What is he best known for?
The fields of study he is best known for:
The scientist’s investigation covers issues in RNA splicing, Genetics, Cell biology, RNA and NXF1.
His RNA splicing research integrates issues from Computational biology, Intron and splice.
His study in Messenger RNA, Splicing factor and Ribonucleoprotein is carried out as part of his Genetics studies.
He works mostly in the field of Ribonucleoprotein, limiting it down to concerns involving Exonic splicing enhancer and, occasionally, snRNP.
His study in Cell biology is interdisciplinary in nature, drawing from both Molecular biology and SR protein.
His research in RNA tackles topics such as Transcription which are related to areas like RNA polymerase II and Immunoprecipitation.
His most cited work include:
- An extensive network of coupling among gene expression machines (1007 citations)
- SF3B1 and Other Novel Cancer Genes in Chronic Lymphocytic Leukemia (840 citations)
- Comprehensive proteomic analysis of the human spliceosome. (758 citations)
What are the main themes of his work throughout his whole career to date?
Robin Reed mainly focuses on RNA splicing, Cell biology, Genetics, Spliceosome and Molecular biology.
His research integrates issues of Ribonucleoprotein, Messenger RNA and Intron in his study of RNA splicing.
His Cell biology research includes themes of Gene expression, RNA polymerase II, RNA-binding protein and Small nuclear RNA.
His Spliceosome study combines topics from a wide range of disciplines, such as RNA Splicing Factors, Minor spliceosome and Saccharomyces cerevisiae.
In his work, Sequence is strongly intertwined with Biophysics, which is a subfield of Molecular biology.
His RNA research is multidisciplinary, relying on both Computational biology, Transcription and Cell-free system.
He most often published in these fields:
- RNA splicing (64.96%)
- Cell biology (41.03%)
- Genetics (38.46%)
What were the highlights of his more recent work (between 2014-2019)?
- RNA splicing (64.96%)
- snRNP (26.50%)
- Cell biology (41.03%)
In recent papers he was focusing on the following fields of study:
His main research concerns RNA splicing, snRNP, Cell biology, Genetics and RNA polymerase II.
His RNA splicing research is multidisciplinary, incorporating elements of Mutation and Cancer research.
Robin Reed combines subjects such as Molecular biology, RNA-Binding Protein FUS and Morpholino with his study of snRNP.
The various areas that Robin Reed examines in his Cell biology study include MRNA Decay and RNA polymerase II holoenzyme.
His study on Cancer, Protein biosynthesis and Messenger RNA is often connected to Vulnerability and Dependency as part of broader study in Genetics.
His studies deal with areas such as Wild type, Gene and Spliceosome as well as Cancer.
Between 2014 and 2019, his most popular works were:
- Transcriptomic Characterization of SF3B1 Mutation Reveals Its Pleiotropic Effects in Chronic Lymphocytic Leukemia (105 citations)
- FUS functions in coupling transcription to splicing by mediating an interaction between RNAP II and U1 snRNP (58 citations)
- Evidence that C9ORF72 Dipeptide Repeat Proteins Associate with U2 snRNP to Cause Mis-splicing in ALS/FTD Patients (56 citations)
In his most recent research, the most cited papers focused on:
RNA splicing, snRNP, Spliceosome, Mutation and Cancer research are his primary areas of study.
His studies link Molecular biology with RNA splicing.
His Molecular biology research is multidisciplinary, incorporating perspectives in Small Nuclear Ribonucleoprotein Particle, Cytoplasm and Exon.
He works mostly in the field of snRNP, limiting it down to topics relating to RNA-Binding Protein FUS and, in certain cases, Cell biology, Transcription, RNA polymerase II, Small nuclear RNA and Morpholino, as a part of the same area of interest.
His studies in Spliceosome integrate themes in fields like Mutant, Notch signaling pathway, Alternative splicing and Gene expression profiling.
Robin Reed works mostly in the field of Mutation, limiting it down to topics relating to Ribonucleoprotein and, in certain cases, Splicing factor.
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