World's Best Scientists 2026 revealed!

D-Index & Metrics

Biology and Biochemistry

D-Index
40
Citations
9056
World Ranking
19578
National Ranking
590

Kate E. Lawlor publication distribution in Biology and Biochemistry in 2026

The chart shows the distribution of publications by all Research.com ranked scientists in the field of Biology and Biochemistry in 2026. The highlighted bar marks where Kate E. Lawlor sits on this spectrum.

47–56 publications: 8 scientists 57–66 publications: 35 scientists 67–76 publications: 106 scientists 77–86 publications: 231 scientists 87–96 publications: 414 scientists 97–106 publications: 546 scientists 107–116 publications: 704 scientists 117–126 publications: 849 scientists 127–136 publications: 980 scientists 137–146 publications: 942 scientists 147–156 publications: 969 scientists 157–166 publications: 950 scientists 167–176 publications: 951 scientists 177–186 publications: 915 scientists 187–196 publications: 787 scientists 197–206 publications: 841 scientists 207–216 publications: 735 scientists 217–226 publications: 709 scientists 227–236 publications: 651 scientists 237–246 publications: 605 scientists 247–256 publications: 510 scientists 257–266 publications: 524 scientists 267–276 publications: 434 scientists 277–286 publications: 418 scientists 287–296 publications: 350 scientists 297–306 publications: 363 scientists 307–316 publications: 315 scientists 317–326 publications: 296 scientists 327–336 publications: 261 scientists 337–346 publications: 240 scientists 347–356 publications: 219 scientists 357–366 publications: 197 scientists 367–376 publications: 154 scientists 377–386 publications: 161 scientists 387–396 publications: 155 scientists 397–406 publications: 145 scientists 407–416 publications: 124 scientists 417–426 publications: 112 scientists 427–436 publications: 132 scientists 437–446 publications: 116 scientists 447–456 publications: 99 scientists 457–466 publications: 81 scientists 467–476 publications: 91 scientists 477–486 publications: 80 scientists 487–496 publications: 80 scientists 497–506 publications: 60 scientists 507–516 publications: 36 scientists 517–526 publications: 46 scientists 527–536 publications: 54 scientists 537–546 publications: 44 scientists 547–556 publications: 43 scientists 557–566 publications: 43 scientists 567–576 publications: 42 scientists 577–586 publications: 25 scientists 587–596 publications: 34 scientists 597–606 publications: 23 scientists 607–616 publications: 33 scientists 617–626 publications: 31 scientists 627–636 publications: 27 scientists 637–646 publications: 25 scientists 647–656 publications: 28 scientists 657–666 publications: 34 scientists 667–676 publications: 18 scientists 677–686 publications: 16 scientists 687–696 publications: 10 scientists 697–706 publications: 12 scientists 707–716 publications: 21 scientists 717–726 publications: 12 scientists 727–736 publications: 12 scientists 737–746 publications: 10 scientists 747–756 publications: 7 scientists 757–766 publications: 13 scientists 767–776 publications: 15 scientists 777–786 publications: 13 scientists 787–796 publications: 9 scientists 797–806 publications: 9 scientists 807–816 publications: 7 scientists 817–826 publications: 4 scientists 827–836 publications: 9 scientists 837–846 publications: 7 scientists 847–856 publications: 3 scientists 857–866 publications: 5 scientists 867–876 publications: 5 scientists 877–886 publications: 11 scientists 887–896 publications: 3 scientists 897–906 publications: 4 scientists 907–916 publications: 7 scientists 917–926 publications: 5 scientists 927–936 publications: 6 scientists 937–946 publications: 6 scientists 947–956 publications: 3 scientists 957–966 publications: 7 scientists 967–976 publications: 2 scientists 977–986 publications: 2 scientists 987–996 publications: 1 scientists 997–1,006 publications: 5 scientists 1,007–1,016 publications: 2 scientists 1,017–1,026 publications: 2 scientists 1,027 publications: 1 scientists 1,028+ publications: 100 scientists
47 publications 1,028+

This scientist: 69 publications — 1st percentile

1% of scientists in this discipline score the same or lower.

The last bar groups every scientist with 1,028 publications or more.

Kate E. Lawlor D-index placement in Biology and Biochemistry in 2026

The chart shows the D-index (discipline H-index) distribution of Biology and Biochemistry scientists ranked by Research.com in 2026. The highlighted bar marks where Kate E. Lawlor sits on this spectrum.

