His scientific interests lie mostly in Cell biology, Apoptosis, Signal transduction, Programmed cell death and Molecular biology. The Cell biology study combines topics in areas such as Cytokine, Puma, Caspase, Caspase 8 and Caspase 1. His Apoptosis study integrates concerns from other disciplines, such as Chlamydia trachomatis, Kinase, Transfection and Interferon type I.
His Signal transduction research integrates issues from Cancer research and TRIF. Georg Häcker does research in Programmed cell death, focusing on Ripoptosome specifically. His Molecular biology research is multidisciplinary, relying on both Cytotoxic T cell, Apoptotic DNA fragmentation and T cell, Thymocyte.
His primary scientific interests are in Cell biology, Apoptosis, Programmed cell death, Immunology and Molecular biology. His work carried out in the field of Cell biology brings together such families of science as T cell and Chlamydia trachomatis. His work deals with themes such as Cell, Cell culture and Immune system, which intersect with Apoptosis.
His studies deal with areas such as Cancer research, Downregulation and upregulation and Virology as well as Cell culture. His Programmed cell death research incorporates elements of Necrosis, Phagocytosis, Cellular differentiation and Bacteria. His work in Signal transduction tackles topics such as Caspase 8 which are related to areas like FADD and Inhibitor of apoptosis.
His main research concerns Cell biology, Apoptosis, Chlamydia, Immunology and Microbiology. His studies examine the connections between Cell biology and genetics, as well as such issues in Programmed cell death, with regards to Intracellular. Georg Häcker interconnects Progenitor cell, Mitosis, DNA damage and Function in the investigation of issues within Apoptosis.
His Chlamydia research incorporates themes from Cell autonomous, Chlamydia trachomatis and Protein biosynthesis. His studies deal with areas such as Tuberculosis, Mycobacterium tuberculosis and Candidate gene as well as Immunology. His Microbiology study combines topics in areas such as Inflammation, Antibody and Intestinal mucosa.
His primary areas of study are Cancer research, Immune system, Function, Apoptosis and Immunology. Georg Häcker interconnects XIAP, NOD2, Pattern recognition receptor, XIAP Deficiency and Mitophagy in the investigation of issues within Cancer research. Georg Häcker combines subjects such as Cell, Chlamydia trachomatis, Signalling pathways and Epithelium with his study of Immune system.
The various areas that Georg Häcker examines in his Function study include Secretion, Cytokine secretion, Immunity and Apoptotic signaling pathway. His work deals with themes such as Programmed cell death and Intracellular, Cell biology, which intersect with Secretion. As part of his studies on Cell biology, he often connects relevant subjects like Lysis.
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Specificity in Toll-like receptor signalling through distinct effector functions of TRAF3 and TRAF6
Hans Häcker;Vanessa Redecke;Blagoy Blagoev;Irina Kratchmarova.
cIAPs Block Ripoptosome Formation, a RIP1/Caspase-8 Containing Intracellular Cell Death Complex Differentially Regulated by cFLIP Isoforms
Maria Feoktistova;Maria Feoktistova;Peter Geserick;Peter Geserick;Beate Kellert;Diana Panayotova Dimitrova.
Molecular Cell (2011)
The morphology of apoptosis.
Cell and Tissue Research (2000)
FOXO3a-dependent regulation of Puma in response to cytokine/growth factor withdrawal
Han You;Marc Pellegrini;Marc Pellegrini;Katsuya Tsuchihara;Katsuya Tsuchihara;Kazuo Yamamoto;Kazuo Yamamoto.
Journal of Experimental Medicine (2006)
Inhibitor of Apoptosis Proteins Limit RIP3 Kinase-Dependent Interleukin-1 Activation
James E. Vince;W. Wei Lynn Wong;W. Wei Lynn Wong;Ian Gentle;Kate E. Lawlor;Kate E. Lawlor.
Proapoptotic signaling induced by RIG-I and MDA-5 results in type I interferon–independent apoptosis in human melanoma cells
Robert Besch;Hendrik Poeck;Tobias Hohenauer;Daniela Senft.
Journal of Clinical Investigation (2009)
5′-triphosphate-siRNA: turning gene silencing and Rig-I activation against melanoma
Hendrik Poeck;Hendrik Poeck;Hendrik Poeck;Robert Besch;Cornelius Maihoefer;Cornelius Maihoefer;Marcel Renn.
Nature Medicine (2008)
NFκB activation by Fas is mediated through FADD, caspase-8, and RIP and is inhibited by FLIP
Sebastian Kreuz;Daniela Siegmund;Jost-Julian Rumpf;Dierk Samel.
Journal of Cell Biology (2004)
Chlamydia inhibit host cell apoptosis by degradation of proapoptotic BH3-only proteins.
Silke F. Fischer;Juliane Vier;Susanne Kirschnek;Andreas Klos.
Journal of Experimental Medicine (2004)
Puma cooperates with Bim, the rate-limiting BH3-only protein in cell death during lymphocyte development, in apoptosis induction
Miriam Erlacher;Verena Labi;Claudia Manzl;Günther Böck.
Journal of Experimental Medicine (2006)
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