2026 Marco J. Herold: Biology and Biochemistry Researcher – H-Index, Publications & Awards

Discipline name	D-Index	World Ranking	Current World Ranking	National Ranking	Current National Ranking	Publications	Citations
Biology and Biochemistry	60	11850	10914	325	313	396	14718

Overview

Marco J. Herold is affiliated with the Walter and Eliza Hall Institute of Medical Research in Australia. Their research contributions span fields including Biochemistry, Genetics and Molecular Biology, Medicine, and Immunology and Microbiology. Within these broader fields, their work focuses on subfields such as Molecular Biology, Immunology, Oncology, Genetics, and Hematology.

Their research topics cover a diverse array of key areas:

CRISPR and Genetic Engineering
Immune Cell Function and Interaction
Cell death mechanisms and regulation
Inflammasome and immune disorders
T-cell and B-cell Immunology
Interferon and immune responses
Cancer-related Molecular Pathways

Selected recent publications by Marco J. Herold include:

"Emerging connectivity of programmed cell death pathways and its physiological implications," 2020, Nature Reviews Molecular Cell Biology
"The NK cell granule protein NKG7 regulates cytotoxic granule exocytosis and inflammation," 2020, Nature Immunology
"Flexible Usage and Interconnectivity of Diverse Cell Death Pathways Protect against Intracellular Infection," 2020, Immunity
"PRMT1-mediated H4R3me2a recruits SMARCA4 to promote colorectal cancer progression by enhancing EGFR signaling," 2021, Genome Medicine
"Interferon-γ primes macrophages for pathogen ligand-induced killing via a caspase-8 and mitochondrial cell death pathway," 2022, Immunity

Marco J. Herold has collaborated frequently with several scientists, with the most notable coauthors including:

Andrew J. Kueh
Andreas Strasser
Lin Tai
Gemma L. Kelly
Sarah T. Diepstraten

Their publications have appeared in various scientific venues, predominantly in:

bioRxiv (Cold Spring Harbor Laboratory)
Nature Communications
Cell Death and Differentiation
Cell Death and Disease
Nature Immunology

Best Publications

The MCL1 inhibitor S63845 is tolerable and effective in diverse cancer models

András Kotschy;Zoltán Szlavik;James Murray;James Davidson

1004
Citations
How does p53 induce apoptosis and how does this relate to p53-mediated tumour suppression?

Brandon J Aubrey;Gemma L Kelly;Gemma L Kelly;Ana Janic;Ana Janic;Marco J Herold;Marco J Herold

980
Citations
Apoptotic Caspases Suppress mtDNA-Induced STING-Mediated Type I IFN Production

Michael J. White;Michael J. White;Kate McArthur;Kate McArthur;Donald Metcalf;Donald Metcalf;Rachael M. Lane

964
Citations
Emerging connectivity of programmed cell death pathways and its physiological implications

Sammy Bedoui;Marco J Herold;Marco J Herold;Andreas Strasser;Andreas Strasser

681
Citations
Glucocorticoids in T cell apoptosis and function.

M. J. Herold;K. G. McPherson;H. M. Reichardt

484
Citations
Anti-apoptotic Mcl-1 is essential for the development and sustained growth of acute myeloid leukemia

Stefan P. Glaser;Erinna F. Lee;Evelyn Trounson;Philippe Bouillet

447
Citations
An Inducible Lentiviral Guide RNA Platform Enables the Identification of Tumor-Essential Genes and Tumor-Promoting Mutations In Vivo

Brandon J. Aubrey;Gemma L. Kelly;Andrew J. Kueh;Andrew J. Kueh;Margs S. Brennan;Margs S. Brennan

382
Citations
NLRP3 inflammasome activation downstream of cytoplasmic LPS recognition by both caspase-4 and caspase-5.

Paul J. Baker;Paul J. Baker;Dave Boucher;Damien Bierschenk;Christina Tebartz

380
Citations
Mutations that prevent caspase cleavage of RIPK1 cause autoinflammatory disease

Najoua Lalaoui;Najoua Lalaoui;Steven E. Boyden;Hirotsugu Oda;Geryl M. Wood

309
Citations
The transcription factor T-bet is essential for the development of NKp46+ innate lymphocytes via the Notch pathway

Lucille C Rankin;Lucille C Rankin;Joanna R Groom;Joanna R Groom;Michaël Chopin;Michaël Chopin;Marco J Herold;Marco J Herold

296
Citations
Antiapoptotic Mcl-1 is critical for the survival and niche-filling capacity of Foxp3 + regulatory T cells

Wim Pierson;Bénédicte Cauwe;Antonia Policheni;Antonia Policheni;Susan M Schlenner

269
Citations
Glucocorticoids exert opposing effects on macrophage function dependent on their concentration.

Hee-Young Lim;Nora Müller;Marco J Herold;Jens van den Brandt

266
Citations
Inducible and reversible gene silencing by stable integration of an shRNA-encoding lentivirus in transgenic rats.

Marco J. Herold;Jens van den Brandt;Jost Seibler;Holger M. Reichardt

221
Citations
Targeting of MCL-1 kills MYC-driven mouse and human lymphomas even when they bear mutations in p53

Gemma L. Kelly;Stephanie Grabow;Stephanie Grabow;Stefan P. Glaser;Stefan P. Glaser;Leah Fitzsimmons

183
Citations
Synergistic action of the MCL-1 inhibitor S63845 with current therapies in preclinical models of triple-negative and HER2-amplified breast cancer.

Delphine Merino;Delphine Merino;James R. Whittle;James R. Whittle;James R. Whittle;François Vaillant;François Vaillant;Antonin Serrano;Antonin Serrano

179
Citations
Myeloid-derived suppressor activity is mediated by monocytic lineages maintained by continuous inhibition of extrinsic and intrinsic death pathways

Jessica M. Haverkamp;Amber M. Smith;Ricardo Weinlich;Christopher P. Dillon

159
Citations
BCL-XL and MCL-1 are the key BCL-2 family proteins in melanoma cell survival.

Erinna F Lee;Tiffany J Harris;Sharon Tran;Marco Evangelista

156
Citations
VDAC2 enables BAX to mediate apoptosis and limit tumor development.

Hui San Chin;Hui San Chin;Mark X. Li;Mark X. Li;Iris K. L. Tan;Robert L. Ninnis;Robert L. Ninnis

152
Citations
Hierarchy for targeting prosurvival BCL2 family proteins in multiple myeloma: pivotal role of MCL1.

Jia Nan Gong;Jia Nan Gong;Tiffany Khong;David Segal;David Segal;Yuan Yao;Yuan Yao;Yuan Yao

150
Citations
DNA repair processes are critical mediators of p53-dependent tumor suppression.

Ana Janic;Ana Janic;Liz J Valente;Liz J Valente;Liz J Valente;Matthew J Wakefield;Matthew J Wakefield;Leon Di Stefano

149
Citations