World's Best Scientists 2026 revealed!

D-Index & Metrics

Biology and Biochemistry

D-Index
60
Citations
14718
World Ranking
11845
National Ranking
325

Marco J. Herold publication distribution in Biology and Biochemistry in 2026

The chart shows the distribution of publications by all Research.com ranked scientists in the field of Biology and Biochemistry in 2026. The highlighted bar marks where Marco J. Herold sits on this spectrum.

47–56 publications: 8 scientists 57–66 publications: 35 scientists 67–76 publications: 106 scientists 77–86 publications: 231 scientists 87–96 publications: 413 scientists 97–106 publications: 546 scientists 107–116 publications: 704 scientists 117–126 publications: 848 scientists 127–136 publications: 980 scientists 137–146 publications: 942 scientists 147–156 publications: 969 scientists 157–166 publications: 949 scientists 167–176 publications: 951 scientists 177–186 publications: 915 scientists 187–196 publications: 787 scientists 197–206 publications: 840 scientists 207–216 publications: 733 scientists 217–226 publications: 708 scientists 227–236 publications: 651 scientists 237–246 publications: 605 scientists 247–256 publications: 510 scientists 257–266 publications: 524 scientists 267–276 publications: 434 scientists 277–286 publications: 417 scientists 287–296 publications: 350 scientists 297–306 publications: 363 scientists 307–316 publications: 315 scientists 317–326 publications: 296 scientists 327–336 publications: 261 scientists 337–346 publications: 240 scientists 347–356 publications: 219 scientists 357–366 publications: 196 scientists 367–376 publications: 154 scientists 377–386 publications: 161 scientists 387–396 publications: 155 scientists 397–406 publications: 145 scientists 407–416 publications: 124 scientists 417–426 publications: 112 scientists 427–436 publications: 132 scientists 437–446 publications: 116 scientists 447–456 publications: 99 scientists 457–466 publications: 81 scientists 467–476 publications: 91 scientists 477–486 publications: 80 scientists 487–496 publications: 80 scientists 497–506 publications: 59 scientists 507–516 publications: 36 scientists 517–526 publications: 46 scientists 527–536 publications: 54 scientists 537–546 publications: 44 scientists 547–556 publications: 43 scientists 557–566 publications: 43 scientists 567–576 publications: 42 scientists 577–586 publications: 25 scientists 587–596 publications: 34 scientists 597–606 publications: 23 scientists 607–616 publications: 33 scientists 617–626 publications: 31 scientists 627–636 publications: 27 scientists 637–646 publications: 25 scientists 647–656 publications: 28 scientists 657–666 publications: 34 scientists 667–676 publications: 18 scientists 677–686 publications: 16 scientists 687–696 publications: 10 scientists 697–706 publications: 12 scientists 707–716 publications: 21 scientists 717–726 publications: 12 scientists 727–736 publications: 12 scientists 737–746 publications: 10 scientists 747–756 publications: 7 scientists 757–766 publications: 13 scientists 767–776 publications: 15 scientists 777–786 publications: 13 scientists 787–796 publications: 9 scientists 797–806 publications: 9 scientists 807–816 publications: 7 scientists 817–826 publications: 4 scientists 827–836 publications: 9 scientists 837–846 publications: 7 scientists 847–856 publications: 3 scientists 857–866 publications: 5 scientists 867–876 publications: 5 scientists 877–886 publications: 11 scientists 887–896 publications: 3 scientists 897–906 publications: 4 scientists 907–916 publications: 7 scientists 917–926 publications: 5 scientists 927–936 publications: 6 scientists 937–946 publications: 6 scientists 947–956 publications: 3 scientists 957–966 publications: 7 scientists 967–976 publications: 2 scientists 977–986 publications: 2 scientists 987–996 publications: 1 scientists 997–1,006 publications: 5 scientists 1,007–1,016 publications: 2 scientists 1,017–1,026 publications: 2 scientists 1,027 publications: 1 scientists 1,028+ publications: 100 scientists
47 publications 1,028+

This scientist: 396 publications — 89th percentile

89% of scientists in this discipline score the same or lower.

The last bar groups every scientist with 1,028 publications or more.

Marco J. Herold D-index placement in Biology and Biochemistry in 2026

The chart shows the D-index (discipline H-index) distribution of Biology and Biochemistry scientists ranked by Research.com in 2026. The highlighted bar marks where Marco J. Herold sits on this spectrum.

