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Benjamin T. Kile

D-Index & Metrics

Biology and Biochemistry

D-Index
68
Citations
15133
World Ranking
7866
National Ranking
196

Benjamin T. Kile publication distribution in Biology and Biochemistry in 2026

The chart shows the distribution of publications by all Research.com ranked scientists in the field of Biology and Biochemistry in 2026. The highlighted bar marks where Benjamin T. Kile sits on this spectrum.

47–56 publications: 8 scientists 57–66 publications: 35 scientists 67–76 publications: 106 scientists 77–86 publications: 231 scientists 87–96 publications: 414 scientists 97–106 publications: 546 scientists 107–116 publications: 704 scientists 117–126 publications: 849 scientists 127–136 publications: 980 scientists 137–146 publications: 942 scientists 147–156 publications: 969 scientists 157–166 publications: 950 scientists 167–176 publications: 951 scientists 177–186 publications: 915 scientists 187–196 publications: 787 scientists 197–206 publications: 841 scientists 207–216 publications: 735 scientists 217–226 publications: 709 scientists 227–236 publications: 651 scientists 237–246 publications: 605 scientists 247–256 publications: 510 scientists 257–266 publications: 524 scientists 267–276 publications: 434 scientists 277–286 publications: 418 scientists 287–296 publications: 350 scientists 297–306 publications: 363 scientists 307–316 publications: 315 scientists 317–326 publications: 296 scientists 327–336 publications: 261 scientists 337–346 publications: 240 scientists 347–356 publications: 219 scientists 357–366 publications: 197 scientists 367–376 publications: 154 scientists 377–386 publications: 161 scientists 387–396 publications: 155 scientists 397–406 publications: 145 scientists 407–416 publications: 124 scientists 417–426 publications: 112 scientists 427–436 publications: 132 scientists 437–446 publications: 116 scientists 447–456 publications: 99 scientists 457–466 publications: 81 scientists 467–476 publications: 91 scientists 477–486 publications: 80 scientists 487–496 publications: 80 scientists 497–506 publications: 60 scientists 507–516 publications: 36 scientists 517–526 publications: 46 scientists 527–536 publications: 54 scientists 537–546 publications: 44 scientists 547–556 publications: 43 scientists 557–566 publications: 43 scientists 567–576 publications: 42 scientists 577–586 publications: 25 scientists 587–596 publications: 34 scientists 597–606 publications: 23 scientists 607–616 publications: 33 scientists 617–626 publications: 31 scientists 627–636 publications: 27 scientists 637–646 publications: 25 scientists 647–656 publications: 28 scientists 657–666 publications: 34 scientists 667–676 publications: 18 scientists 677–686 publications: 16 scientists 687–696 publications: 10 scientists 697–706 publications: 12 scientists 707–716 publications: 21 scientists 717–726 publications: 12 scientists 727–736 publications: 12 scientists 737–746 publications: 10 scientists 747–756 publications: 7 scientists 757–766 publications: 13 scientists 767–776 publications: 15 scientists 777–786 publications: 13 scientists 787–796 publications: 9 scientists 797–806 publications: 9 scientists 807–816 publications: 7 scientists 817–826 publications: 4 scientists 827–836 publications: 9 scientists 837–846 publications: 7 scientists 847–856 publications: 3 scientists 857–866 publications: 5 scientists 867–876 publications: 5 scientists 877–886 publications: 11 scientists 887–896 publications: 3 scientists 897–906 publications: 4 scientists 907–916 publications: 7 scientists 917–926 publications: 5 scientists 927–936 publications: 6 scientists 937–946 publications: 6 scientists 947–956 publications: 3 scientists 957–966 publications: 7 scientists 967–976 publications: 2 scientists 977–986 publications: 2 scientists 987–996 publications: 1 scientists 997–1,006 publications: 5 scientists 1,007–1,016 publications: 2 scientists 1,017–1,026 publications: 2 scientists 1,027 publications: 1 scientists 1,028+ publications: 100 scientists
47 publications 1,028+

This scientist: 210 publications — 54th percentile

54% of scientists in this discipline score the same or lower.

