H-Index & Metrics Best Publications

H-Index & Metrics

Discipline name H-index Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 59 Citations 14,983 209 World Ranking 5839 National Ranking 149

Research.com Recognitions

Awards & Achievements

2016 - Fellow of the Australian Academy of Health and Medical Science

2013 - Gottschalk Medal, Australian Academy of Science

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Mutation
  • Genetics

Cell biology, Programmed cell death, Apoptosis, Platelet and Intrinsic apoptosis are his primary areas of study. He interconnects Inflammasome and Cytoskeleton in the investigation of issues within Cell biology. His study in the field of Bcl-2 Homologous Antagonist-Killer Protein is also linked to topics like Bcl-2-associated X protein.

His Bcl-2 Homologous Antagonist-Killer Protein research incorporates elements of Platelet activation and Platelet aggregation inhibitor. His research in Apoptosis intersects with topics in Wound healing and Cytoplasm. Platelet is frequently linked to Cdc42 GTP-Binding Protein in his study.

His most cited work include:

  • Programmed Anuclear Cell Death Delimits Platelet Life Span (726 citations)
  • Programmed Anuclear Cell Death Delimits Platelet Life Span (726 citations)
  • Apoptotic Caspases Suppress mtDNA-Induced STING-Mediated Type I IFN Production (384 citations)

What are the main themes of his work throughout his whole career to date?

His primary areas of investigation include Cell biology, Platelet, Apoptosis, Immunology and Cancer research. His Cell biology research includes elements of Caspase, Programmed cell death, Intrinsic apoptosis and Bcl-2 Homologous Antagonist-Killer Protein. His Platelet research is multidisciplinary, incorporating perspectives in Megakaryocyte, Thrombopoiesis and Thrombopoietin.

His Apoptosis research incorporates themes from Platelet activation, Hemostasis, Mitochondrion and In vivo. His study looks at the relationship between Immunology and fields such as Haematopoiesis, as well as how they intersect with chemical problems. His studies deal with areas such as Leukemia and Transcription factor as well as Cancer research.

He most often published in these fields:

  • Cell biology (71.32%)
  • Platelet (48.16%)
  • Apoptosis (45.59%)

What were the highlights of his more recent work (between 2017-2021)?

  • Cell biology (71.32%)
  • Apoptosis (45.59%)
  • Intrinsic apoptosis (25.37%)

In recent papers he was focusing on the following fields of study:

His primary areas of study are Cell biology, Apoptosis, Intrinsic apoptosis, Cancer research and Platelet. The various areas that Benjamin T. Kile examines in his Cell biology study include RNA, Programmed cell death and Stimulator of interferon genes. His Caspase study in the realm of Apoptosis interacts with subjects such as AMP-activated protein kinase.

His Intrinsic apoptosis study combines topics in areas such as Pyroptosis, Inflammasome and Bcl-2 Homologous Antagonist-Killer Protein. His Cancer research study integrates concerns from other disciplines, such as Phenotype, Epigenetics and Mutation. His work deals with themes such as Hemostasis, Thrombosis and Necrosis, which intersect with Platelet.

Between 2017 and 2021, his most popular works were:

  • BAK/BAX macropores facilitate mitochondrial herniation and mtDNA efflux during apoptosis (221 citations)
  • BAK/BAX macropores facilitate mitochondrial herniation and mtDNA efflux during apoptosis (221 citations)
  • BAK/BAX macropores facilitate mitochondrial herniation and mtDNA efflux during apoptosis (221 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Mutation
  • Genetics

His main research concerns Cell biology, Programmed cell death, Bcl-2-associated X protein, Intrinsic apoptosis and Bcl-2 Homologous Antagonist-Killer Protein. Cell biology is often connected to Apoptosis in his work. His studies in Apoptosis integrate themes in fields like Damage-associated molecular pattern, Lineage, Cellular differentiation and Mitochondrion.

The concepts of his Mitochondrion study are interwoven with issues in Type I interferon production, Function, Mitochondrial DNA, Platelet and Pathophysiology. Benjamin T. Kile has included themes like Phenotype, Missense mutation, Compound heterozygosity, Mutation and Heterozygote advantage in his Programmed cell death study. The various areas that Benjamin T. Kile examines in his Signal transduction study include TANK-binding kinase 1, Interferon, IRF3 and Stimulator of interferon genes.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Programmed Anuclear Cell Death Delimits Platelet Life Span

Kylie D Mason;Kylie D Mason;Marina Carpinelli;Jamie I Fletcher;Janelle Elyse Collinge.
Cell (2007)

954 Citations

The SOCS box: a tale of destruction and degradation

Benjamin T Kile;Brenda A Schulman;Warren S Alexander;Nicos A Nicola.
Trends in Biochemical Sciences (2002)

505 Citations

Apoptotic Caspases Suppress mtDNA-Induced STING-Mediated Type I IFN Production

Michael J. White;Michael J. White;Kate McArthur;Kate McArthur;Donald Metcalf;Donald Metcalf;Rachael M. Lane.
Cell (2014)

453 Citations

Interleukin-11 Is the Dominant IL-6 Family Cytokine during Gastrointestinal Tumorigenesis and Can Be Targeted Therapeutically

Tracy L. Putoczki;Tracy L. Putoczki;Stefan Thiem;Andrea Loving;Rita A. Busuttil;Rita A. Busuttil.
Cancer Cell (2013)

317 Citations

Two distinct pathways regulate platelet phosphatidylserine exposure and procoagulant function

Simone Marianne Schoenwaelder;Yu-Ping Yuan;Emma C Josefsson;Michael J White.
Blood (2009)

295 Citations

The Dendritic Cell Receptor Clec9A Binds Damaged Cells via Exposed Actin Filaments

Jian Guo Zhang;Jian Guo Zhang;Peter E. Czabotar;Peter E. Czabotar;Antonia N. Policheni;Antonia N. Policheni;Irina Caminschi;Irina Caminschi;Irina Caminschi.
Immunity (2012)

295 Citations

The transcription factor Erg is essential for definitive hematopoiesis and the function of adult hematopoietic stem cells.

Stephen J Loughran;Elizabeth A Kruse;Elizabeth A Kruse;Douglas F Hacking;Douglas F Hacking;Carolyn A de Graaf;Carolyn A de Graaf.
Nature Immunology (2008)

248 Citations

BAK/BAX macropores facilitate mitochondrial herniation and mtDNA efflux during apoptosis

Kate McArthur;Kate McArthur;Kate McArthur;Lachlan W. Whitehead;Lachlan W. Whitehead;John M. Heddleston;Lucy Li.
Science (2018)

244 Citations

NLRP1 Inflammasome Activation Induces Pyroptosis of Hematopoietic Progenitor Cells

Seth L. Masters;Motti Gerlic;Motti Gerlic;Donald Metcalf;Donald Metcalf;Simon Preston;Simon Preston.
Immunity (2012)

239 Citations

Bcl-xL inhibitory BH3 mimetics can induce a transient thrombocytopathy that undermines the hemostatic function of platelets

Simone M. Schoenwaelder;Kate E. Jarman;Elizabeth E. Gardiner;My Hua.
Blood (2011)

223 Citations

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