40–41 D-Index: 80 scientists 42–43 D-Index: 183 scientists 44–45 D-Index: 316 scientists 46–47 D-Index: 504 scientists 48–49 D-Index: 718 scientists 50–51 D-Index: 899 scientists 52–53 D-Index: 1,025 scientists 54–55 D-Index: 1,149 scientists 56–57 D-Index: 1,235 scientists 58–59 D-Index: 1,253 scientists 60–61 D-Index: 1,162 scientists 62–63 D-Index: 1,130 scientists 64–65 D-Index: 1,031 scientists 66–67 D-Index: 897 scientists 68–69 D-Index: 814 scientists 70–71 D-Index: 714 scientists 72–73 D-Index: 709 scientists 74–75 D-Index: 596 scientists 76–77 D-Index: 512 scientists 78–79 D-Index: 473 scientists 80–81 D-Index: 412 scientists 82–83 D-Index: 373 scientists 84–85 D-Index: 358 scientists 86–87 D-Index: 285 scientists 88–89 D-Index: 273 scientists 90–91 D-Index: 227 scientists 92–93 D-Index: 208 scientists 94–95 D-Index: 193 scientists 96–97 D-Index: 153 scientists 98–99 D-Index: 157 scientists 100–101 D-Index: 148 scientists 102–103 D-Index: 120 scientists 104–105 D-Index: 113 scientists 106–107 D-Index: 100 scientists 108–109 D-Index: 86 scientists 110–111 D-Index: 67 scientists 112–113 D-Index: 71 scientists 114–115 D-Index: 73 scientists 116–117 D-Index: 64 scientists 118–119 D-Index: 53 scientists 120–121 D-Index: 60 scientists 122–123 D-Index: 54 scientists 124–125 D-Index: 43 scientists 126–127 D-Index: 38 scientists 128–129 D-Index: 49 scientists 130–131 D-Index: 26 scientists 132–133 D-Index: 18 scientists 134–135 D-Index: 23 scientists 136–137 D-Index: 32 scientists 138–139 D-Index: 32 scientists 140–141 D-Index: 27 scientists 142–143 D-Index: 19 scientists 144–145 D-Index: 22 scientists 146–147 D-Index: 12 scientists 148–149 D-Index: 16 scientists 150–151 D-Index: 14 scientists 152–153 D-Index: 10 scientists 154–155 D-Index: 13 scientists 156–157 D-Index: 10 scientists 158–159 D-Index: 7 scientists 160–161 D-Index: 9 scientists 162–163 D-Index: 13 scientists 164–165 D-Index: 4 scientists 166 D-Index: 4 scientists 167+ D-Index: 98 scientists
40 D-Index 167+

This scientist: 60 D-Index — 41st percentile

41% of scientists in this discipline score the same or lower.

The last bar groups every scientist with 167 D-Index or more.

Overview

Marco J. Herold is affiliated with the Walter and Eliza Hall Institute of Medical Research in Australia. Their research contributions span fields including Biochemistry, Genetics and Molecular Biology, Medicine, and Immunology and Microbiology. Within these broader fields, their work focuses on subfields such as Molecular Biology, Immunology, Oncology, Genetics, and Hematology.

Their research topics cover a diverse array of key areas:

  • CRISPR and Genetic Engineering
  • Immune Cell Function and Interaction
  • Cell death mechanisms and regulation
  • Inflammasome and immune disorders
  • T-cell and B-cell Immunology
  • Interferon and immune responses
  • Cancer-related Molecular Pathways

Selected recent publications by Marco J. Herold include:

  • "Emerging connectivity of programmed cell death pathways and its physiological implications," 2020, Nature Reviews Molecular Cell Biology
  • "The NK cell granule protein NKG7 regulates cytotoxic granule exocytosis and inflammation," 2020, Nature Immunology
  • "Flexible Usage and Interconnectivity of Diverse Cell Death Pathways Protect against Intracellular Infection," 2020, Immunity
  • "PRMT1-mediated H4R3me2a recruits SMARCA4 to promote colorectal cancer progression by enhancing EGFR signaling," 2021, Genome Medicine
  • "Interferon-γ primes macrophages for pathogen ligand-induced killing via a caspase-8 and mitochondrial cell death pathway," 2022, Immunity

Marco J. Herold has collaborated frequently with several scientists, with the most notable coauthors including:

  • Andrew J. Kueh
  • Andreas Strasser
  • Lin Tai
  • Gemma L. Kelly
  • Sarah T. Diepstraten

Their publications have appeared in various scientific venues, predominantly in:

  • bioRxiv (Cold Spring Harbor Laboratory)
  • Nature Communications
  • Cell Death and Differentiation
  • Cell Death and Disease
  • Nature Immunology

Best Publications

  • The MCL1 inhibitor S63845 is tolerable and effective in diverse cancer models

    András Kotschy;Zoltán Szlavik;James Murray;James Davidson

  • How does p53 induce apoptosis and how does this relate to p53-mediated tumour suppression?

    Brandon J Aubrey;Gemma L Kelly;Gemma L Kelly;Ana Janic;Ana Janic;Marco J Herold;Marco J Herold

  • Apoptotic Caspases Suppress mtDNA-Induced STING-Mediated Type I IFN Production

    Michael J. White;Michael J. White;Kate McArthur;Kate McArthur;Donald Metcalf;Donald Metcalf;Rachael M. Lane

  • Emerging connectivity of programmed cell death pathways and its physiological implications

    Sammy Bedoui;Marco J Herold;Marco J Herold;Andreas Strasser;Andreas Strasser

  • Glucocorticoids in T cell apoptosis and function.

