokinesis in several fields, including E2F Transcription Factors, Cell cycle, E2F, E2F1 and Cell fate determination.
His Centrosome research incorporates elements of Multiprotein complex, DNA damage, Neoplastic transformation, Regeneration and Ploidy. His Downregulation and upregulation study combines topics in areas such as Inflammatory bowel disease, T cell, Immune system, Regulatory T cell and Glucocorticoid receptor. His studies deal with areas such as Crosstalk, Venetoclax, Regulation of gene expression, FOXP3 and Glucocorticoid as well as Cancer research.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
p53- and drug-induced apoptotic responses mediated by BH3-only proteins puma and noxa.
Andreas Villunger;Andreas Villunger;Ewa M. Michalak;Leigh Coultas;Franziska Müllauer.
Science (2003)
Essential versus accessory aspects of cell death: recommendations of the NCCD 2015
L. Galluzzi;J. M. Bravo-San Pedro;I. Vitale;S. A. Aaronson.
Cell Death & Differentiation (2015)
LDHA-Associated Lactic Acid Production Blunts Tumor Immunosurveillance by T and NK Cells.
Almut Brand;Katrin Singer;Gudrun E. Koehl;Marlene Kolitzus.
Cell Metabolism (2016)
Bmf: a proapoptotic BH3-only protein regulated by interaction with the myosin V actin motor complex, activated by anoikis.
Hamsa Puthalakath;Andreas Villunger;Lorraine A. O'Reilly;Jennifer G. Beaumont.
Science (2001)
Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018
Lorenzo Galluzzi;Ilio Vitale;Stuart A. Aaronson;John M. Abrams.
Nature (2018)
FOXO3a-dependent regulation of Puma in response to cytokine/growth factor withdrawal
Han You;Marc Pellegrini;Marc Pellegrini;Katsuya Tsuchihara;Katsuya Tsuchihara;Kazuo Yamamoto;Kazuo Yamamoto.
Journal of Experimental Medicine (2006)
Activation of Fas by FasL induces apoptosis by a mechanism that cannot be blocked by Bcl-2 or Bcl-xL
David C. S. Huang;Michael Hahne;Michael Schroeter;Karl Frei.
Proceedings of the National Academy of Sciences of the United States of America (1999)
Bim and Bad mediate imatinib-induced killing of Bcr/Abl+ leukemic cells, and resistance due to their loss is overcome by a BH3 mimetic
Junya Kuroda;Hamsa Puthalakath;Mark S. Cragg;Priscilla N. Kelly;Priscilla N. Kelly.
Proceedings of the National Academy of Sciences of the United States of America (2006)
Bcl2 family proteins in carcinogenesis and the treatment of cancer.
Anna Frenzel;Francesca Grespi;Waldemar Chmelewskij;Andreas Villunger.
Apoptosis (2009)
BH3-only proteins Puma and Bim are rate-limiting for γ-radiation– and glucocorticoid-induced apoptosis of lymphoid cells in vivo
Miriam Erlacher;Miriam Erlacher;Ewa M. Michalak;Ewa M. Michalak;Priscilla N. Kelly;Priscilla N. Kelly;Verena Labi;Verena Labi.
Blood (2005)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below:
Walter and Eliza Hall Institute of Medical Research
University of Freiburg
Walter and Eliza Hall Institute of Medical Research
Walter and Eliza Hall Institute of Medical Research
Walter and Eliza Hall Institute of Medical Research
Innsbruck Medical University
Paracelsus Medical University
University of Freiburg
Walter and Eliza Hall Institute of Medical Research
Walter and Eliza Hall Institute of Medical Research
State University of Campinas
City College of New York
Lumentum Japan, Inc.
Aalborg University
Arizona State University
Technical University of Berlin
Leibniz Institute for Baltic Sea Research
University of Reading
University of Wisconsin–Madison
University of Naples Federico II
University of Glasgow
Tohoku University
Utrecht University
University at Buffalo, State University of New York
University of Copenhagen
Hong Kong Shue Yan University