Robert G. Bristow mainly investigates Cancer research, Prostate cancer, DNA repair, Molecular biology and Radiation therapy. His Cancer research study incorporates themes from Radiosensitivity, Bioinformatics, Genome instability, Immunology and Gene. His study in Prostate cancer is interdisciplinary in nature, drawing from both Copy number analysis, Prostate, Oncology and Clinical trial.
His studies deal with areas such as Cancer cell, Cell cycle, DNA damage and Homologous recombination as well as DNA repair. Robert G. Bristow has researched Molecular biology in several fields, including Pre-replication complex, Eukaryotic DNA replication, RAD51 and Cell biology. His research in Radiation therapy intersects with topics in Hypoxia and Urology.
Robert G. Bristow spends much of his time researching Prostate cancer, Internal medicine, Oncology, Radiation therapy and Cancer research. His biological study deals with issues like Prostate, which deal with fields such as Nuclear medicine. His work deals with themes such as Biomarker, Androgen deprivation therapy, Pathological and Biochemical recurrence, which intersect with Oncology.
His Radiation therapy research incorporates themes from Clinical trial, Toxicity and Urology. His studies in Cancer research integrate themes in fields like Radiosensitivity, Hypoxia, Immunology, Radioresistance and DNA repair. His DNA repair research integrates issues from Molecular biology, Cell cycle, DNA damage and Homologous recombination.
His scientific interests lie mostly in Prostate cancer, Oncology, Internal medicine, Cancer research and Radiation therapy. Robert G. Bristow interconnects Transcriptome, Prostate, Disease and Genome instability in the investigation of issues within Prostate cancer. Robert G. Bristow combines subjects such as Regulation of gene expression and DNA repair with his study of Genome instability.
His Oncology research includes themes of Confidence interval, Androgen deprivation therapy, Carcinoma, Biomarker and Cohort. His study in Cancer research is interdisciplinary in nature, drawing from both Hypoxia, Tumor hypoxia, Gene, Lung cancer and PTEN. His work carried out in the field of Radiation therapy brings together such families of science as Biochemical recurrence, Medical physics, In vivo and Urology.
The scientist’s investigation covers issues in Prostate cancer, Prostate, Internal medicine, Oncology and Cancer research. His Prostate cancer research includes themes of Hypoxia, Clinical trial, Transcriptome, Genome instability and Computational biology. In Prostate, Robert G. Bristow works on issues like Metastasis, which are connected to Hazard ratio, Area under the curve and Hormone therapy.
His research in Cancer research intersects with topics in Mutation, Regulation of gene expression and Gene. His research integrates issues of Bladder cancer and Stage in his study of Radiation therapy. The study incorporates disciplines such as Cancer cell and Adenocarcinoma in addition to Carcinoma.
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Hypoxia and metabolism. Hypoxia, DNA repair and genetic instability.
Robert G. Bristow;Richard P. Hill.
Nature Reviews Cancer (2008)
Pan-cancer analysis of whole genomes
Peter J. Campbell;Gad Getz;Jan O. Korbel;Joshua M. Stuart.
Gold Nanoparticles as Radiation Sensitizers in Cancer Therapy
Devika B. Chithrani;Salomeh Jelveh;Farid Jalali;Monique van Prooijen.
Radiation Research (2010)
DNA Double-Strand Break Repair Pathway Choice Is Directed by Distinct MRE11 Nuclease Activities.
Atsushi Shibata;Atsushi Shibata;Davide Moiani;Davide Moiani;Andrew S. Arvai;Andrew S. Arvai;Jefferson Perry;Jefferson Perry;Jefferson Perry.
Molecular Cell (2014)
Management of Patients with Advanced Prostate Cancer: The Report of the Advanced Prostate Cancer Consensus Conference APCCC 2017
Silke Gillessen;Gerhardt Attard;Tomasz M Beer;Himisha Beltran.
European Urology (2018)
Genomic hallmarks of localized, non-indolent prostate cancer
Michael Fraser;Veronica Y. Sabelnykova;Takafumi N. Yamaguchi;Lawrence E. Heisler.
Chk2 is a tumor suppressor that regulates apoptosis in both an ataxia telangiectasia mutated (ATM)-dependent and an ATM-independent manner.
Atsushi Hirao;Alison Cheung;Gordon Duncan;Pierre Marie Girard.
Molecular and Cellular Biology (2002)
Spatial genomic heterogeneity within localized, multifocal prostate cancer
Paul C. Boutros;Paul C. Boutros;Michael Fraser;Nicholas J. Harding;Richard De Borja.
Nature Genetics (2015)
Analysis of the genetic phylogeny of multifocal prostate cancer identifies multiple independent clonal expansions in neoplastic and morphologically normal prostate tissue
Colin S Cooper;Colin S Cooper;Rosalind Eeles;Rosalind Eeles;David C Wedge;Peter Van Loo;Peter Van Loo;Peter Van Loo.
Nature Genetics (2015)
Down-Regulation of Rad51 and Decreased Homologous Recombination in Hypoxic Cancer Cells
Ranjit S. Bindra;Paul J. Schaffer;Alice Meng;Jennifer Woo.
Molecular and Cellular Biology (2004)
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