    M. J. Herold;K. G. McPherson;H. M. Reichardt

  • Anti-apoptotic Mcl-1 is essential for the development and sustained growth of acute myeloid leukemia

    Stefan P. Glaser;Erinna F. Lee;Evelyn Trounson;Philippe Bouillet

  • An Inducible Lentiviral Guide RNA Platform Enables the Identification of Tumor-Essential Genes and Tumor-Promoting Mutations In Vivo

    Brandon J. Aubrey;Gemma L. Kelly;Andrew J. Kueh;Andrew J. Kueh;Margs S. Brennan;Margs S. Brennan

  • NLRP3 inflammasome activation downstream of cytoplasmic LPS recognition by both caspase-4 and caspase-5.

    Paul J. Baker;Paul J. Baker;Dave Boucher;Damien Bierschenk;Christina Tebartz

  • Mutations that prevent caspase cleavage of RIPK1 cause autoinflammatory disease

    Najoua Lalaoui;Najoua Lalaoui;Steven E. Boyden;Hirotsugu Oda;Geryl M. Wood

  • The transcription factor T-bet is essential for the development of NKp46+ innate lymphocytes via the Notch pathway

    Lucille C Rankin;Lucille C Rankin;Joanna R Groom;Joanna R Groom;Michaël Chopin;Michaël Chopin;Marco J Herold;Marco J Herold

  • Antiapoptotic Mcl-1 is critical for the survival and niche-filling capacity of Foxp3 + regulatory T cells

    Wim Pierson;Bénédicte Cauwe;Antonia Policheni;Antonia Policheni;Susan M Schlenner

  • Glucocorticoids exert opposing effects on macrophage function dependent on their concentration.

    Hee-Young Lim;Nora Müller;Marco J Herold;Jens van den Brandt

  • Inducible and reversible gene silencing by stable integration of an shRNA-encoding lentivirus in transgenic rats.

    Marco J. Herold;Jens van den Brandt;Jost Seibler;Holger M. Reichardt

  • Targeting of MCL-1 kills MYC-driven mouse and human lymphomas even when they bear mutations in p53

    Gemma L. Kelly;Stephanie Grabow;Stephanie Grabow;Stefan P. Glaser;Stefan P. Glaser;Leah Fitzsimmons

  • Synergistic action of the MCL-1 inhibitor S63845 with current therapies in preclinical models of triple-negative and HER2-amplified breast cancer.

    Delphine Merino;Delphine Merino;James R. Whittle;James R. Whittle;James R. Whittle;François Vaillant;François Vaillant;Antonin Serrano;Antonin Serrano

  • Myeloid-derived suppressor activity is mediated by monocytic lineages maintained by continuous inhibition of extrinsic and intrinsic death pathways

    Jessica M. Haverkamp;Amber M. Smith;Ricardo Weinlich;Christopher P. Dillon

  • BCL-XL and MCL-1 are the key BCL-2 family proteins in melanoma cell survival.

    Erinna F Lee;Tiffany J Harris;Sharon Tran;Marco Evangelista

  • VDAC2 enables BAX to mediate apoptosis and limit tumor development.

    Hui San Chin;Hui San Chin;Mark X. Li;Mark X. Li;Iris K. L. Tan;Robert L. Ninnis;Robert L. Ninnis

  • Hierarchy for targeting prosurvival BCL2 family proteins in multiple myeloma: pivotal role of MCL1.

    Jia Nan Gong;Jia Nan Gong;Tiffany Khong;David Segal;David Segal;Yuan Yao;Yuan Yao;Yuan Yao

  • DNA repair processes are critical mediators of p53-dependent tumor suppression.

    Ana Janic;Ana Janic;Liz J Valente;Liz J Valente;Liz J Valente;Matthew J Wakefield;Matthew J Wakefield;Leon Di Stefano

Frequent Co-Authors

Andreas Strasser
Andreas Strasser Walter and Eliza Hall Institute of Medical Research
David C. S. Huang
David C. S. Huang Walter and Eliza Hall Institute of Medical Research
Gordon K. Smyth
Gordon K. Smyth Walter and Eliza Hall Institute of Medical Research
Philippe Bouillet
Philippe Bouillet Walter and Eliza Hall Institute of Medical Research
Guillaume Lessene
Guillaume Lessene Walter and Eliza Hall Institute of Medical Research
Lorraine A. O'Reilly
Lorraine A. O'Reilly Walter and Eliza Hall Institute of Medical Research
Stephen L. Nutt
Stephen L. Nutt Walter and Eliza Hall Institute of Medical Research
Warren S. Alexander
Warren S. Alexander Walter and Eliza Hall Institute of Medical Research
Holger M. Reichardt
Holger M. Reichardt University of Göttingen
David J. Segal
David J. Segal University of California, Davis

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