What is he best known for?
The fields of study he is best known for:
Jerry M. Adams mainly focuses on Cell biology, Apoptosis, Programmed cell death, BH3 Mimetic ABT-737 and Tissue homeostasis.
His research integrates issues of Caspase, Peptide sequence, Puma and Protein family in his study of Cell biology.
His Apoptosis study combines topics in areas such as Proteases, Cancer and MAPK/ERK pathway.
Jerry M. Adams has researched Programmed cell death in several fields, including Cancer research and Immunology.
The Cancer research study combines topics in areas such as Cell culture and Lymphoma.
His BH3 Mimetic ABT-737 research incorporates themes from Cell cycle and Bcl-2 Homologous Antagonist-Killer Protein.
His most cited work include:
- The Bcl-2 Protein Family: Arbiters of Cell Survival (4804 citations)
- The Bcl2 family: regulators of the cellular life-or-death switch. (3050 citations)
- Bcl-2 gene promotes haemopoietic cell survival and cooperates with c-myc to immortalize pre-B cells. (2763 citations)
What are the main themes of his work throughout his whole career to date?
Jerry M. Adams spends much of his time researching Molecular biology, Cell biology, Apoptosis, Cancer research and Gene.
His Molecular biology research includes themes of Transgene, Immunoglobulin heavy chain, Chromosomal translocation, RNA and Oncogene.
His Cell biology research is multidisciplinary, incorporating perspectives in Caspase, Tissue homeostasis and Cytokine.
His work on Programmed cell death, Bcl-2 family and Puma as part of general Apoptosis research is frequently linked to Bcl-2-associated X protein, bridging the gap between disciplines.
Jerry M. Adams interconnects Carcinogenesis, Cancer, Haematopoiesis, Lymphoma and Genetically modified mouse in the investigation of issues within Cancer research.
Jerry M. Adams has included themes like Antibody and DNA in his Gene study.
He most often published in these fields:
- Molecular biology (32.58%)
- Cell biology (37.83%)
- Apoptosis (37.08%)
What were the highlights of his more recent work (between 2009-2020)?
- Apoptosis (37.08%)
- Cancer research (31.46%)
- Cell biology (37.83%)
In recent papers he was focusing on the following fields of study:
Jerry M. Adams focuses on Apoptosis, Cancer research, Cell biology, Programmed cell death and Bcl-2-associated X protein.
His Apoptosis research includes elements of Cell, Haematopoiesis, Cell culture, Cytokine and Protein family.
The Cancer research study combines topics in areas such as Cancer, Immunology, Lymphoma, Venetoclax and Genetically modified mouse.
Jerry M. Adams usually deals with Cell biology and limits it to topics linked to Carcinogenesis and Progenitor cell.
His Programmed cell death research focuses on Tissue homeostasis and how it connects with Signal transduction.
His studies in Mitochondrion integrate themes in fields like Caspase, Activator and Cytosol.
Between 2009 and 2020, his most popular works were:
- Control of apoptosis by the BCL-2 protein family: implications for physiology and therapy (1632 citations)
- Deciphering the rules of programmed cell death to improve therapy of cancer and other diseases (377 citations)
- Deciphering the rules of programmed cell death to improve therapy of cancer and other diseases (377 citations)
In his most recent research, the most cited papers focused on:
His main research concerns Apoptosis, Cell biology, Cancer research, Programmed cell death and Mitochondrion.
His Apoptosis study frequently links to adjacent areas such as Protein family.
His Cell biology study combines topics in areas such as Cell, Tissue homeostasis, Carcinogenesis, Peptide sequence and Bcl-2 family.
His Cancer research study integrates concerns from other disciplines, such as Venetoclax, Chronic lymphocytic leukemia and Cancer.
His work carried out in the field of Chronic lymphocytic leukemia brings together such families of science as Myeloid, Cancer development and BH3 Mimetic ABT-737.
As a part of the same scientific family, Jerry M. Adams mostly works in the field of Mitochondrion, focusing on Protein structure and, on occasion, Bcl-2 Homologous Antagonist-Killer Protein, Biophysics and Molecular biology.
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