The last bar groups every scientist with 1,028 publications or more.

Benjamin T. Kile D-index placement in Biology and Biochemistry in 2026

The chart shows the D-index (discipline H-index) distribution of Biology and Biochemistry scientists ranked by Research.com in 2026. The highlighted bar marks where Benjamin T. Kile sits on this spectrum.

40–41 D-Index: 80 scientists 42–43 D-Index: 183 scientists 44–45 D-Index: 317 scientists 46–47 D-Index: 504 scientists 48–49 D-Index: 718 scientists 50–51 D-Index: 900 scientists 52–53 D-Index: 1,026 scientists 54–55 D-Index: 1,150 scientists 56–57 D-Index: 1,236 scientists 58–59 D-Index: 1,253 scientists 60–61 D-Index: 1,163 scientists 62–63 D-Index: 1,131 scientists 64–65 D-Index: 1,032 scientists 66–67 D-Index: 897 scientists 68–69 D-Index: 814 scientists 70–71 D-Index: 715 scientists 72–73 D-Index: 709 scientists 74–75 D-Index: 596 scientists 76–77 D-Index: 512 scientists 78–79 D-Index: 473 scientists 80–81 D-Index: 412 scientists 82–83 D-Index: 373 scientists 84–85 D-Index: 358 scientists 86–87 D-Index: 285 scientists 88–89 D-Index: 273 scientists 90–91 D-Index: 227 scientists 92–93 D-Index: 208 scientists 94–95 D-Index: 193 scientists 96–97 D-Index: 153 scientists 98–99 D-Index: 157 scientists 100–101 D-Index: 148 scientists 102–103 D-Index: 120 scientists 104–105 D-Index: 113 scientists 106–107 D-Index: 100 scientists 108–109 D-Index: 86 scientists 110–111 D-Index: 67 scientists 112–113 D-Index: 72 scientists 114–115 D-Index: 73 scientists 116–117 D-Index: 64 scientists 118–119 D-Index: 53 scientists 120–121 D-Index: 60 scientists 122–123 D-Index: 54 scientists 124–125 D-Index: 43 scientists 126–127 D-Index: 38 scientists 128–129 D-Index: 49 scientists 130–131 D-Index: 26 scientists 132–133 D-Index: 18 scientists 134–135 D-Index: 23 scientists 136–137 D-Index: 32 scientists 138–139 D-Index: 32 scientists 140–141 D-Index: 27 scientists 142–143 D-Index: 19 scientists 144–145 D-Index: 22 scientists 146–147 D-Index: 12 scientists 148–149 D-Index: 16 scientists 150–151 D-Index: 14 scientists 152–153 D-Index: 10 scientists 154–155 D-Index: 13 scientists 156–157 D-Index: 10 scientists 158–159 D-Index: 7 scientists 160–161 D-Index: 9 scientists 162–163 D-Index: 13 scientists 164–165 D-Index: 4 scientists 166 D-Index: 4 scientists 167+ D-Index: 98 scientists
40 D-Index 167+

This scientist: 68 D-Index — 61st percentile

61% of scientists in this discipline score the same or lower.

The last bar groups every scientist with 167 D-Index or more.

Research.com Recognitions

  • 2016 - Fellow of the Australian Academy of Health and Medical Science
  • 2013 - Gottschalk Medal, Australian Academy of Science

Overview

Benjamin T. Kile is affiliated with the University of Adelaide in Australia. Their research spans several interconnected fields, primarily within biochemistry, genetics, molecular biology, medicine, immunology, and microbiology.