40–41 D-Index: 80 scientists 42–43 D-Index: 183 scientists 44–45 D-Index: 317 scientists 46–47 D-Index: 504 scientists 48–49 D-Index: 718 scientists 50–51 D-Index: 900 scientists 52–53 D-Index: 1,026 scientists 54–55 D-Index: 1,150 scientists 56–57 D-Index: 1,236 scientists 58–59 D-Index: 1,253 scientists 60–61 D-Index: 1,163 scientists 62–63 D-Index: 1,131 scientists 64–65 D-Index: 1,032 scientists 66–67 D-Index: 897 scientists 68–69 D-Index: 814 scientists 70–71 D-Index: 715 scientists 72–73 D-Index: 709 scientists 74–75 D-Index: 596 scientists 76–77 D-Index: 512 scientists 78–79 D-Index: 473 scientists 80–81 D-Index: 412 scientists 82–83 D-Index: 373 scientists 84–85 D-Index: 358 scientists 86–87 D-Index: 285 scientists 88–89 D-Index: 273 scientists 90–91 D-Index: 227 scientists 92–93 D-Index: 208 scientists 94–95 D-Index: 193 scientists 96–97 D-Index: 153 scientists 98–99 D-Index: 157 scientists 100–101 D-Index: 148 scientists 102–103 D-Index: 120 scientists 104–105 D-Index: 113 scientists 106–107 D-Index: 100 scientists 108–109 D-Index: 86 scientists 110–111 D-Index: 67 scientists 112–113 D-Index: 72 scientists 114–115 D-Index: 73 scientists 116–117 D-Index: 64 scientists 118–119 D-Index: 53 scientists 120–121 D-Index: 60 scientists 122–123 D-Index: 54 scientists 124–125 D-Index: 43 scientists 126–127 D-Index: 38 scientists 128–129 D-Index: 49 scientists 130–131 D-Index: 26 scientists 132–133 D-Index: 18 scientists 134–135 D-Index: 23 scientists 136–137 D-Index: 32 scientists 138–139 D-Index: 32 scientists 140–141 D-Index: 27 scientists 142–143 D-Index: 19 scientists 144–145 D-Index: 22 scientists 146–147 D-Index: 12 scientists 148–149 D-Index: 16 scientists 150–151 D-Index: 14 scientists 152–153 D-Index: 10 scientists 154–155 D-Index: 13 scientists 156–157 D-Index: 10 scientists 158–159 D-Index: 7 scientists 160–161 D-Index: 9 scientists 162–163 D-Index: 13 scientists 164–165 D-Index: 4 scientists 166 D-Index: 4 scientists 167+ D-Index: 98 scientists
40 D-Index 167+

This scientist: 40 D-Index — 1st percentile

1% of scientists in this discipline score the same or lower.

The last bar groups every scientist with 167 D-Index or more.

Overview

Kate E. Lawlor is affiliated with the Hudson Institute of Medical Research in Australia. Their research primarily intersects the fields of immunology, microbiology, biochemistry, genetics, molecular biology, and medicine. Through these disciplines, Lawlor has contributed to understanding complex biological and immune processes.

The main fields of study encompass:

  • Immunology and Microbiology
  • Biochemistry, Genetics and Molecular Biology
  • Medicine

Their work narrows down to several subfields, including:

  • Immunology
  • Molecular Biology
  • Infectious Diseases
  • Epidemiology
  • Hematology

Kate E. Lawlor has researched extensively on topics such as:

  • Inflammasome and immune disorders
  • Interferon and immune responses
  • Cell death mechanisms and regulation
  • Immune Response and Inflammation
  • Immune Cell Function and Interaction
  • Phagocytosis and Immune Regulation
  • Autoimmune and Inflammatory Disorders Research

Their frequent publication venues include:

  • bioRxiv (Cold Spring Harbor Laboratory)
  • Cell Death and Disease
  • Cell Reports
  • Immunity
  • Nature Communications

Recent papers authored or coauthored by Kate E. Lawlor include:

  • TBK1 and IKKε Act Redundantly to Mediate STING-Induced NF-κB Responses in Myeloid Cells, 2020, Cell Reports
  • Mitochondrial dysfunction caused by outer membrane vesicles from Gram-negative bacteria activates intrinsic apoptosis and inflammation, 2020, Nature Microbiology
  • Interferon-γ primes macrophages for pathogen ligand-induced killing via a caspase-8 and mitochondrial cell death pathway, 2022, Immunity
  • The ubiquitylation of IL-1β limits its cleavage by caspase-1 and targets it for proteasomal degradation, 2021, Nature Communications
  • Die Another Way: Interplay between Influenza A Virus, Inflammation and Cell Death, 2020, Viruses

Kate E. Lawlor frequently collaborates with a group of scientists in related fields, including:

  • James E. Vince
  • James M. Murphy
  • Mary Speir
  • Daniel S. Simpson
  • Maryam Rashidi

Best Publications

  • ADAMTS5 is the major aggrecanase in mouse cartilage in vivo and in vitro

    Heather Stanton;Fraser M. Rogerson;Charlotte J. East;Suzanne B. Golub

  • RIPK3 promotes cell death and NLRP3 inflammasome activation in the absence of MLKL

    Kate E. Lawlor;Nufail Khan;Alison Mildenhall;Motti Gerlic

  • RIPK1 regulates RIPK3-MLKL-driven systemic inflammation and emergency hematopoiesis.

    James A Rickard;James A Rickard;Joanne A O'Donnell;Joanne A O'Donnell;Joseph M Evans;Joseph M Evans;Najoua Lalaoui;Najoua Lalaoui

  • Inhibitor of apoptosis proteins limit RIP3 kinase-dependent interleukin-1 activation.

