What is he best known for?
The fields of study he is best known for:
His primary areas of study are Cell biology, Inhibitor of apoptosis, Programmed cell death, XIAP and Apoptosis.
His Cell biology study incorporates themes from Inflammasome and NLRC4.
The concepts of his Inhibitor of apoptosis study are interwoven with issues in Ubiquitin, Ubiquitin ligase and Ubiquitin-conjugating enzyme.
His Programmed cell death study combines topics in areas such as Tumor necrosis factor alpha and Kinase activity.
His Tumor necrosis factor alpha study combines topics from a wide range of disciplines, such as Kinase and RIPK1, Necroptosis.
His research investigates the connection between Apoptosis and topics such as Cancer cell that intersect with issues in Caspase, Activator, Proteases and Targeted therapy.
His most cited work include:
- Differential activation of the inflammasome by caspase-1 adaptors ASC and Ipaf. (1408 citations)
- IAP antagonists induce autoubiquitination of c-IAPs, NF-kappaB activation, and TNFalpha-dependent apoptosis. (979 citations)
- Targeting IAP proteins for therapeutic intervention in cancer. (566 citations)
What are the main themes of his work throughout his whole career to date?
The scientist’s investigation covers issues in Cell biology, Inhibitor of apoptosis, Apoptosis, Programmed cell death and XIAP.
Domagoj Vucic interconnects Ubiquitin and Ubiquitin ligase in the investigation of issues within Cell biology.
His study looks at the relationship between Inhibitor of apoptosis and fields such as Cancer cell, as well as how they intersect with chemical problems.
The study incorporates disciplines such as Cancer, Cancer research and Immunology in addition to Apoptosis.
His Programmed cell death research is multidisciplinary, relying on both Tumor necrosis factor alpha, Proinflammatory cytokine and Drug discovery.
His work carried out in the field of XIAP brings together such families of science as Baculoviral IAP repeat-containing protein 3, Plasma protein binding, Antagonist, Activator and Cellular homeostasis.
He most often published in these fields:
- Cell biology (55.32%)
- Inhibitor of apoptosis (45.74%)
- Apoptosis (44.68%)
What were the highlights of his more recent work (between 2018-2021)?
- Programmed cell death (44.68%)
- Kinase (21.28%)
- RIPK1 (9.57%)
In recent papers he was focusing on the following fields of study:
Domagoj Vucic focuses on Programmed cell death, Kinase, RIPK1, Kinase activity and Inflammation.
As part of his studies on Programmed cell death, he often connects relevant areas like Cell biology.
The study incorporates disciplines such as Ubiquitin and Epigenetics in addition to Cell biology.
His studies examine the connections between Kinase and genetics, as well as such issues in Necroptosis, with regards to Cancer research, Caspase, Neurodegeneration and Protein kinase A.
Particularly relevant to Inhibitor of apoptosis is his body of work in Apoptosis.
His Inhibitor of apoptosis research incorporates themes from Proinflammatory cytokine, XIAP, Chemokine and Caspase 8.
Between 2018 and 2021, his most popular works were:
- The Indian cobra reference genome and transcriptome enables comprehensive identification of venom toxins. (42 citations)
- RIP1 inhibition blocks inflammatory diseases but not tumor growth or metastases. (37 citations)
- RIP1 kinase activity is critical for skin inflammation but not for viral propagation (19 citations)
In his most recent research, the most cited papers focused on:
His main research concerns Kinase, Inflammation, Cancer research, Necroptosis and Kinase activity.
His study in the field of Kinase inhibition also crosses realms of IRAK4.
The concepts of his Inflammation study are interwoven with issues in Bruton's tyrosine kinase and Bioinformatics.
His Cancer research research is multidisciplinary, incorporating perspectives in RIPK1, Immune system, Caspase, Programmed cell death and Tumor progression.
His Necroptosis research integrates issues from Signal transduction and Arthritis.
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