Their main fields of study include:

  • Biochemistry, Genetics and Molecular Biology
  • Medicine
  • Immunology and Microbiology

Within these broad areas, the subfields frequently addressed are:

  • Molecular Biology
  • Immunology
  • Infectious Diseases
  • Hematology
  • Epidemiology

Kile's research engages with a range of scientific topics, especially focusing on:

  • Interferon and immune responses
  • Viral infections and vectors
  • Inflammasome and immune disorders
  • Cell death mechanisms and regulation
  • Immune response and inflammation
  • Autophagy in disease and therapy
  • Acute myeloid leukemia research

Recent publications demonstrate Kile's active involvement in understanding immune mechanisms and cell biology. Notable papers include:

  • "TBK1 and IKKε Act Redundantly to Mediate STING-Induced NF-κB Responses in Myeloid Cells" (2020, Cell Reports)
  • "Mitochondrial dysfunction caused by outer membrane vesicles from Gram-negative bacteria activates intrinsic apoptosis and inflammation" (2020, Nature Microbiology)
  • "A missense mutation in the MLKL brace region promotes lethal neonatal inflammation and hematopoietic dysfunction" (2020, Nature Communications)
  • "Termination of STING responses is mediated via ESCRT-dependent degradation" (2023, The EMBO Journal)
  • "COVID-19 patients exhibit reduced procoagulant platelet responses" (2020, Journal of Thrombosis and Haemostasis)

These publications appear frequently in well-regarded journals, with Kile contributing multiple works to venues such as:

  • Nature Communications
  • Cell Reports
  • Life Science Alliance
  • bioRxiv (Cold Spring Harbor Laboratory)
  • Blood

Collaborations play a significant role in their research output. Frequent coauthors include:

  • Katherine R. Balka
  • Dominic De Nardo
  • Stéphane Chappaz
  • Tahnee L. Saunders
  • Kate E. Lawlor

Kile's work has been recognized with awards such as:

  • Fellow of the Australian Academy of Health and Medical Science (2016)
  • Gottschalk Medal, Australian Academy of Science (2013)

Best Publications

  • Programmed Anuclear Cell Death Delimits Platelet Life Span

    Kylie D Mason;Marina Carpinelli;Jamie I Fletcher;Janelle Elyse Collinge

  • Apoptotic Caspases Suppress mtDNA-Induced STING-Mediated Type I IFN Production

    Michael J. White;Michael J. White;Kate McArthur;Kate McArthur;Donald Metcalf;Donald Metcalf;Rachael M. Lane

  • BAK/BAX macropores facilitate mitochondrial herniation and mtDNA efflux during apoptosis

    Kate McArthur;Kate McArthur;Kate McArthur;Lachlan W. Whitehead;Lachlan W. Whitehead;John M. Heddleston;Lucy Li

  • The SOCS box: a tale of destruction and degradation

    Benjamin T Kile;Brenda A Schulman;Warren S Alexander;Nicos A Nicola

  • Interleukin-11 Is the Dominant IL-6 Family Cytokine during Gastrointestinal Tumorigenesis and Can Be Targeted Therapeutically

    Tracy L. Putoczki;Tracy L. Putoczki;Stefan Thiem;Andrea Loving;Rita A. Busuttil;Rita A. Busuttil

  • TBK1 and IKKε Act Redundantly to Mediate STING-Induced NF-κB Responses in Myeloid Cells.

    Katherine R. Balka;Katherine R. Balka;Katherine R. Balka;Cynthia Louis;Cynthia Louis;Tahnee L. Saunders;Amber M. Smith

  • The Dendritic Cell Receptor Clec9A Binds Damaged Cells via Exposed Actin Filaments

    Jian Guo Zhang;Jian Guo Zhang;Peter E. Czabotar;Peter E. Czabotar;Antonia N. Policheni;Antonia N. Policheni;Irina Caminschi;Irina Caminschi;Irina Caminschi

  • NLRP1 Inflammasome Activation Induces Pyroptosis of Hematopoietic Progenitor Cells

