2026 Benjamin T. Kile: Biology and Biochemistry Researcher – H-Index, Publications & Awards

Discipline name	D-Index	World Ranking	Current World Ranking	National Ranking	Current National Ranking	Publications	Citations
Biology and Biochemistry	68	7866	7255	196	192	210	15133

Research.com Recognitions

2016 - Fellow of the Australian Academy of Health and Medical Science
2013 - Gottschalk Medal, Australian Academy of Science

Overview

Benjamin T. Kile is affiliated with the University of Adelaide in Australia. Their research spans several interconnected fields, primarily within biochemistry, genetics, molecular biology, medicine, immunology, and microbiology.

Their main fields of study include:

Biochemistry, Genetics and Molecular Biology
Medicine
Immunology and Microbiology

Within these broad areas, the subfields frequently addressed are:

Molecular Biology
Immunology
Infectious Diseases
Hematology
Epidemiology

Kile's research engages with a range of scientific topics, especially focusing on:

Interferon and immune responses
Viral infections and vectors
Inflammasome and immune disorders
Cell death mechanisms and regulation
Immune response and inflammation
Autophagy in disease and therapy
Acute myeloid leukemia research

Recent publications demonstrate Kile's active involvement in understanding immune mechanisms and cell biology. Notable papers include:

"TBK1 and IKKε Act Redundantly to Mediate STING-Induced NF-κB Responses in Myeloid Cells" (2020, Cell Reports)
"Mitochondrial dysfunction caused by outer membrane vesicles from Gram-negative bacteria activates intrinsic apoptosis and inflammation" (2020, Nature Microbiology)
"A missense mutation in the MLKL brace region promotes lethal neonatal inflammation and hematopoietic dysfunction" (2020, Nature Communications)
"Termination of STING responses is mediated via ESCRT-dependent degradation" (2023, The EMBO Journal)
"COVID-19 patients exhibit reduced procoagulant platelet responses" (2020, Journal of Thrombosis and Haemostasis)

These publications appear frequently in well-regarded journals, with Kile contributing multiple works to venues such as:

Nature Communications
Cell Reports
Life Science Alliance
bioRxiv (Cold Spring Harbor Laboratory)
Blood

Collaborations play a significant role in their research output. Frequent coauthors include:

Katherine R. Balka
Dominic De Nardo
Stéphane Chappaz
Tahnee L. Saunders
Kate E. Lawlor

Kile's work has been recognized with awards such as:

Fellow of the Australian Academy of Health and Medical Science (2016)
Gottschalk Medal, Australian Academy of Science (2013)

Best Publications

Programmed Anuclear Cell Death Delimits Platelet Life Span

Kylie D Mason;Marina Carpinelli;Jamie I Fletcher;Janelle Elyse Collinge

1175
Citations
Apoptotic Caspases Suppress mtDNA-Induced STING-Mediated Type I IFN Production

Michael J. White;Michael J. White;Kate McArthur;Kate McArthur;Donald Metcalf;Donald Metcalf;Rachael M. Lane

964
Citations
BAK/BAX macropores facilitate mitochondrial herniation and mtDNA efflux during apoptosis

Kate McArthur;Kate McArthur;Kate McArthur;Lachlan W. Whitehead;Lachlan W. Whitehead;John M. Heddleston;Lucy Li

773
Citations
The SOCS box: a tale of destruction and degradation

Benjamin T Kile;Brenda A Schulman;Warren S Alexander;Nicos A Nicola

556
Citations
Interleukin-11 Is the Dominant IL-6 Family Cytokine during Gastrointestinal Tumorigenesis and Can Be Targeted Therapeutically

Tracy L. Putoczki;Tracy L. Putoczki;Stefan Thiem;Andrea Loving;Rita A. Busuttil;Rita A. Busuttil

424
Citations
TBK1 and IKKε Act Redundantly to Mediate STING-Induced NF-κB Responses in Myeloid Cells.

Katherine R. Balka;Katherine R. Balka;Katherine R. Balka;Cynthia Louis;Cynthia Louis;Tahnee L. Saunders;Amber M. Smith

424
Citations
The Dendritic Cell Receptor Clec9A Binds Damaged Cells via Exposed Actin Filaments

Jian Guo Zhang;Jian Guo Zhang;Peter E. Czabotar;Peter E. Czabotar;Antonia N. Policheni;Antonia N. Policheni;Irina Caminschi;Irina Caminschi;Irina Caminschi

404
Citations
NLRP1 Inflammasome Activation Induces Pyroptosis of Hematopoietic Progenitor Cells

Seth L. Masters;Motti Gerlic;Motti Gerlic;Donald Metcalf;Donald Metcalf;Simon Preston;Simon Preston

350
Citations
Two distinct pathways regulate platelet phosphatidylserine exposure and procoagulant function

Simone Marianne Schoenwaelder;Yu-Ping Yuan;Emma C Josefsson;Michael J White

344
Citations
Bcl-xL inhibitory BH3 mimetics can induce a transient thrombocytopathy that undermines the hemostatic function of platelets

Simone M. Schoenwaelder;Kate E. Jarman;Elizabeth E. Gardiner;My Hua

342
Citations
The transcription factor Erg is essential for definitive hematopoiesis and the function of adult hematopoietic stem cells.

Stephen J Loughran;Elizabeth A Kruse;Elizabeth A Kruse;Douglas F Hacking;Douglas F Hacking;Carolyn A de Graaf;Carolyn A de Graaf

288
Citations
Mitochondrial apoptosis is dispensable for NLRP3 inflammasome activation but non-apoptotic caspase-8 is required for inflammasome priming

Ramanjaneyulu Allam;Kate E Lawlor;Kate E Lawlor;Eric Chi-Wang Yu;Alison L Mildenhall;Alison L Mildenhall

244
Citations
The suppressors of cytokine signalling (SOCS).

B T Kile;W S Alexander

230
Citations
Functional genetic analysis of mouse chromosome 11

Benjamin T. Kile;Kathryn E. Hentges;Amander T. Clark;Hisashi Nakamura

228
Citations
Megakaryocytes possess a functional intrinsic apoptosis pathway that must be restrained to survive and produce platelets

Emma C. Josefsson;Chloé James;Katya J. Henley;Marlyse A. Debrincat;Marlyse A. Debrincat

202
Citations
The Mitochondrial Apoptotic Effectors BAX/BAK Activate Caspase-3 and -7 to Trigger NLRP3 Inflammasome and Caspase-8 Driven IL-1β Activation

James E. Vince;James E. Vince;Dominic De Nardo;Dominic De Nardo;Wenqing Gao;Angelina J. Vince

186
Citations
Suppressors of cytokine signaling (SOCS): Negative regulators of signal transduction

Warren S. Alexander;Robyn Starr;Donald Metcalf;Sandra E. Nicholson

169
Citations
IL-18 Production from the NLRP1 Inflammasome Prevents Obesity and Metabolic Syndrome.

Andrew J. Murphy;Andrew J. Murphy;Michael J. Kraakman;Helene L. Kammoun;Dragana Dragoljevic;Dragana Dragoljevic

165
Citations
Apoptotic Caspases: Multiple or Mistaken Identities?

Kate McArthur;Benjamin T. Kile

161
Citations
Mitochondrial dysfunction caused by outer membrane vesicles from Gram-negative bacteria activates intrinsic apoptosis and inflammation

Pankaj Deo;Seong H Chow;Mei-Ling Han;Mary Speir

155
Citations