    James E. Vince;W. Wei Lynn Wong;W. Wei Lynn Wong;Ian Gentle;Kate E. Lawlor;Kate E. Lawlor

  • Active MLKL triggers the NLRP3 inflammasome in a cell-intrinsic manner.

    Stephanie A. Conos;Stephanie A. Conos;Kaiwen W Chen;Dominic De Nardo;Dominic De Nardo;Hideki Hara

  • TBK1 and IKKε Act Redundantly to Mediate STING-Induced NF-κB Responses in Myeloid Cells.

    Katherine R. Balka;Katherine R. Balka;Katherine R. Balka;Cynthia Louis;Cynthia Louis;Tahnee L. Saunders;Amber M. Smith

  • FcgammaRIIb controls bone marrow plasma cell persistence and apoptosis.

    Zou Xiang;Antony J Cutler;Antony J Cutler;Rebecca J Brownlie;Kirsten Fairfax

  • Mutations that prevent caspase cleavage of RIPK1 cause autoinflammatory disease

    Najoua Lalaoui;Najoua Lalaoui;Steven E. Boyden;Hirotsugu Oda;Geryl M. Wood

  • TNFR1-dependent cell death drives inflammation in Sharpin-deficient mice

    James A Rickard;Holly Anderton;Nima Etemadi;Ueli Nachbur

  • Mitochondrial apoptosis is dispensable for NLRP3 inflammasome activation but non-apoptotic caspase-8 is required for inflammasome priming

    Ramanjaneyulu Allam;Kate E Lawlor;Kate E Lawlor;Eric Chi-Wang Yu;Alison L Mildenhall;Alison L Mildenhall

  • Blocking aggrecanase cleavage in the aggrecan interglobular domain abrogates cartilage erosion and promotes cartilage repair

    Christopher B. Little;Clare T. Meeker;Suzanne B. Golub;Kate E. Lawlor

  • Distinct cell-specific control of autoimmunity and infection by FcγRIIb

    Rebecca J. Brownlie;Kate E. Lawlor;Heather A. Niederer;Antony J. Cutler

  • The Mitochondrial Apoptotic Effectors BAX/BAK Activate Caspase-3 and -7 to Trigger NLRP3 Inflammasome and Caspase-8 Driven IL-1β Activation

    James E. Vince;James E. Vince;Dominic De Nardo;Dominic De Nardo;Wenqing Gao;Angelina J. Vince

  • cIAPs and XIAP regulate myelopoiesis through cytokine production in an RIPK1- And RIPK3-dependent manner

    W. Wei-Lynn Wong;W. Wei-Lynn Wong;W. Wei-Lynn Wong;James E. Vince;James E. Vince;Najoua Lalaoui;Najoua Lalaoui;Kate E. Lawlor;Kate E. Lawlor

  • Severe inflammatory arthritis and lymphadenopathy in the absence of TNF

    Ian K. Campbell;Kristy O’Donnell;Kate E. Lawlor;Ian P. Wicks

  • TNF can activate RIPK3 and cause programmed necrosis in the absence of RIPK1.

    D M Moujalled;W D Cook;T Okamoto;J J Murphy;J J Murphy

  • IL-18 Production from the NLRP1 Inflammasome Prevents Obesity and Metabolic Syndrome.

    Andrew J. Murphy;Andrew J. Murphy;Michael J. Kraakman;Helene L. Kammoun;Dragana Dragoljevic;Dragana Dragoljevic

  • Mitochondrial dysfunction caused by outer membrane vesicles from Gram-negative bacteria activates intrinsic apoptosis and inflammation

    Pankaj Deo;Seong H Chow;Mei-Ling Han;Mary Speir

  • Suppressor of cytokine signaling-1 regulates acute inflammatory arthritis and T cell activation

    Paul J. Egan;Kate E. Lawlor;Warren S. Alexander;Ian P. Wicks

  • Author response: TNFR1-dependent cell death drives inflammation in Sharpin-deficient mice

    James A Rickard;James A Rickard;Holly Anderton;Holly Anderton;Nima Etemadi;Nima Etemadi;Ueli Nachbur;Ueli Nachbur

Frequent Co-Authors

James E. Vince
James E. Vince Walter and Eliza Hall Institute of Medical Research
Ian P. Wicks
Ian P. Wicks Walter and Eliza Hall Institute of Medical Research
David L. Vaux
David L. Vaux Walter and Eliza Hall Institute of Medical Research
John Silke
John Silke Walter and Eliza Hall Institute of Medical Research
Seth L. Masters
Seth L. Masters Hudson Institute of Medical Research
James M. Murphy
James M. Murphy Walter and Eliza Hall Institute of Medical Research
Benjamin T. Kile
Benjamin T. Kile University of Adelaide
Lorraine A. O'Reilly
Lorraine A. O'Reilly Walter and Eliza Hall Institute of Medical Research
Warren S. Alexander
Warren S. Alexander Walter and Eliza Hall Institute of Medical Research
Andreas Strasser
Andreas Strasser Walter and Eliza Hall Institute of Medical Research

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