    Seth L. Masters;Motti Gerlic;Motti Gerlic;Donald Metcalf;Donald Metcalf;Simon Preston;Simon Preston

  • Two distinct pathways regulate platelet phosphatidylserine exposure and procoagulant function

    Simone Marianne Schoenwaelder;Yu-Ping Yuan;Emma C Josefsson;Michael J White

  • Bcl-xL inhibitory BH3 mimetics can induce a transient thrombocytopathy that undermines the hemostatic function of platelets

    Simone M. Schoenwaelder;Kate E. Jarman;Elizabeth E. Gardiner;My Hua

  • The transcription factor Erg is essential for definitive hematopoiesis and the function of adult hematopoietic stem cells.

    Stephen J Loughran;Elizabeth A Kruse;Elizabeth A Kruse;Douglas F Hacking;Douglas F Hacking;Carolyn A de Graaf;Carolyn A de Graaf

  • Mitochondrial apoptosis is dispensable for NLRP3 inflammasome activation but non-apoptotic caspase-8 is required for inflammasome priming

    Ramanjaneyulu Allam;Kate E Lawlor;Kate E Lawlor;Eric Chi-Wang Yu;Alison L Mildenhall;Alison L Mildenhall

  • The suppressors of cytokine signalling (SOCS).

    B T Kile;W S Alexander

  • Functional genetic analysis of mouse chromosome 11

    Benjamin T. Kile;Kathryn E. Hentges;Amander T. Clark;Hisashi Nakamura

  • Megakaryocytes possess a functional intrinsic apoptosis pathway that must be restrained to survive and produce platelets

    Emma C. Josefsson;Chloé James;Katya J. Henley;Marlyse A. Debrincat;Marlyse A. Debrincat

  • The Mitochondrial Apoptotic Effectors BAX/BAK Activate Caspase-3 and -7 to Trigger NLRP3 Inflammasome and Caspase-8 Driven IL-1β Activation

    James E. Vince;James E. Vince;Dominic De Nardo;Dominic De Nardo;Wenqing Gao;Angelina J. Vince

  • Suppressors of cytokine signaling (SOCS): Negative regulators of signal transduction

    Warren S. Alexander;Robyn Starr;Donald Metcalf;Sandra E. Nicholson

  • IL-18 Production from the NLRP1 Inflammasome Prevents Obesity and Metabolic Syndrome.

    Andrew J. Murphy;Andrew J. Murphy;Michael J. Kraakman;Helene L. Kammoun;Dragana Dragoljevic;Dragana Dragoljevic

  • Apoptotic Caspases: Multiple or Mistaken Identities?

    Kate McArthur;Benjamin T. Kile

  • Mitochondrial dysfunction caused by outer membrane vesicles from Gram-negative bacteria activates intrinsic apoptosis and inflammation

    Pankaj Deo;Seong H Chow;Mei-Ling Han;Mary Speir

Frequent Co-Authors

Warren S. Alexander
Warren S. Alexander Walter and Eliza Hall Institute of Medical Research
Douglas J. Hilton
Douglas J. Hilton Walter and Eliza Hall Institute of Medical Research
Donald Metcalf
Donald Metcalf Walter and Eliza Hall Institute of Medical Research
David C. S. Huang
David C. S. Huang Walter and Eliza Hall Institute of Medical Research
Andrew W. Roberts
Andrew W. Roberts Walter and Eliza Hall Institute of Medical Research
Seth L. Masters
Seth L. Masters Hudson Institute of Medical Research
Nicos A. Nicola
Nicos A. Nicola Walter and Eliza Hall Institute of Medical Research
Andreas Strasser
Andreas Strasser Walter and Eliza Hall Institute of Medical Research
Tracy A. Willson
Tracy A. Willson Walter and Eliza Hall Institute of Medical Research
Lorraine A. O'Reilly
Lorraine A. O'Reilly Walter and Eliza Hall Institute of Medical Research

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Best Scientists Citing Benjamin T. Kile

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