Cell biology is often connected to Effector in his work. His Cell biology research extends to Effector, which is thematically connected. His Biochemistry study frequently links to adjacent areas such as Biogenesis. By researching both Biogenesis and Gene, he produces research that crosses academic boundaries. In his works, James E Vince performs multidisciplinary study on Gene and Gene expression. His Genetics research extends to the thematically linked field of Gene expression. As part of his studies on Genetics, he often connects relevant areas like Cancer cell. In his articles, he combines various disciplines, including Cancer cell and Apoptosis. He conducted interdisciplinary study in his works that combined Apoptosis and Inhibitor of apoptosis.
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IAP Antagonists Target cIAP1 to Induce TNFα-Dependent Apoptosis
James E. Vince;W. Wei-Lynn Wong;Nufail Khan;Rebecca Feltham.
Recruitment of the Linear Ubiquitin Chain Assembly Complex Stabilizes the TNF-R1 Signaling Complex and Is Required for TNF-Mediated Gene Induction
Tobias L. Haas;Christoph H. Emmerich;Christoph H. Emmerich;Bjã¶rn Gerlach;Bjã¶rn Gerlach;Anna C. Schmukle.
Molecular Cell (2009)
RIPK1 regulates RIPK3-MLKL-driven systemic inflammation and emergency hematopoiesis.
James A Rickard;James A Rickard;Joanne A O'Donnell;Joanne A O'Donnell;Joseph M Evans;Joseph M Evans;Najoua Lalaoui;Najoua Lalaoui.
Inhibitor of apoptosis proteins limit RIP3 kinase-dependent interleukin-1 activation.
James E. Vince;W. Wei Lynn Wong;W. Wei Lynn Wong;Ian Gentle;Kate E. Lawlor;Kate E. Lawlor.
RIPK3 promotes cell death and NLRP3 inflammasome activation in the absence of MLKL
Kate E. Lawlor;Nufail Khan;Alison Mildenhall;Motti Gerlic.
Nature Communications (2015)
AIM2 and NLRP3 inflammasomes activate both apoptotic and pyroptotic death pathways via ASC
V Sagulenko;S J Thygesen;D P Sester;A Idris.
Cell Death & Differentiation (2013)
Pyroptosis versus necroptosis: similarities, differences, and crosstalk
Daniel Frank;Daniel Frank;James E. Vince;James E. Vince.
Cell Death & Differentiation (2019)
The NLRP3 inflammasome in health and disease: the good, the bad and the ugly
P Menu;J E Vince.
Clinical and Experimental Immunology (2011)
Active MLKL triggers the NLRP3 inflammasome in a cell-intrinsic manner.
Stephanie A. Conos;Stephanie A. Conos;Kaiwen W Chen;Dominic De Nardo;Dominic De Nardo;Hideki Hara.
Proceedings of the National Academy of Sciences of the United States of America (2017)
TWEAK-FN14 signaling induces lysosomal degradation of a cIAP1–TRAF2 complex to sensitize tumor cells to TNFα
James E. Vince;Diep Chau;Bernard Callus;W. Wei-Lynn Wong.
Journal of Cell Biology (2008)
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